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12 publications mentioning rno-mir-351-2

Open access articles that are associated with the species Rattus norvegicus and mention the gene name mir-351-2. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

[+] score: 20
The miR-351 expression after UFP-512 treatment showed an initial slight yet significant upregulation following 1-day treatment, followed by a substantial downregulation by 45% after 5 days. [score:9]
Hypoxia alone downregulated the miR-351 expression after 10 days of exposure (Figure 7). [score:6]
Relative miRNA expression levels of miR-351 and miR-29a in the kidney following either 1, 5, or 10 days of hypoxia as determined by quantitative RT-PCR. [score:3]
In the case of miR-351, 10-day hypoxia induced a 30% decrease though no significant reduction was seen after 1- or 5-day hypoxia. [score:1]
Chronic UFP-512 treatment did not alter miR-351 levels under hypoxia. [score:1]
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[+] score: 9
On the other hand, miR-764-5p and miR-351, which are localized on chromosome X showed significant downregulation. [score:4]
[60] Several axonal guidance, cytoskeleton and cell adhesion genes (ABL1, EPHA10, NFATC1, PLCB3, ACTC1) that are critical for structural plasticity [61] are predicted to be targeted by miR-351 and miR-146a. [score:3]
For example, miR-218, miR-324-5p, miR-365 and miR-146a were localized on chromosome 10; miR-764-5p and miR-351 on chromosome X; miR-101 and miR-30e on chromosome 5; miR-582 and miR-137 on chromosome 2; miR-153 and miR-203 on chromosome 6; miR-124 and 181a on chromosome 3 and miR-135a*/miR-135a-3p and let-7i on chromosome 7. Some of the miRNAs that were localized on the chromosome and in close proximity showed the same direction of changes. [score:2]
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[+] score: 9
In the normoxic cortex, UFP-512 induced a significant upregulation in miR-351 expression by over 65%, (Figure 4c), but did increase miR-20b-5p (Figure 4a) and miR-212 (Figure 4b) expressions. [score:8]
These same conditions depressed miR-212 levels by 3-fold (Figure 4b) and miR-351 by 5-fold (Figure 4c). [score:1]
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[+] score: 8
Among the miRNAs, miR-214, miR-199a-5p, miR-150, miR-199a-3p, miR-351, miR-145, miR-764, miR-497 and miR-92b were upregulated, whilst miR-7a, miR-325-5p, miR-485, miR-708, miR-344-3p, let-7f, miR-26b, miR-129, miR-29c and let-7a were downregulated. [score:7]
These miRNAs include miR-214, miR-199a-5p, miR-150, miR-351, miR-145, miR-92b, miR-7a, miR-485, miR-708, let-7f, miR-26b, miR-129, miR-29c and let-7a. [score:1]
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[+] score: 8
Among these differentially expressed miRNAs, rno-miR-130b-3p, rno-miR-132-3p, rno-miR-181a-1-3p, rno-miR-222-3p, rno-miR-351-5p, and rno-miR-21-3p had the largest positive fold changes, while rno-miR-335, rno-miR-192-3p, rno-miR-194-5p, rno-miR-192-5p, rno-miR-499-5p, and rno-miR-210-3p had the largest negative fold changes (Table 1). [score:3]
In addition, as predicted by the bioinformatics databases, SLC4A1 was the potential target gene of rno-miR-34b, rno-miR-146b, rno-miR-214, rno-miR-223, and rno-miR-351. [score:3]
Relative expression levels of the selected miRNAs and mRNAs were depicted in Figure 3. Consistent with the microarray data, real-time PCR confirmed that, compared with controls, rno-miR-132-3p, rno-miR-181a-1-3p, rno-miR-222-3p, and rno-miR-351-5p were significantly increased, while rno-miR-192-3p, rno-miR-194-5p, rno-miR-29c-3p, rno-miR-185-5p, and rno-miR-30c-5p were significantly decreased in stone-forming rat kidneys. [score:2]
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[+] score: 6
In the mouse embryo at Theiler Stage 14, for example, we observed the expression of mir-133a-2 and mir-351 in heart ventricle. [score:3]
Similarly, longSAGE tags also suggest the expression of two human (mir-7-1 and mir-125a) and three mouse (mir-331, mir-351, and mir-495) miRNAs that have not been experimentally confirmed (Table 1). [score:3]
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[+] score: 4
To confirm the deep sequencing results, we used qRT-PCR to assess the expressions of 10 of the miRNAs (miR-183-5p, miR-9a-5p, miR-199a-5p, miR-351-5p, miR200b-3p, miR-191a-3p, miR-181c-3p, miR-330-5p, miR-126a-5p and miR-351-3p) in the 12-pair plasma samples from the hyperoxia rats and controls. [score:3]
Comparisons of the high-throughput sequencing and qRT-PCR results indicated that the two plarforms exhibited good correlation; and (b) The 5p/3p ratio validation results for miR-126a, miR-330 and miR-351 by qPCR also indicated good correlation between the two platforms. [score:1]
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[+] score: 4
Moreover, miR-351-5p directly target Ang-2 and VEGF and has been shown to participate in hypoxia -induced microvascular dysfunction [19]. [score:4]
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[+] score: 4
The upregulated miRNAs in the colon tissues of UC rats changed by HM were miR-149-5p, miR-351-5p, let-7d-5p, miR-98-5p, let-7a-5p, miR-3559-5p, let-7f-1-3p, miR-3596b, miR-224-5p, miR-411-3p, miR-184, miR-26b-3p, and miR-92b-3p. [score:4]
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[+] score: 3
The results showed that the expression of miR-322 and miR-351 was increased significantly with the duration of hypoxia exposure (Fig. 1b). [score:3]
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[+] score: 2
Ten miRNAs, miR-20b, miR-145, miR-152, miR-155, miR-181a, miR-203, miR-222, miR-301a, miR-324-5p, and miR-351. [score:1]
Similar analysis of “late” group, including c-Maf, Ets1, N-Myc, Nfat5, and Nfib, yielded 10 miRNAs: miR-20b, miR-145, miR-152, miR-155, miR-181a, miR-203, miR-222, miR-301a, miR-324-5p, and miR-351, with multiple connections. [score:1]
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[+] score: 1
Reference miRNAs (rno-miR-27b-3p, rno-miR-21-5p, rno-miR-151-3p, rno-miR-191a-5p, mmu-miR-351-5p, rno-miR-125a-5p, rno-miR-181a-5p) were selected using geNorm [6], and reached stability criteria. [score:1]
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