sort by

25 publications mentioning ssc-mir-27a

Open access articles that are associated with the species Sus scrofa and mention the gene name mir-27a. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 64
Other miRNAs from this paper: ssc-mir-224, cgr-mir-27a
Moreover, inducing the reduction of miR-27a expression using a miR-27a inhibitor led to an increase in DKK2 expression (Fig. 6E,F). [score:7]
A previous study of miR-27a showed that the down-regulation of miR-27a inhibited the proliferation of gastric cancer cells 29. [score:6]
These results suggest that miR-27a inhibits the proliferation of CHO cells by targeting DKK2. [score:5]
The DKK2 gene is regulated by the transcription factor C/EBPβ and miR-27a, and this process likely contributes to follicle development by inhibiting canonical WNT/β-catenin signaling. [score:5]
These findings indicate that DKK2 is a direct target of miR-27a. [score:4]
To determine whether DKK2 is a direct target of miR-27a, the pmirGLO-DKK2 luciferase reporter was co -transfected with miR-27a mimics or a negative control (NC) into CHO cells. [score:4]
How to cite this article: Tao, H. et al. The transcription factor ccaat/enhancer binding protein β (C/EBPβ) and miR-27a regulate the expression of porcine Dickkopf2 (DKK2). [score:4]
DKK2 is a direct target of miR-27a. [score:4]
MiR-27a dramatically suppressed endogenous DKK2 expression in CHO cells (Fig. 6E,F). [score:4]
As shown in Fig. 6B, overexpression of miR-27a reduced the luciferase activity of the pmirGLO-DKK2 reporter. [score:3]
TargetScan and RNAhybrid software predicted that the DKK2 3′ UTR contains a putative binding site for miR-27a (Fig. 6A). [score:3]
MiR-27a was a differentially expressed miRNA in the pre-ovulatory follicles from Large White and Chinese Taihu sows 27. [score:3]
DKK2 is a target of miR-27a. [score:3]
The relative expression of mus musculus (mmu) miR-27a (0.57 ± 0.18, P = 0.016) was significantly lower in the GCs of follicles containing MII oocytes compared with those of MI oocytes 27. [score:2]
Transfection with a mmu-miR-27a -mimic sequence decreased the oocyte maturation rate compared with that of the control (9.4 versus 18.9%, P = 0.042), and transfection with mmu-miR-27a inhibitor sequences increased the oocyte maturation rate by 1.67-folds compared with that of the control (31.6% versus 18.9%, P = 0.013) 28. [score:1]
To further understand the specificity of this miR-27a- DKK2 binding site, we constructed mutated pmirGLO-DKK2-mut luciferase reporters (Fig. 6A). [score:1]
CHO cells were transfected with pooled miR-27a mimics or NC when the cell index reached 1.0, and cell growth dynamics were then continuously monitored using an xCELLigence system. [score:1]
To determine the effects of miR-27a and DKK2 on cell proliferation, CHO cells were transfected with pc-DKK2 or pcDNA3.1 plasmids and pooled miR-27a mimics or a NC when the cell index reached 1.0. [score:1]
And the DKK2 promoter activity was significantly enhanced with allele T of DKK2 c. −1130 T > C compared with allele C. Our findings strongly suggest that WNT/β-catenin signaling pathway genes are regulated by C/EBPβ and miR-27a via DKK2. [score:1]
In addition, the miR-27a binding seed sequence in the DKK2 3′UTR is highly conserved in mammals (Fig. 6B). [score:1]
Red font indicates sequences that were mutated to abolish the interaction between miR-27a and the DKK2 3′UTR. [score:1]
[1 to 20 of 21 sentences]
2
[+] score: 17
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-17, hsa-mir-21, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-27a, hsa-mir-30a, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-107, mmu-let-7g, mmu-let-7i, mmu-mir-27b, mmu-mir-30a, mmu-mir-30b, mmu-mir-125b-2, mmu-mir-9-2, mmu-mir-150, mmu-mir-24-1, mmu-mir-204, hsa-mir-30c-2, hsa-mir-30d, mmu-mir-30e, hsa-mir-204, hsa-mir-210, hsa-mir-221, hsa-mir-222, mmu-let-7d, hsa-let-7g, hsa-let-7i, hsa-mir-27b, hsa-mir-30b, hsa-mir-125b-1, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-125b-2, hsa-mir-150, mmu-mir-30c-1, mmu-mir-30c-2, mmu-mir-30d, mmu-let-7a-1, mmu-let-7a-2, mmu-let-7b, mmu-let-7c-1, mmu-let-7c-2, mmu-let-7e, mmu-let-7f-1, mmu-let-7f-2, mmu-mir-15a, mmu-mir-21a, mmu-mir-24-2, mmu-mir-27a, mmu-mir-103-1, mmu-mir-103-2, mmu-mir-326, mmu-mir-107, mmu-mir-17, mmu-mir-210, mmu-mir-221, mmu-mir-222, mmu-mir-9-1, mmu-mir-9-3, mmu-mir-125b-1, hsa-mir-30c-1, hsa-mir-30e, hsa-mir-378a, mmu-mir-378a, hsa-mir-326, ssc-mir-125b-2, ssc-mir-24-1, ssc-mir-326, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-103-1, ssc-mir-107, ssc-mir-204, ssc-mir-21, ssc-mir-30c-2, ssc-mir-9-1, ssc-mir-9-2, hsa-mir-378d-2, hsa-mir-103b-1, hsa-mir-103b-2, ssc-mir-15a, ssc-mir-17, ssc-mir-30b, ssc-mir-210, ssc-mir-221, ssc-mir-30a, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-378-1, ssc-mir-30d, ssc-mir-30e, ssc-mir-103-2, ssc-mir-27b, ssc-mir-24-2, ssc-mir-222, ssc-mir-125b-1, hsa-mir-378b, hsa-mir-378c, ssc-mir-30c-1, ssc-mir-378-2, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-378f, hsa-mir-378g, hsa-mir-378h, hsa-mir-378i, mmu-mir-378b, ssc-let-7a-2, hsa-mir-378j, mmu-mir-21b, mmu-let-7j, mmu-mir-378c, mmu-mir-21c, mmu-mir-378d, mmu-mir-30f, ssc-let-7d, ssc-let-7f-2, ssc-mir-9-3, ssc-mir-150-1, ssc-mir-150-2, mmu-let-7k, ssc-mir-378b, mmu-mir-9b-2, mmu-mir-9b-1, mmu-mir-9b-3
Cai et al. (2014) found that 18 miRNAs were differentially expressed between intact and castrated male pigs, including miR-15a, miR-21, miR-27, miR-30, and so on [23]; Bai et al. (2014) reported that 177 miRNAs had more than 2-fold differential expression between castrated and intact male pigs, including miR-21, miR-30, miR-27, miR-103, and so on [22]. [score:5]
These indicated that miR-21, miR-30, and miR-27 and their target lncRNAs may play an important role in the androgen deficiency-related fat deposition, as it is wi dely known that miR-30a targets the androgen receptor (AR) gene [22]. [score:5]
Five depressing-adipogenesis miRNAs (miR-27, miR-150, miR-221, miR-222, and miR-326) target 217 lncRNAs. [score:3]
Our results were consisted with these reports, it was predicted that there were lncRNAs were the target genes for miR-21, miR-30, and miR-27. [score:3]
We analyzed the relationship between the 343 identified lncRNAs with the 13 promoting adipogenesis miRNAs (let-7、miR-9、miR-15a、miR-17、miR-21、miR-24、miR-30、miR-103、miR-107、miR-125b、miR-204、miR-210、and miR-378) and five depressing adipogenesis miRNAs (miR-27, miR-150, miR-221, miR-222, and miR-326). [score:1]
[1 to 20 of 5 sentences]
3
[+] score: 15
Among them, miR-27a is a known suppressor of adipocyte differentiation via suppression of PPARγ expression, which is a master transcription factor for adipocyte differentiation [15, 18] and is a regulator of porcine adipocyte lipid metabolism [34]. [score:8]
miR-27 can suppress the terminal differentiation of pre-adipocytes by targeting the adipogenic master genes, PPARγ and prohibitin [15- 17]. [score:5]
However, the expression of miR-27a and miR-31 was higher in the H group compared to the L group. [score:2]
[1 to 20 of 3 sentences]
4
[+] score: 15
This result is consistent with a previous report showing that ssc-miR-27a negatively regulates adipocyte differentiation by inhibiting the expression of PPARγ[10] (Additional file 9), and that miR-378 expression is also negatively correlated with cattle backfat thickness [35]. [score:8]
The expression levels of ssc-miR-21, ssc-miR-142-5p, ssc-miR-27a, ssc-miR-7134-3p and ssc-miR-103 were significantly higher in the intact male pigs than in the castrated male pigs, while the expression levels of ssc-miR-30a, ssc-miR-143 and ssc-miR-F3-C13 were lower in the intact male pigs (Figure  5). [score:5]
As an example, ssc-miR-27a and ssc-miR-378 were decreased by 3.14-fold and 6.06-fold in the castrated male pigs, respectively. [score:1]
A total of eight miRNAs were validated in the castrated and intact male pigs: ssc-miR-21, ssc-miR-30a, sssc-miR-27a, ssc-miR-143, ssc-miR-103, ssc-miR-142-5p ssc-miR-F3-C13 and ssc-miR-7134-3p. [score:1]
[1 to 20 of 4 sentences]
5
[+] score: 14
When testing miRNAs that were down-regulated, no miRNAs had a significant number of target genes that were up-regulated, but 4 miRNAs (miR-222, miR-27a, miR-574-5p, and miR-485-3p) had a significant number of target genes that were also down-regulated when using both DEG lists of <5% and <10%. [score:14]
[1 to 20 of 1 sentences]
6
[+] score: 12
To further support our results, miR-10a has been linked to the regulation of inflammation in endothelial cells [35], litter size in pigs was negatively associated with a single nucleotide polymorphism (SNP) in miR-27a expression [36], and miR-29c suppressed migration, invasion, and metastasis in nasopharyngeal carcinoma [37], [38]. [score:6]
Of these 5 genes, 3 (miR-27a, 29a, 99a) followed the microarray results in direction, but were greater in magnitude (p<0.05). [score:2]
Of the 38 significant differences observed by microarray in miRNA transcripts between arresting endometrium and arresting trophoblast, 5 (miR-27a, 29a, 29c, 99a, and 323) were assessed by Real-Time PCR. [score:1]
Of these, 4 (miR-27a, 29a, 29c, 99a) were confirmed to be significantly elevated in healthy endometrium than healthy trophoblast. [score:1]
Three of the 12 miRNAs (miR-27a, 29c, and 148a) could not be positively identified by sequencing. [score:1]
Three of the 12 miRNAs (miR-27a, 29c, and 148a) selected for Real-Time PCR validation of microarray results could not be positively identified by sequencing, even after cloning was repeated two additional times. [score:1]
[1 to 20 of 6 sentences]
7
[+] score: 11
Secondly, targets for a sub-set of down-regulated miR (miR-15, miR-16, miR-27, miR-29, miR-34 and miR-106), of which 44 targets were identified based on our previous criteria were predicted (see Additional file 4). [score:8]
These miR have been implicated in multiple cellular processes including cell growth (miR-24), apoptosis (miR-16, miR-24 and miR-29), and cell cycle regulation in normal (miR-16) and cancerous cells (miR-24, miR-27, miR-29 and miR-34) [9, 40- 46]. [score:2]
Conversely, miR-24 and miR-27 exhibited greatest abundance in proliferating satellite cells and the adult, while abundance of miR-368, miR-376, and miR-423-5p was greatest in the neonate. [score:1]
[1 to 20 of 3 sentences]
8
[+] score: 10
Although, the editing sites in miR-126, miR-744 and miR-27a were not in conserved seed sequences, they might affect the processing of mature microRNAs by down -regulating their expression levels. [score:4]
Compared with control groups, miR-27a was down-regulated at 3 dpi (log [2]FC = -0.75), 5 dpi (log [2]FC = -0.64) and 7 dpi (log [2]FC = -0.26), in Landrace pigs possibly due to microRNA editing altering immune responses to PRRSV infection. [score:3]
A previous study showed that miR-27a can keep inflammation from over responding 57. [score:1]
Further study showed that microRNA editing occurred in pre-miRNA-126, pre-miRNA-744 in Tongcheng and in pre-miRNA-27a in Landrace at 3, 5 and 7 dpi (Supplementary Table S5). [score:1]
Two out of 13 microRNAs (miR-126 and miR-744) were edited in the lungs of Tongcheng at all the three time points (3, 5, 7 dpi) and one microRNA (miR-27a) was edited in the lungs of Landrace at 3, 5, 7 dpi (Fig. 7c). [score:1]
[1 to 20 of 5 sentences]
9
[+] score: 9
Moreover, 13 miRNAs were differentially expressed: 8 were upregulated (ssc-miR-132, ssc-miR-146b, ssc-miR-215, ssc-miR-371, ssc-miR-27a, ssc-miR-331-3p, ssc-miR-432-5p and ssc-miR-199a/b-3p), while 5 were down-regulated after PRV infection (ssc-mir-10a-5p, ssc-mir-148-3p, ssc-mir-219a, ssc-mir-374b-3p and ssc-miR-532-5p) (Fig 7). [score:9]
[1 to 20 of 1 sentences]
10
[+] score: 8
Other miRNAs from this paper: ssc-mir-26a, ssc-mir-1, ssc-mir-29a, ssc-mir-27b, ssc-mir-26b
Expression rates were corrected for PCR efficiencies using dilution curves and the expressions of the candidate reference genes (let7a, miR-26, miR-27, RNU6B) were entered in Normfinder [13] for identification of the most stable transcripts. [score:5]
Accordingly, miRNA expression was analyzed in relation to the geometric mean of miR-27 and let7a. [score:3]
[1 to 20 of 2 sentences]
11
[+] score: 7
Moreover, miR-27a/b, a potential regulator of myogenesis, could induce skeletal muscle hypertrophy by down -regulating myostatin, an inhibitor of myogenesis (Huang et al., 2012; Sharma et al., 2014) and miR-27b inhibition leads to more proliferation and delays the onset of differentiation (Crist et al., 2009). [score:7]
[1 to 20 of 1 sentences]
12
[+] score: 7
Some miRs differentially expressed during adipogenesis have been identified, including miR-24 [8, 9], miR-31 [9] and the miR-17-92 cluster which alter cell proliferation [10]; miR-8 which represses Wnt signaling [11]; and miR-27 [12– 14] and miR-130 which target PPARγ [15]. [score:5]
For example, the miR-27 family negatively regulates adipogenesis by repressing PPARγ protein levels [12, 14]. [score:2]
[1 to 20 of 2 sentences]
13
[+] score: 6
In our previous studies, we found that 1.5% CLA was the most appropriate dose for improving the carcass traits and meat quality of pigs, and CLA significantly altered the expression of miR-27, miR-143, and adipocyte differentiation genes in adipose tissue of growing pigs [21]. [score:3]
Qi R. L. Chen Y. Huang J. X. Yang F. Y. Effects of conjugated linoleic acid on the expression levels of miR-27 and miR-143 in pig adipose tissueGenet. [score:3]
[1 to 20 of 2 sentences]
14
[+] score: 5
As illustrated in Figure 2, one of the poly-miRTSs identified by TargetSpy (a created site for miR-224 at position 50 of SLA-3 3′-UTR) was always associated with a miR-27a/b created site predicted by TargetScan and PACMIT at position 298. [score:5]
[1 to 20 of 1 sentences]
15
[+] score: 5
Liu et al. showed that miR-140 is a facilitator of adipocyte lineage commitment [2], whereas several other groups found that miR-27 can suppress the terminal differentiation of preadipocytes by targeting the adipogenic master gene, peroxisome proliferator-activated receptor γ (PPARγ) and prohibitin [3, 4]. [score:5]
[1 to 20 of 1 sentences]
16
[+] score: 4
Other miRNAs from this paper: ssc-mir-133a-1, ssc-mir-133b, ssc-mir-27b, ssc-mir-133a-2
The miR27 has been previously associated with an anti-adipogenic effect in mouse as it targets PPARG (reviewed in [83]), while the miR133 has been associated to brown adipose differentiation [84]. [score:3]
Two miRNA were deemed to be among the most induced during adipogenesis: miR27 and miR133. [score:1]
[1 to 20 of 2 sentences]
17
[+] score: 4
In this study, miR-27 was identified to be associated with PFK activity – a rate-limiting enzyme in glycolysis and potentially target cytochrome P450, family 24, subfamily A, polypeptide 1 (CYP24A1), which catalyzes the side-chain oxidation of vitamin D [82]. [score:3]
569 AMP miR-10, miR-126, let-7, miR-27, miR-450 9.860E-05–5.804E-04 0.789–0.730 ADP miR-15, miR-885, miR-322, miR-450, miR-338 1.316E-04–4.540E-03 0.781–0.636 ATP miR-15, miR-450, miR-210, miR-885, miR-451 4.811E-04–9.562E-03 0.737–0.593 Correlations between gene expression derived from post quality-filtered 17,820 mRNA probes and each phenotypic- trait were calculated for both Duroc and PiNN pigs. [score:1]
[1 to 20 of 2 sentences]
18
[+] score: 3
Many studies have demonstrated that miRNA expression profiles are subject to change in different cells when stimulated by LPS via TLR-signaling pathways, including miR-146a, miR-155, miR-132, miR-15a/16, miR-27a and miR-532-5p [19, 20, 21, 22, 23]. [score:3]
[1 to 20 of 1 sentences]
19
[+] score: 3
We found that miRNAs with higher expression in WBCs includes different miRNA families: mir-15, mir-17, mir-181, mir-23, mir-27 and mir-29 families. [score:3]
[1 to 20 of 1 sentences]
20
[+] score: 3
Other miRNAs from this paper: ssc-mir-143, ssc-mir-27b
Qi RL Chen Y Huang JX Yang FY Effects of conjugated linoleic acid on the expression levels of miR-27 and miR-143 in pig adipose tissueGenet. [score:3]
[1 to 20 of 1 sentences]
21
[+] score: 3
The selected miRNAs for the analysis include: miR-99b, miR-204, miR-27a, miR-24, miR-7, miR-145, miR-124, miR-21, miR-125b, miR-30b, miR-128a, miR-122, miR-183, and miR-103. [score:1]
0024883.g003 Figure 3 The selected miRNAs for the analysis include: miR-99b, miR-204, miR-27a, miR-24, miR-7, miR-145, miR-124, miR-21, miR-125b, miR-30b, miR-128a, miR-122, miR-183, and miR-103. [score:1]
0024883.g002 Figure 2 The selected miRNAs for the analysis include: miR-99b, miR-204, miR-27a, miR-24, miR-7, miR-145, miR-124, miR-21, miR-125b, miR-30b, miR-128a, miR-122, miR-183, and miR-103. [score:1]
[1 to 20 of 3 sentences]
22
[+] score: 2
Other miRNAs from this paper: ssc-mir-122, ssc-mir-135-1, ssc-mir-135-2, ssc-mir-148a, ssc-mir-19a, ssc-mir-20a, ssc-mir-224, ssc-mir-24-1, ssc-mir-323, ssc-mir-140, ssc-mir-183, ssc-mir-214, ssc-mir-325, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-103-1, ssc-mir-107, ssc-mir-136, ssc-mir-153, ssc-mir-18a, ssc-mir-186, ssc-mir-196a-2, ssc-mir-204, ssc-mir-21, bta-mir-18b, bta-let-7f-2, bta-mir-101-2, bta-mir-103-1, bta-mir-148a, bta-mir-18a, bta-mir-20a, bta-mir-21, bta-mir-221, bta-mir-27a, bta-mir-27b, bta-mir-107, bta-mir-140, bta-mir-20b, bta-mir-215, bta-let-7d, bta-mir-17, bta-mir-186, bta-mir-199b, bta-mir-210, bta-mir-214, bta-mir-450a-2, bta-let-7g, bta-mir-24-2, bta-let-7a-1, bta-let-7f-1, bta-mir-122, bta-let-7i, bta-let-7a-2, bta-let-7a-3, bta-let-7b, bta-let-7c, bta-let-7e, bta-mir-103-2, bta-mir-15a, bta-mir-19a, bta-mir-204, ssc-mir-15a, ssc-mir-17, ssc-mir-199b, ssc-mir-210, ssc-mir-221, bta-mir-101-1, bta-mir-133a-2, bta-mir-133a-1, bta-mir-135a-2, bta-mir-135a-1, bta-mir-135b, bta-mir-136, bta-mir-146b, bta-mir-153-1, bta-mir-153-2, bta-mir-183, bta-mir-24-1, bta-mir-196a-2, bta-mir-196a-1, bta-mir-196b, bta-mir-224, bta-mir-323, ssc-mir-101-1, ssc-mir-101-2, ssc-mir-133a-1, ssc-mir-450a, ssc-mir-146b, ssc-mir-215, bta-mir-1343, bta-mir-2320, bta-mir-2326, bta-mir-2366, bta-mir-2411, bta-mir-2483, bta-mir-450a-1, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-103-2, ssc-mir-27b, ssc-mir-24-2, ssc-mir-196b-1, ssc-mir-450b, ssc-mir-450c, ssc-mir-133a-2, ssc-let-7a-2, ssc-mir-18b, ssc-mir-1343, ssc-mir-2320, bta-mir-450b, ssc-let-7d, ssc-let-7f-2, ssc-mir-20b-1, ssc-mir-20b-2, ssc-mir-196a-1, ssc-mir-196b-2, ssc-mir-2366-1, ssc-mir-2366-2, ssc-mir-2411, ssc-mir-2483
Moreover, published report showed that miR-196, miR-214, miR-199b, miR-186, miR-101 and miR-27a were related to bovine backfat thickness [36]. [score:1]
Several miRNAs such as let-7, miR-27 and miR-103 are reported to perform very important functions in adipogenesis [33]– [35]. [score:1]
[1 to 20 of 2 sentences]
23
[+] score: 2
Lin Q., Gao Z., Alarcon R. M., Ye J., and Yun Z. 2009 A role of miR-27 in the regulation of adipogenesis. [score:2]
[1 to 20 of 1 sentences]
24
[+] score: 1
The first one harbored known miRNAs such as miR-26a, miR-27a, miR-125b, miR-130a and miR-145 but the latter was mainly composed of novel and not conserved miRNAs. [score:1]
[1 to 20 of 1 sentences]
25
[+] score: 1
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-17, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-26a-1, hsa-mir-27a, hsa-mir-29a, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-199a-1, hsa-mir-208a, hsa-mir-148a, hsa-mir-10a, hsa-mir-181a-2, hsa-mir-181c, hsa-mir-199a-2, hsa-mir-181a-1, hsa-mir-214, hsa-mir-221, hsa-let-7g, hsa-let-7i, hsa-mir-1-2, hsa-mir-23b, hsa-mir-27b, hsa-mir-125b-1, hsa-mir-128-1, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-143, hsa-mir-125b-2, hsa-mir-126, hsa-mir-127, hsa-mir-206, hsa-mir-1-1, hsa-mir-128-2, hsa-mir-29c, hsa-mir-26a-2, hsa-mir-378a, hsa-mir-148b, hsa-mir-133b, hsa-mir-424, ssc-mir-125b-2, ssc-mir-148a, ssc-mir-23a, ssc-mir-24-1, ssc-mir-26a, ssc-mir-29b-1, ssc-mir-181c, ssc-mir-214, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-103-1, ssc-mir-128-1, ssc-mir-29c, hsa-mir-486-1, hsa-mir-499a, hsa-mir-503, hsa-mir-411, hsa-mir-378d-2, hsa-mir-208b, hsa-mir-103b-1, hsa-mir-103b-2, ssc-mir-17, ssc-mir-221, ssc-mir-133a-1, ssc-mir-1, ssc-mir-503, ssc-mir-181a-1, ssc-mir-206, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-378-1, ssc-mir-133b, ssc-mir-29a, ssc-mir-199a-2, ssc-mir-128-2, ssc-mir-499, ssc-mir-143, ssc-mir-10a, ssc-mir-486-1, ssc-mir-103-2, ssc-mir-181a-2, ssc-mir-27b, ssc-mir-24-2, ssc-mir-23b, ssc-mir-148b, ssc-mir-208b, ssc-mir-424, ssc-mir-127, ssc-mir-125b-1, hsa-mir-378b, hsa-mir-378c, ssc-mir-411, ssc-mir-133a-2, ssc-mir-126, ssc-mir-199a-1, ssc-mir-378-2, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-378f, hsa-mir-378g, hsa-mir-378h, hsa-mir-378i, hsa-mir-499b, ssc-let-7a-2, ssc-mir-486-2, hsa-mir-378j, ssc-let-7d, ssc-let-7f-2, ssc-mir-29b-2, hsa-mir-486-2, ssc-mir-378b
In addition to the best-studied myomiRs (miR-1, -206 and miR-133 families), 11 other DE muscle-related miRNAs (miR-378 [24], miR-148a [27], miR-26a [28, 29], miR-27a/b [30, 31], miR-23a [32, 33], miR-125b [34], miR-24 [35], miR-128 [36], miR-199a [37] and miR-424 [38]) with high abundance (average RPM >1,000) and another 14 (miR-181a/b/c/d-5p [26], miR-499-5p [11], miR-503 [38], miR-486 [39], miR-214 [40], miR-29a/b/c [41– 43], miR-221/222 [44] and miR-208 [11] with low abundance (average RPM <1,000) were detected in myogenesis of pig. [score:1]
[1 to 20 of 1 sentences]