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36 publications mentioning hsa-mir-95

Open access articles that are associated with the species Homo sapiens and mention the gene name mir-95. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

[+] score: 162
b The disease-free survival rate of osteosarcoma patients with high miR-95-3p expression were significantly higher than those with low miR-95-3p expression (P = 0.008) Table 2 Multivariate survival analysis of overall survival in 133 osteosarcoma patients Variables Hazard ratio 95% CI P Sex 1.16 0.674–1.818 0.557 Age 0.87 0.469–2.473 0.682 Location 0.94 0.693–2.197 0.728 Tumor diameter 2.74 1.825–5.426 0.066 Clinical stage 3.12 1.725–6.429 0.018 Metastasis 3.35 1.794–7.531 0.031 miR-95-3p expression 4.22 2.314–8.072 0.014 miRNAs play crucial roles in multiple cellular processes and are associated with development and progression of various cancers and diseases. [score:12]
b The disease-free survival rate of osteosarcoma patients with high miR-95-3p expression were significantly higher than those with low miR-95-3p expression (P = 0.008) Table 2 Multivariate survival analysis of overall survival in 133 osteosarcoma patients Variables Hazard ratio 95% CI P Sex 1.16 0.674–1.818 0.557 Age 0.87 0.469–2.473 0.682 Location 0.94 0.693–2.197 0.728 Tumor diameter 2.74 1.825–5.426 0.066 Clinical stage 3.12 1.725–6.429 0.018 Metastasis 3.35 1.794–7.531 0.031 miR-95-3p expression 4.22 2.314–8.072 0.014 was conducted to determine the expression levels of serum miR-95-3p in patients with osteosarcoma and sex- and age-matched healthy controls. [score:11]
Hwang et al. (2015) revealed that overexpression of microRNA-95-3p inhibited brain metastasis of lung adenocarcinoma through downregulation of cyclin D1. [score:8]
In glioma cells, downregulation of miR-95-3p inhibits proliferation, invasion and promotes apoptosis (Fan et al. 2015). [score:6]
The Kaplan–Meier curves for overall survival showed that osteosarcoma patients with high miR-95-3p expression in serum survived significantly longer than those with low miR-95-3p expression (log-rank test, P = 0.017, shown in Fig.   3a). [score:5]
a The overall survival rate of osteosarcoma patients with high miR-95-3p expression were significantly higher than those with low miR-95-3p expression (P = 0.017). [score:5]
The correlation between miR-95-3p expression level, overall survival (Fig.   3a, P = 0.017) and disease-free survival (Fig.   3b, P = 0.008) of the patients with osteosarcoma was assessed using Kaplan–Meier survival analysis. [score:5]
The overall survival curves and disease-free survival curves for two groups of osteosarcoma patients with low and high expression of serum miR-95-3p. [score:5]
Taken together, these results demonstrated that low miR-95-3p expression in serum was associated with the progression and development of osteosarcoma. [score:4]
The level of miR-95-3p was upregulated in higher-grade glioma tissues of patients with glioma. [score:4]
a Serum miR-95-3p expression levels in 133 osteosarcoma patients and 133 sex- and age-matched healthy controls were determined by. [score:3]
In the present study, we demonstrated that miR-95-3p worked as an anti-oncogenic miRNA in osteosarcoma and decreased expression of miR-95-3p was a statistically significant risk factor reducing overall survival in osteosarcoma patients. [score:3]
In contrast, no significant association was found between the expression level of miR-95-3p with patients’ age (P = 0.234), sex (P = 0.634), location (P = 0.375), and tumor diameter (P = 0.627). [score:3]
qRT-PCR was conducted to determine the expression levels of serum miR-95-3p in patients with osteosarcoma and sex- and age-matched healthy controls. [score:3]
Furthermore, Kaplan–Meier analysis and log-rank test for overall survival showed that patients with low serum level of miR-95-3p had remarkably shorter overall survival time than those with high expression of serum miR-95-3p. [score:3]
The diagnostic value of serum miR-95-3p expression for osteosarcoma patients was assessed. [score:3]
The multivariate Cox proportional hazards mo del suggested that clinical stage, metastasis, and decreased expression of serum miR-95-3p were detected to be as independent prognostic factors for overall survival. [score:3]
As shown in Table  1, low-expressed miR-95-3p showed significant association with clinical stage (P = 0.000), metastasis (P = 0.000), and response to chemotherapy (P = 0.000). [score:3]
Our results suggested that miR-95-3p expression may decreased in serum of osteosarcoma patients. [score:3]
The nuclear RNA U6 was used as an endogenous control for the expression of precursor miR-95. [score:3]
miR-95-3p were downregulated in both high and low grade osteosarcoma tissues compared with normal controls (Novello et al. 2013). [score:3]
Multivariate Cox proportional hazards mo del analysis suggested that expression level of serum miR-95-3p was a significant independent prognostic factor of overall survival for patients with osteosarcoma (HR 4.22, 95% CI 2.314–8.072, P = 0.014, shown in Table  2). [score:3]
Of 133 osteosarcoma specimens, 69 osteosarcoma specimens (51.87%) showed low expression of miR-95-3p. [score:3]
The expression of miR-95-3p was elevated in colorectal carcinoma, Hela cells and pancreatic carcinoma, which suggested that miR-95-3p may play an oncogenic role in these carcinomas (Huang et al. 2011). [score:3]
Their data suggested that targeting miR-95-3p may be a novel therapeutic strategy for brain metastasis of lung adenocarcinoma cells. [score:3]
The prognostic value of miR-95-3p expression level was demonstrated for overall survival of patients with osteosarcoma. [score:3]
Fig.  2Diagnostic value of serum miR-95-3p expression for osteosarcoma patients. [score:3]
d, e Histological data showed fusiform osteosarcoma cells Table 1 Association of serum miR-95-3p expression with clinicopathological features of osteosarcoma Clinicopathological features No. [score:3]
In mice, experiments showed that miR-95-3p overexpression promoted tumor growth and increased radiation resistance in tumor xenografts (Huang et al. 2013). [score:3]
The 2 [−ΔΔCt] cycle threshold method was used to qualify the relative expression level of miR-95-3p. [score:3]
In this study, the expression levels of miR-95-3p in serum were determined in osteosarcoma patients and healthy controls. [score:3]
Decreased expression of miR-95-3p in serum associates with poor prognosis in osteosarcoma patients. [score:3]
These findings suggested that a lower expression level of serum miR-95-3p was implicated in the pathogenesis and progression of osteosarcoma. [score:3]
b ROC curve analysis showed that serum miR-95-3p was a novel potential biomarker for early diagnosis of osteosarcoma (AUC = 0.863) We divided the patients into two groups using the median value of serum miR-95-3p expression levels in osteosarcoma patients as a cutoff point (0.75). [score:3]
Fig.  3Kaplan-Meier survival curves for osteosarcoma patients with high or low expression of miR-95-3p. [score:3]
miR-95-3p expression levels were significantly decreased in serum of patients with osteosarcoma in comparison with those of healthy controls (mean ± SD 0.78 ± 0.32 vs. [score:3]
In the present study, we detected the expression levels of miR-95-3p in serum samples of osteosarcoma patients and healthy individuals. [score:3]
In addition, we found that downregulation of serum miR-95-3p in osteosarcoma patients was significantly associated with unfavorable clinicopathological characteristics, including metastasis, clinical stage and chemotherapy resistance. [score:2]
To further investigate the roles of miR-95-3p in the development and progression of osteosarcoma, the association between miR-95-3p expression and clinicopathological features of patients with osteosarcoma was analyzed using Chi square test. [score:2]
The expression level of precursor miR-95 was quantified using SYBR green assay (Invitrogen, Carlsbad, CA, USA) and real-time PCR was performed on a MyiQ Real-Time PCR Detection System (Bio-Rad, Richmond, CA, USA). [score:2]
The serum levels of miR-95-3p were significantly decreased in osteosarcoma patients in comparison with healthy controls. [score:1]
Further studies should be conducted to clarify the exact mechanism of miR-95-3p in osteosarcoma. [score:1]
Association between decreased serum levels of miR-95-3p expression and clinicopathologic characteristics in osteosarcoma patients. [score:1]
Detection of serum miR-95-3p levels may have clinical potentials as a valuable diagnostic and prognostic biomarker for osteosarcoma patients. [score:1]
Decreased levels of serum miR-95-3p in osteosarcoma patients. [score:1]
Osteosarcoma miR-95-3p Serum Diagnosis Prognosis Osteosarcomas are aggressive neoplasms of the bone that occurs predominantly in adolescents and young adults. [score:1]
We firstly demonstrated that decreased serum level of miR-95-3p may act as a diagnostic and prognostic biomarker for patients with osteosarcoma. [score:1]
However, the molecular mechanism of miR-95-3p in osteosarcoma tumorigenesis and progression has not yet been elucidated. [score:1]
However, the potential roles of miR-95-3p in pathogenesis and prognosis of osteosarcoma are not yet illustrated. [score:1]
Our data showed that miR-95-3p could distinguish osteosarcoma patients from healthy controls efficiently (AUC = 0.863). [score:1]
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[+] score: 26
We found that miR-432, miR-1286, miR-641, miR-1290, miR-1287 and miR-95 were highly upregulated just in cells with HPV infection upon 5-AZA treatment, whereas miR-625 was significantly downregulated (P<0.05) (Figure  3). [score:7]
Contradictorily, our results showed that after treatment of 5-AZA, miR-95 was significantly up-regulated in cell lines, whereas the expression of it was lower in cervical cancer than normal tissues. [score:6]
However in head and neck cancers, miR-95 has been suggested downregulated (Nurul-Syakima AM, et al., [23]). [score:4]
In HeLa cells, inhibition of miR-95 caused a decrease in cell growth (Cheng AM, et al., [24]). [score:3]
Interestingly, expression of miR-641 and miR-1287 in cervical tissue was lower than control, while miR-95 was higher (P<0.05). [score:3]
We found hypermethylation of miR-432, miR-1286, miR-641, miR-1290, miR-1287 and miR-95 may have some relationship with HPV infection in cervical cell lines. [score:1]
MiR-432, miR-1286, miR-641, miR-1290, miR-1287 and miR-95 were found up-modulated in Caski, Hela and Siha but not in C33A induced on treatment, while miR-625 was down-modulated. [score:1]
MiR-641, miR-1287 and miR-95 changing most significantly in cell lines were chosen for further study in cervical tissue. [score:1]
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[+] score: 17
The results indicate that miR-95 was up-regulated by P [4] and reached maximal levels with E [2]+P [4], while miR-96 and miR-135b were both down-regulated by E [2] or P [4] (Figure 1A). [score:7]
Next, we examined the effect of individual ovarian steroids on the regulation of miR-95, this being the most up-regulated miRNA in our in vitro decidualization assay, as well as miR-135 b and miR-96. [score:4]
0041080.g001 Figure 1 A, miR-95, miR-135b and miR-96 expression levels by quantitative PCR in hESCs treated with 17β-estradiol (E), progesterone (P), or both (E+P), for 9 days. [score:3]
A, miR-95, miR-135b and miR-96 expression levels by quantitative PCR in hESCs treated with 17β-estradiol (E), progesterone (P), or both (E+P), for 9 days. [score:3]
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[+] score: 16
MiR-95 is overexpressed in pancreatic, prostate, breast, and colorectal cancer, where it promotes cell proliferation by targeting Nexin-1 [39]. [score:4]
Huang X miRNA-95 mediates radioresistance in tumors by targeting the sphingolipid phosphatase SGPP1Cancer Res. [score:3]
c qRT-PCR for miR-95, -339, and -663b in A549 and H1299 cells untreated (Con) or transfected with a plasmid -expressing wild-type uc. [score:3]
MiR-95 promotes resistance to radiotherapy in prostate and breast cancer cells by targeting Sphingolipid Phosphatase SGPP1 [40]. [score:2]
Huang Z MicroRNA-95 promotes cell proliferation and targets sorting Nexin 1 in human colorectal carcinomaCancer Res. [score:2]
The precursors of miR-95-5p, -339-3p, and -663b-3p and si-miR-339-3p, -663b-3p, and -95-5p (as well as the scrambled miRNA negative control #1) were purchased from Ambion, and were transfected in cell lines at a final concentration of 100 nM, by using Lipofectamine [®] 2000 (Invitrogen), according to the manufacturer’s instructions. [score:1]
Of the three interacting miRNAs, miR-339-3p is very conserved among species, miR-95-5p is conserved in humans, pig, dog, cow, and horse, but not in mouse; so it evolved after the mouse appearance, but long before humans, whereas miR-663b-3p is primate specific. [score:1]
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[+] score: 16
It is conceivable that several of the down-regulated miRNAs with no defined functions at this point (i. e., miR-873, miR-95, miR-543) may exhibit tumor suppressor activity and normally target components of key signaling pathways that promote and maintain the growth and survival of glioma cells as has recently been reported for miR-10b [17]. [score:8]
While the aberrant expression of many of these miRNAs is consistent with previous profiling studies (Table S6) [9], [14]– [16], [33], deep sequencing analysis further revealed at least four significantly down-regulated miRNAs (miR-95, miR-543. [score:6]
We identified several new miRNAs that were not previously reported to be dysregulated in glioblastomas, including miR-95, miR-543, miR-598, and miR-873 (Fig. 1, Table S6). [score:2]
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[+] score: 14
They have observed an increase in SUMF1 expression and in its enzymatic activity as a result of miR-95 inhibition. [score:5]
These data were confirmed in cells of MSD patients, since authors proved that inhibition of miR-95 could raise residual SUMF expression and also SUMF1 activity. [score:5]
Frankel et al. (26) demonstrated that miR-95 controls the expression of sulfatase-modifying factor 1 (SUMF1) gene, which encodes an activator of sulfatases. [score:3]
In addition, miR-95 transfected cells showed higher GFP-LC3II positive cells and the accumulation of early endosomes, multivesicular bodies, autophagic substrates, and autophagic vacuoles, which reinforces the interference caused by miR-95 in endocytic and autophagic mechanisms. [score:1]
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[+] score: 9
Seven miRNAs had different expression levels between active TB and healthy controls: six miRNAs (hsa-miR-16, hsa-miR-137, hsa-miR-140-3p, hsa-miR-193a-3p, hsa-miR-501-5p, and hsa-miR-598) were upregulated while hsa-miR-95 was down-regulated. [score:9]
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[+] score: 8
In an in-vitro study using H-69 lung cancer cell line, Pak et al. (2014) reported upregulation of miR-16-2, miR-93, miR-95, mir-153, mir-195, miR-199a-3p, and down-regulation of miR let7a, let7i, miR-124a in the presence of excretory secretory protein of C. sinensis. [score:7]
Among the miRNAs, seven of them (miR-16-2, miR-93, miR-95, miR-136, miR-153, miR-195, and miR-199a-3p) were mainly associated with esophageal adenocarcinoma, breast cancer and colorectal carcinoma, suggesting the role of these miRs in cell-proliferation and cell-signaling. [score:1]
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[+] score: 7
Furthermore, we found 8 miRNAs (miR-95, miR-139, miR-379, miR-429, miR-509, miR-518e, miR-542-5p, and miR-659) downregulated in both 6 h aDCs and tDCs with respect to iDCs. [score:4]
Interestingly, 5 miRNAs (miR-95, miR-429, miR-509, miR-542, and miR-659) were downregulated in tDCs and aDCs compared with iDCs. [score:3]
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[+] score: 6
Our results in the two cell lines agree with previously published data on the upregulation of MIR93, MIR95, MIR135B, MIR181C, MIR181D, MIR182, MIR183, MIR190, MIR196B and MIR203, and downregulation of MIR20A and MIR29C in pancreatic intraepithelial neoplasms (PanIns) or pancreatic adenocarcinomas (PDACs) as compared to normal pancreatic tissue [34- 36]. [score:6]
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[+] score: 6
Among them, only miRNA-95 expression was found to be related to adipocyte size in both fat depots. [score:3]
SC miR-95 expression is related to serum concentrations of adiponectin (r = 0.52, p<0.05), CrP (r = −0.59, p<0.05), and IL-6 (r = −0.53, p<0.05). [score:3]
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[+] score: 6
The predicted MREs for the significantly -upregulated circRNAs are listed in Table 2, with hsa-miR-329-5p being the most frequently targeted miRNA by three circRNAs (i. e., hsa_circRNA_104566, hsa_circRNA_104565, and hsa_circRNA_105038) followed by hsa-miR-450b-5p, hsa-miR-205-3p, hsa-miR-95-5p, hsa-miR-339-5p, and hsa-miR-646. [score:6]
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[+] score: 6
Other miRNAs from this paper: hsa-mir-96, hsa-mir-149
Invasion and migration assay underlying GPC1, miR-95-5p, and miR-149 overexpression and inhibition of GPC1 expression. [score:6]
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[+] score: 5
Twelve of 116 TS miRNAs (miR-1, miR-126, miR-208, miR-128a, miR-133a, miR-133b, miR-134, miR-146a, miR-377, miR-483, miR-92a and miR-95) were specifically expressed in two tissues; whereas, the remaining TS miRNAs were specifically expressed in only one tissue. [score:5]
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[+] score: 5
Alterations in miRNA expression have been observed in CRC, and several dysregulated miRNAs, including miR-625-3p [8], miR-99-5b [9], miR-361-5p [10], miR-17-5p [11], miR-137 [12], miR-95 [13], miR-23a [14, 15], miR-155 [16], miR-150 [17], miR-191[18], miR-339-5p [19], miR-429 [20], miR-345 [21], miR-22 [22], miR-638 [23] and miR-138 [24], have been shown to regulate CRC cell growth, apoptosis and metastasis. [score:5]
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[+] score: 5
Our previous work revealed that miR-95, which is overexpressed in CRC, could promote CRC cell proliferation by targeting SNX1 [16]. [score:5]
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[+] score: 5
Instead, miR-378, miR-10b and miR-95 were expressed mainly in HLSCs and miR-369-5p, miR-594 and miR-654, were expressed mainly in MSCs rather than in their MVs, suggesting that these miRNAs were not compartmentalized within MVs and therefore not secreted. [score:5]
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[+] score: 4
Colorectal cancer cell lines with KRAS mutations showed an over -expression of miR-9, miR-9*, miR-95, miR-148a, miR-190 and miR-372, in relation to the human normal colon cell line. [score:4]
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[+] score: 4
For example, four transporters genes were negatively correlated with rifampin -induced miRNA expression: SLC47A1/miR-95, SLC29A1/miR-30d#, SLC22A9/miR-202, and SLCO1B3/miR-92a (Table 3). [score:3]
Three miRNAs, miR-320, miR-616, and miR-95, each correlated with two different drug transporters. [score:1]
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[+] score: 4
Conversely, 15 miRNAs resulted downregulated in activated B cells: mir-483, mir-95, mir-326, mir-135a, mir-184, mir-185, mir-516-3p, mir-30b, mir-203, mir-216, mir-150, mir-182*, mir-141 and mir-211 (Table 3). [score:4]
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[+] score: 3
Expression pattern of 30 miRNAs (miR-17-3p, miR-92, miR-135b, miR-222, miR-95, etc. ) [score:3]
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[+] score: 3
Seven miRNAs (miR-31, miR-95, miR-99a, miR-130b, miR-10a, miR-134, and miR-146a) were expressed at 6-fold lower level in SLE patients than that of healthy controls (Tang et al., 2009). [score:3]
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[+] score: 3
34 hsa-miR-335 −0.35hsa-miR-345 [44], [53], [71] 1.16 hsa-miR-363 0.99 hsa-miR-371-5p 0.55 hsa-miR-421 0.50 hsa-miR-483-5p 1.33 hsa-miR-494 0.87 hsa-miR-505* −0.40 hsa-miR-513a-5p 1.06 hsa-miR-513b 1.19 hsa-miR-513c 1.22 hsa-miR-551b −0.40 hsa-miR-574-5p 0.97hsa-miR-630 [68], [73] 0.96 hsa-miR-769-5p −0.34 hsa-miR-801 0.66 hsa-miR-873 −0.64 hsa-miR-877* 0.72 hsa-miR-923 0.89 hsa-miR-940 0.49 hsa-miR-95 −0.44 hsa-miR-99a −0.64Irradiated and non-irradiated PBL of the same donors were incubated in static gravity (1 g) for 4 and 24 h, and miRNA expression profile was analyzed at the end of each incubation time. [score:3]
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[+] score: 3
showed that the level of the CHOP protein was significantly decreased by miR-95-3p mimics and increased by miR-183-5p inhibitor (Fig.   5e). [score:3]
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[+] score: 3
In addition, FTY720 increased the radio-sensitivity of prostate cancer cells overexpressing miR-95, a microRNA associated with resistance to radiation) 72. [score:3]
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[+] score: 3
For example, while MIR95 is highly expressed in both prostate and lung cancers and have oncogenic function [11, 12]; the anti-proliferative role of MIR95 was observed in breast cancer MCF-7 cells [13]. [score:3]
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[+] score: 2
Three miRNAs miR-146a-5p, miR-95-5p and miR-181d show their expected expression profile in HER2 -positive breast cancers when compared with other subtypes of breast cancers. [score:2]
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[+] score: 2
Our results also suggest the involvement of miR-127, miR-27b, miR-30b, miR-655, miR-95 and miR-495 in skeletal muscle development (S1B Fig. ). [score:2]
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[+] score: 1
2 −2.40(52) hsa-miR-127-5p 14q32.2 −2.35(12, 29) hsa-miR-127-3p 14q32.2 −2.35(52, 59) hsa-miR-411* 14q32.2 −2.30 hsa-miR-125b-1* 11q24.1/21q21.1 −2.27 hsa-miR-411 14q32.2 −2.23(29) hsa-miR-379 14q32.2 −2.22(29, 52) hsa-miR-431* 14q32.2 −2.22 hsa-miR-767-5p Xq28 −2.20 hsa-miR-139-3p 11q13.4 −2.17 hsa-miR-154 14q32.2 −2.16(12) hsa-miR-1224-5p 3q27.2 −2.15 hsa-miR-187 18q12.1 −2.14(12) hsa-miR-95 4p16.1 −2.10(14) hsa-miR-369-5p 14q32.2 −2.05 hsa-miR-665 14q32.2 −2.05 hsa-miR-494 14q32. [score:1]
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[+] score: 1
For instance, mir-28, mir-95 and mir-151 are derived from LINE-2 TEs, and mir-548 family is derived from Made1 TEs [21– 23]. [score:1]
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[+] score: 1
Validation was performed on five miRNAs (miR-191, miR-95, miR-17, miR-181a, miR-106b), using housekeeping small RNA (RNU6B) as a control. [score:1]
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[+] score: 1
However, similarly to some studies already reporting gender effects on some miRNAs for e. g. human brain tissue [44], we found gender specific differences for miR-106a, miR-17 and miR-320 in set A and miR-19a, miR-221, miR-532, miR-95 in set B. Therefore, we decided to control the results towards age and sex by considering these variables as covariates in the confirmatory MANCOVAs. [score:1]
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MiR-186, miR-769, miR-95, miR-202 and let-7 g were also relevant to cancer pathways, but did not serve other functions. [score:1]
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[+] score: 1
Finally, miR-95, significantly associated with adipocyte size in the aforementioned study [26], increased significantly during adipocyte differentiation (current results). [score:1]
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[+] score: 1
Other miRNAs from this paper: hsa-mir-25, hsa-mir-28, mmu-mir-151, mmu-mir-290a, mmu-mir-297a-1, mmu-mir-297a-2, mmu-mir-130b, mmu-mir-340, mmu-mir-25, mmu-mir-28a, hsa-mir-130b, hsa-mir-367, hsa-mir-372, hsa-mir-378a, mmu-mir-378a, hsa-mir-340, hsa-mir-151a, mmu-mir-466a, mmu-mir-467a-1, hsa-mir-505, hsa-mir-506, mmu-mir-367, hsa-mir-92b, hsa-mir-548a-1, hsa-mir-548b, hsa-mir-548a-2, hsa-mir-548a-3, hsa-mir-548c, hsa-mir-648, hsa-mir-548d-1, hsa-mir-548d-2, hsa-mir-659, hsa-mir-421, hsa-mir-151b, hsa-mir-1271, hsa-mir-378d-2, mmu-mir-467b, mmu-mir-297b, mmu-mir-505, mmu-mir-297a-3, mmu-mir-297a-4, mmu-mir-297c, mmu-mir-421, mmu-mir-466b-1, mmu-mir-466b-2, mmu-mir-466b-3, mmu-mir-466c-1, mmu-mir-466e, mmu-mir-466f-1, mmu-mir-466f-2, mmu-mir-466f-3, mmu-mir-466g, mmu-mir-466h, mmu-mir-467c, mmu-mir-467d, mmu-mir-92b, mmu-mir-466d, hsa-mir-297, mmu-mir-467e, mmu-mir-466l, mmu-mir-669g, mmu-mir-466i, mmu-mir-466f-4, mmu-mir-466k, mmu-mir-467f, mmu-mir-466j, mmu-mir-467g, mmu-mir-467h, mmu-mir-1195, hsa-mir-548e, hsa-mir-548j, hsa-mir-1285-1, hsa-mir-1285-2, hsa-mir-1289-1, hsa-mir-1289-2, hsa-mir-548k, hsa-mir-1299, hsa-mir-548l, hsa-mir-1302-1, hsa-mir-1302-2, hsa-mir-1302-3, hsa-mir-1302-4, hsa-mir-1302-5, hsa-mir-1302-6, hsa-mir-1302-7, hsa-mir-1302-8, hsa-mir-548f-1, hsa-mir-548f-2, hsa-mir-548f-3, hsa-mir-548f-4, hsa-mir-548f-5, hsa-mir-1255a, hsa-mir-548g, hsa-mir-548n, hsa-mir-548m, hsa-mir-548o, hsa-mir-1268a, hsa-mir-548h-1, hsa-mir-548h-2, hsa-mir-548h-3, hsa-mir-548h-4, hsa-mir-548p, hsa-mir-548i-1, hsa-mir-548i-2, hsa-mir-548i-3, hsa-mir-548i-4, hsa-mir-1255b-1, hsa-mir-1255b-2, mmu-mir-1906-1, hsa-mir-1972-1, hsa-mir-548q, mmu-mir-466m, mmu-mir-466o, mmu-mir-467a-2, mmu-mir-467a-3, mmu-mir-466c-2, mmu-mir-467a-4, mmu-mir-466b-4, mmu-mir-467a-5, mmu-mir-466b-5, mmu-mir-467a-6, mmu-mir-466b-6, mmu-mir-467a-7, mmu-mir-466b-7, mmu-mir-467a-8, mmu-mir-467a-9, mmu-mir-467a-10, mmu-mir-466p, mmu-mir-466n, mmu-mir-466b-8, hsa-mir-3116-1, hsa-mir-3116-2, hsa-mir-3118-1, hsa-mir-3118-2, hsa-mir-3118-3, hsa-mir-548s, hsa-mir-378b, hsa-mir-466, hsa-mir-548t, hsa-mir-548u, hsa-mir-548v, hsa-mir-3156-1, hsa-mir-3118-4, hsa-mir-3174, hsa-mir-3179-1, hsa-mir-3179-2, hsa-mir-3179-3, hsa-mir-548w, hsa-mir-3156-2, hsa-mir-3156-3, hsa-mir-548x, mmu-mir-3470a, mmu-mir-3470b, mmu-mir-3471-1, mmu-mir-3471-2, hsa-mir-378c, hsa-mir-1972-2, hsa-mir-1302-9, hsa-mir-1302-10, hsa-mir-1302-11, mmu-mir-1906-2, hsa-mir-3683, hsa-mir-3690-1, hsa-mir-548y, hsa-mir-548z, hsa-mir-548aa-1, hsa-mir-548aa-2, hsa-mir-548o-2, hsa-mir-1268b, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-548h-5, hsa-mir-548ab, hsa-mir-378f, hsa-mir-378g, hsa-mir-548ac, hsa-mir-548ad, hsa-mir-548ae-1, hsa-mir-548ae-2, hsa-mir-548ag-1, hsa-mir-548ag-2, hsa-mir-548ah, hsa-mir-378h, hsa-mir-548ai, hsa-mir-548aj-1, hsa-mir-548aj-2, hsa-mir-548x-2, hsa-mir-548ak, hsa-mir-548al, hsa-mir-378i, hsa-mir-548am, hsa-mir-548an, mmu-mir-28c, mmu-mir-378b, mmu-mir-28b, hsa-mir-548ao, hsa-mir-548ap, mmu-mir-466q, hsa-mir-548aq, hsa-mir-548ar, hsa-mir-548as, hsa-mir-548at, hsa-mir-548au, hsa-mir-548av, hsa-mir-548aw, hsa-mir-548ax, hsa-mir-378j, mmu-mir-378c, mmu-mir-378d, hsa-mir-548ay, hsa-mir-548az, hsa-mir-3690-2, mmu-mir-290b, hsa-mir-548ba, hsa-mir-548bb, hsa-mir-3179-4, mmu-mir-466c-3, hsa-mir-548bc, mmu-mir-1271
The functional networks of miR-92b (PRdmiR, mir-25 family, derived from GC rich tandem repeats), miR-28 (RdmiR, mir-28 family, derived from LINE), miR-151 (RdmiR, mir-28 family, derived from LINE), miR-421 (RdmiR, mir-95 family, derived from LINE), miR-1271 (RdmiR, mir-1271 family, derived from LINE), miR-340 (RdmiR, mir-340 family, derived from DNA transportable element) and miR-378 (RdmiR, mir-378 family, derived from SINE) have been reconstructed (Figure 8). [score:1]
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[+] score: 1
170 hsa-miR-34b2.061.00E-04 hsa-miR-30a-5p−2.130 hsa-miR-34c2.022.00E-04 hsa-miR-95−2.161.00E-04 hsa-miR-4552.063.00E-04 hsa-miR-378−2.081.00E-04 hsa-miR-91.993.00E-04 hsa-miR-218−1.881.00E-04 hsa-miR-2961.933.00E-04 hsa-miR-368−2.002.00E-04 hsa-miR-3012.023.00E-04 hsa-miR-363−1.832.00E-04 hsa-miR-130b1.973.00E-04 hsa-miR-128a−1.904.00E-04 hsa-miR-196b1.934.00E-04 hsa-miR-655−1.946.00E-04 hsa-miR-200a1. [score:1]
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