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38 publications mentioning ath-MIR160a

Open access articles that are associated with the species Arabidopsis thaliana and mention the gene name MIR160a. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 50
The downregulation of ARFs by miR160 during imbibition indicates possible auxin-ABA crosstalk during germination since ABA became downregulated in over expressing miR160 plants [15] and ARFs are known to be involved in auxin signalling. [score:9]
This consistent upregulation of miR160 led to the consistent downregulation of its targets ARF10/ 16/ 17 (Fig.   4e, f, g) during all germination conditions. [score:9]
The expressions of ARF10 (Fig.   4e), ARF 16 (Fig.   4f) and ARF 17 (Fig.   4g) as the target of miR160 were highly downregulated in all germination stages compare to DS. [score:8]
Mutation in ARF10, which is the target of miR160, results in developmental defects in seeds through induced expression of ABA responsive genes [15]. [score:7]
We observed the significant upregulation of miR160 during all of the germination conditions we used and found maximum upregulation at 24 h/4 °C followed by 24 h/RT (Fig.   2d). [score:7]
miR160 is involved in auxin signalling pathway during various plant growth and developmental processes 14, 15, 31 by negatively regulating its target transcription factors ARF10, ARF16 and ARF17 via hormonal crosstalk [32]. [score:5]
The highest expression of miR160a was observed at 24 h/4 °C (Fig.   2d), then gradually in decreasing order at 24 h/RT, 12 h/4 °C, 12 h/RT compared to DS. [score:2]
The other miRNAs for validation were miR165/166 (Fig.   2a), miR172a (Fig.   2b), miR390b (Fig.   2c), miR160a (Fig.   2d), miR156h (Fig.   2e), miR164a (Fig.   3a), miR169b (Fig.   3b), miR161.1 (Fig.   3c), miR399a (Fig.   3d), miR824 (Fig.   3f), miR834 (Fig.   3g), miR854 (Fig.   3h) and miR2112-5p (Fig.   3i). [score:1]
Earlier findings also indicate the role of miR160 in seed germination in rice [21] and Nelumbo nucifera [22]. [score:1]
This indicates the potential significant role of miR160 during early stages of seed germination irrespective of their stratification status. [score:1]
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2
[+] score: 47
Under N-starvation conditions, miR399, miR395, miR850, miR857, miR863, and miR827 were significantly down-regulated, whereas miR160, miR826, miR839, and miR846 were dramatically up-regulated. [score:7]
With the induction of miR160a, its targets ARF16 and ARF17 were down-regulated under N deficiency (Figure 5B). [score:6]
The expression of miR160a and its targets was determined under N -deficient conditions. [score:5]
For miR156, miR160, miR169, miR171, miR172, miR395, miR397, miR398, miR399, miR408, miR775, miR780.1, miR827, miR842, miR846, miR857, and miR2111, their targets have been predicted and most of them were validated previously (Table 2). [score:3]
These results implied that N deficiency induced expression of miR160, which then mediated the cleavage of ARF16 and promoted lateral root production in Arabidopsis. [score:3]
From these results, a conclusion was drawn that in response to N deficiency, plants may enhance their root systems by inducing expression of miR160 and decreasing those of miR167 and miR171 (Figure 5C). [score:3]
As expected, all miR160 over -expressing plants produced more lateral roots than wild type plants (Figure 5A). [score:3]
Our results suggested that changes in the expression levels of miR160, miR167, and miR171 may be important for the enhancement of plant root system under nitrogen deficiency. [score:3]
miR160 facilitates adventitious root outgrowth via repressing ARF17, whereas miR167 negatively regulates adventitious root initiation via cleavage of ARF6 and ARF8. [score:2]
miR160, miR167, and miR171 are involved in development of root system under N starvation coditions. [score:2]
In addition to the plasticity of lateral roots, miR160 and miR167 also play crucial roles in adventitious root development [44]. [score:2]
miR160, miR167, and miR171 are involved in the signaling pathways triggering root system development. [score:2]
Studies suggested that miR160 controls lateral root formation by mediating regulation of ARF16 [43]. [score:2]
Therefore, both the increased miR160 and decreased miR167 favor root growth under N-starvation conditions. [score:1]
The miR160a precursor sequence was inserted into the pOCA30 binary vector. [score:1]
To investigate whether the increased abundance of miR160 facilitates lateral root formation during N starvation, miR160a -overexpressing transgenic plants (35S::miR160a) were constructed. [score:1]
0048951.g005 Figure 5(A) More lateral roots in 35S::miR160a plants than WT (wild-type) plants. [score:1]
[1 to 20 of 17 sentences]
3
[+] score: 43
In addition to the miR393 -mediated regulation of auxin receptors, the expression of several Auxin Response Factors (ARFs) is known to be regulated by different miRNAs in plants (Khraiwesh et al. 2012; Liu et al. 2014) Among them are miR160 (targeting ARF10, ARF16 and ARF17) and miR167 (targeting ARF6 and ARF8) (Rhoades et al. 2002). [score:9]
Overexpression of miR160a or miR398b in transgenic rice results in increased H [2]O [2] accumulation at the infection site and up-regulation of defence gene expression (i. e. PR1 and PR10) and enhanced resistance to M. oryzae (Li et al. 2013). [score:8]
As previously mentioned, disease resistant transgenic rice have been produced by overexpressing either miR160, miR398 or miR7695 (Campo et al. 2013; Li et al. 2013), thus supporting the potential of MIR genes to prevent rice disease. [score:7]
miR160 targets Auxin Response Factors (ARFs) whereas miR398 targets Cu/Zn superoxide dismutase genes. [score:5]
It is also known that miR160a functions as a positive regulator of PAMP -induced callose deposition, whereas miR398 and miR773 negatively regulate PAMP -induced callose deposition and hence disease resistance to P. syringae (Fig.   1). [score:5]
Rice miRNAs for which a function in blast disease resistance has been demonstrated are miR160a, miR398b and miR7695 (Campo et al. 2013; Li et al. 2013). [score:3]
While auxin signalling pathway is regulated by miR160, miR167, miR390 and miR393, the JA biosynthetic pathway is under the control of miR319 and miR159, and miR159 regulate the ABA signalling pathway (Curaba et al. 2014). [score:3]
MiR160 targets Auxin Response Factors (ARFs) involved in auxin signalling. [score:2]
In rice, the involvement of miR160, miR398 and miR7695 (in bold) in PTI responses is documented (Campo et al. 2013; Li et al. 2013). [score:1]
[1 to 20 of 9 sentences]
4
[+] score: 37
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR157d, ath-MIR158a, ath-MIR159a, ath-MIR160b, ath-MIR160c, ath-MIR161, ath-MIR162a, ath-MIR162b, ath-MIR163, ath-MIR164a, ath-MIR164b, ath-MIR165a, ath-MIR165b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR169a, ath-MIR170, ath-MIR172a, ath-MIR172b, ath-MIR173, ath-MIR159b, ath-MIR319a, ath-MIR319b, ath-MIR167d, ath-MIR169b, ath-MIR169c, ath-MIR169d, ath-MIR169e, ath-MIR169f, ath-MIR169g, ath-MIR169h, ath-MIR169i, ath-MIR169j, ath-MIR169k, ath-MIR169l, ath-MIR169m, ath-MIR169n, ath-MIR171b, ath-MIR172c, ath-MIR172d, ath-MIR391, ath-MIR395a, ath-MIR395b, ath-MIR395c, ath-MIR395d, ath-MIR395e, ath-MIR395f, ath-MIR397a, ath-MIR397b, ath-MIR398a, ath-MIR398b, ath-MIR398c, ath-MIR399a, ath-MIR399b, ath-MIR399c, ath-MIR399d, ath-MIR399e, ath-MIR399f, ath-MIR400, ath-MIR408, ath-MIR156g, ath-MIR156h, ath-MIR158b, ath-MIR159c, ath-MIR319c, ath-MIR164c, ath-MIR167c, ath-MIR172e, ath-MIR447a, ath-MIR447b, ath-MIR447c, ath-MIR773a, ath-MIR775, ath-MIR822, ath-MIR823, ath-MIR826a, ath-MIR827, ath-MIR829, ath-MIR833a, ath-MIR837, ath-MIR841a, ath-MIR842, ath-MIR843, ath-MIR845a, ath-MIR848, ath-MIR852, ath-MIR824, ath-MIR854a, ath-MIR854b, ath-MIR854c, ath-MIR854d, ath-MIR857, ath-MIR864, ath-MIR2111a, ath-MIR2111b, ath-MIR773b, ath-MIR841b, ath-MIR854e, ath-MIR833b, ath-MIR156i, ath-MIR156j, ath-MIR826b
Both sequencing data and quantitative RT-PCR results indicated that miR160 was induced, whereas miR167 was suppressed, by –C, –N, and –S, implying that under nutrient deficiency conditions, or at least under –C, –N, and –S conditions, plants regulate root development -associated miRNAs to modulate their root systems. [score:5]
Nitrogen treatment led to a reduction in miR167 and the elevation of its target, ARF8, which mediates the balance between lateral root initiation and emergence 58. miR160 controls root cap formation, lateral root number and primary root length by mediating the cleavage of its targets, ARF10 and ARF16 40. [score:5]
In contrast, the targets of miR160, ARF10 and ARF16, were repressed, whereas the target of miR167, ARF8, was induced by –C, –N, and –S (Fig. 3F,I). [score:5]
In addition, some miRNAs were positively correlated with their targets, such as miR395- APS3 in –S, miR397- LAC2 in –C, and miR160- ARF17 in –C and –S. [score:3]
The only miRNA family induced commonly by –C, –N, and –S was miR160, which is involved in the auxin response by targeting ARF10, ARF16 and ARF17 39 40. [score:3]
miR160 and miR167 are involved in the auxin pathway by targeting ARF genes. [score:3]
The analysis of relative root growth revealed that miR160 overexpression facilitated root growth under –C and –S conditions (Fig. 4B). [score:3]
As expected, ectopic or disrupted expression of miR160 altered the root systems under both nutrient sufficiency and deficiency conditions (Fig. 4A). [score:3]
For construction of STTM160 transgenic plants, a short tandem target mimic for miR160 was designed according to a previous report 60. [score:3]
Under –S conditions, the lateral roots of miR160-ox and miR395a plants were longer than the other plants. [score:1]
The construction of miR160a-ox and miR395a-ox transgenic plants was reported previously 21 41. [score:1]
To investigate the functions of nutrient-responsive miRNAs in nutrient starvation adaptation, miRNA overexpression plants (miR160a-ox, miR395a-ox, and miR399b-ox) and miR160 suppression plants (STTM160) were used to evaluate phenotypes in nutrient starvation conditions. [score:1]
miR160 was induced whereas miR167a/b was repressed by –C, –N, and –S. [score:1]
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5
[+] score: 36
It is also amazing that miR160 in wheat was up-regulated by 40°C for 2 h in the heat tolerant genotype TAM107 (Xin et al., 2010), but down-regulated with the up-regulation of its putative target HSP70 by 42°C for 2 h in another heat tolerant cultivar HD2985 (Kumar et al., 2014). [score:12]
After heat stress, miR160 was up-regulated in Arabidopsis (Zhong et al., 2013), barley (Kruszka et al., 2014) and celery (Apium graveolens; Li et al., 2014a), but down-regulated in cassava (Ballen-Taborda et al., 2013) and Chinese white poplar (Chen et al., 2012). [score:7]
miR160 as well as miR169 in rice showed differential expression in roots and shoots under heat stress, suggesting the different regulation of the target genes by heat in this two different tissues (Sailaja et al., 2014). [score:6]
An increase in barley miR160a during heat stress down-regulated the expression level of ARF17 and ARF13, which might affect shoot morphology and root growth (Kruszka et al., 2014). [score:6]
The regulation of miR160 family by heat is quite different in various species, although they all target auxin response factors (ARFs). [score:4]
Interestingly, the splicing of introns hosting miR160a and miR5175a in barley was heat induced, but the roles of these spliced isoforms in response to heat stress are unclear (Kruszka et al., 2014). [score:1]
[1 to 20 of 6 sentences]
6
[+] score: 18
10 conserved miRNA families had highly increased expression (log [2]fold > 2) in response to Xam infection (Figure 2a), including miR160 and miR167 families which are both known to target auxin response factors (ARFs) [13] and miR393 and miR390 families which are also known to regulate auxin signaling [13]. [score:6]
Predicted targets for miR160 in cassava were ARF-like genes whereas predicted targets for miR167 included phosphatases and peptidases. [score:5]
It has been found that ARF binding sites are over-represented in auxin-related miRNA families like miR160 and miR167, thus forming a regulatory loop [40]. [score:2]
miRNAs induction was found to be involved in regulating auxin signaling: miR160, miR167, miR390 and miR393 [11, 13]. [score:2]
The repertoire of known bacterial-responsive miRNAs has increased and includes several families known to regulate hormone signaling, such as miR160, miR167 and miR390 involved in auxin signaling, miR159 involved in ABA signaling and miR319 involved in jasmonic acid signaling [13- 15]. [score:2]
In our results miR160 was also the family having the most ARF binding sites identified (10 out of 44 ARF binding sites were found in miR160 promoter regions). [score:1]
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7
[+] score: 15
In A. thaliana, mRNAs encoding two out of the fours which have no CTD have also been identified as targets of small RNAs: 3 is the target of AtTAS3a-c and 17 is the target of miR160 [50]. [score:7]
For example, in A. thaliana the expression pattern of both 6 and 8 (involved in female and male reproductive organ development) is controlled by miR167, with miRNA160 also involved in the control of expression in P. patens and A. thaliana as well as in S. moellendorffii, suggesting a conserved mechanism of post-transcriptional regulation [52, 54]. [score:7]
Regulation ofs by miRNA in A. thaliana can be considered as auxin-independent because auxin treatment does not alter appreciably miR160, miR164, and miR167 accumulation, at least in seedlings [49]. [score:1]
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8
[+] score: 15
Turner M. Nizampatnam N. R. Baron M. Coppin S. Damodaran S. Adhikari S. Arunachalam S. P. Yu O. Subramanian S. Ectopic expression of miR160 results in auxin hypersensitivity, cytokinin hyposensitivity, and inhibition of symbiotic nodule development in soybean Plant Physiol. [score:6]
Thus, overexpressing miR160, which targets the ARF10/16/17 family of auxin signaling repressors enhanced auxin sensitivity and drastically reduced nodule primordia development in soybean [76] and Medicago [78]. [score:6]
Bustos-Sanmamed P. Mao G. Deng Y. Elouet M. Khan G. A. Bazin J. Turner M. Subramanian S. Yu O. Crespi M. Overexpression of miR160 affects root growth and nitrogen-fixing nodule number in Medicago truncatula Funct. [score:3]
[1 to 20 of 3 sentences]
9
[+] score: 15
Given the presence of miRs targeting transcription factor families such as SPL (miR156/miR157), MYB/TCP (miR159, miR319), ARF (miR160, miR167), AP2 (miR172), and GRF (miR396) there can be no doubt that miRs modulate the expression of many transcription factors during later stages of pollen development. [score:6]
The remaining microRNAs target genes involved in hormone response like AUXIN-RESPONSE FACTORs (miR160 and miR167, 5 reads), genes involved in general metabolism or those having no identified targets. [score:5]
Mir160 may also be an important contributor to transcriptomic changes in pollen development, playing a part in auxin-regulated development through the elimination of ARF16 and ARF17 transcripts after pollen mitosis II. [score:3]
Derivation of the same mature microRNA sequence from two or more independent genomic loci is common in plant microRNAs, for example miR160. [score:1]
[1 to 20 of 4 sentences]
10
[+] score: 13
Our miRNA arrays indicated that the up-regulation of miR160a might have resulted in the expression of the phenotype observed in the present study. [score:6]
miRNA -mediated signaling is also involved in the development of various tissues; several miRNA families such as miR160, miR164, miR167, and miR390 have been demonstrated to be involved in root cap formation and lateral root development [42]. [score:3]
Since the lateral root formation caused by miR160 was similar to the morphology of BR -treated seedlings, we suspect miR160 might play an important role in lateral root development in BR-supplied plants. [score:2]
Some miRNAs, such as miR159 and miR160, play roles during early development stages including seed germination. [score:2]
[1 to 20 of 4 sentences]
11
[+] score: 13
Unlike some miRNAs, such as miR160, miR167 and miR393, which directly target and regulate the expression of key components of the auxin response pathway, the miR165/166 targets themselves are not major components of hormone response pathways but they regulate the transcription of important components of hormone pathways. [score:10]
Proper regulation of Auxin Response Factor 10 (ARF10), ARF16 and ARF17 by miR160 is required for both shoot and root development [42– 44]. [score:3]
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12
[+] score: 11
b Expression profiles of (i) miRNA159a and (ii) miRNA160a-5p and their targets genes, which have been shown to have a role in regulation during seed germination. [score:6]
For example, miR159, miR160 and their confirmed target genes that encode MYB and auxin responsive factor TFs (Fig.   6b(i), (ii)). [score:3]
For example, miRNA159 and miRNA160 interact with ABA/GA signalling pathways during seed germination in Arabidopsis [19, 21]. [score:1]
Both miR159 and miR160 have a functional role during seed germination via interactions with ABA [3– 5]. [score:1]
[1 to 20 of 4 sentences]
13
[+] score: 11
While the expressions of 14 families (miR156/miR157, miR158, miR160, miR162, miR165/miR166, miR168, miR169, miR171, miR390, miR393, miR394, miR396, miR398, and miR399) were dramatically reduced, 3 families (miR159, miR167, and miR172) were up-regulated in CsCl -treated seedlings. [score:6]
As shown in the radial chart in Fig 4C, expression of the miR157, miR160, miR165, miR168, miR171, miR319, and miR403 families was decreased by around 80% to 140% in CsCl -treated seedlings. [score:3]
At first glance, in accordance with the reduced levels of miR160 and miR166 (Fig 3B), the accumulations of pri-miR160a (~ 3.5-fold) and pri-miR166a (~ 3-fold) seemingly reflected the interference with the miRNA processing pathway. [score:1]
Conversely, the accumulated levels of ARF17 and REV should be monitored due to the reduction of miR160 and miR166. [score:1]
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14
[+] score: 9
Our results imply that the up-regulation of miR160, by decreasing the levels of ARF 10, might contribute to enhance A. thaliana responses induced by LPS. [score:4]
miR160 regulates genes involved in the auxin signaling pathway, including auxin response factors and auxin receptors. [score:2]
Furthermore, miR160 was highly induced in Arabidopsis leaves collected at 1 and 3 h post-inoculation with the hrcC mutant of P. syringae pv. [score:1]
miRNA160 was reported to positively induce Flg22 induced callose deposition [26]. [score:1]
Another category of stress-responsive miRNAs identified in this study is miR160 which was shown to be induced in both callus and leaf tissues following LPS treatment (Tables  1 and 2). [score:1]
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15
[+] score: 8
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR159a, ath-MIR160b, ath-MIR160c, ath-MIR164a, ath-MIR164b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR168a, ath-MIR168b, ath-MIR171a, ath-MIR172a, ath-MIR172b, ath-MIR159b, ath-MIR319a, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR164a, osa-MIR164b, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR171a, ath-MIR167d, ath-MIR172c, ath-MIR172d, ath-MIR393a, ath-MIR393b, ath-MIR396a, ath-MIR396b, ath-MIR398a, osa-MIR393a, osa-MIR396a, osa-MIR396b, osa-MIR396c, osa-MIR398a, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR164c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR319a, osa-MIR160e, osa-MIR160f, osa-MIR164c, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR168a, osa-MIR168b, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR393b, osa-MIR172d, osa-MIR167j, osa-MIR166m, osa-MIR166j, osa-MIR437, osa-MIR396e, osa-MIR444a, osa-MIR528, osa-MIR531a, osa-MIR1425, osa-MIR444b, osa-MIR444c, osa-MIR444d, osa-MIR444e, osa-MIR444f, osa-MIR531b, osa-MIR1862a, osa-MIR1862b, osa-MIR1862c, osa-MIR1873, osa-MIR1862d, osa-MIR1862e, osa-MIR396f, osa-MIR396g, osa-MIR396h, osa-MIR396d, osa-MIR1862f, osa-MIR1862g, ath-MIR5021, osa-MIR5072, osa-MIR5077, ath-MIR156i, ath-MIR156j, osa-MIR531c
Moreover, we found that in A. thaliana ARF6 and ARF8 were targeted by miR167 and ARF10, ARF16, and ARF17 by miR160 and in O. sativa ARF16 by miR160; ARF6, ARF8 were targeted by miR167. [score:5]
In C. borivilianum ARF18 and ARF17 were found to be targeted by miR160; and ARF12 by miR167. [score:3]
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16
[+] score: 8
Serrated and hyponastic leaves were seen in MIM160 plants (Figure 2), in agreement with the phenotype of plants that express non-targetable versions of ARF10 or ARF17, two of the three miR160 targets [44], [45]. [score:7]
MYB33, MYB65, MYB81, MYB97, MYB101, MYB104, MYB120, DUO1 1, 2, 3 MIM160 miR160 Smaller plants, with serrated and curled upward leaves. [score:1]
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17
[+] score: 6
Overexpression of miR156 and miR159 resulted in late-flowering [21, 24], and overexpression of miR160 resulted in increased lateral rooting [25]. [score:5]
Similarly, miRNAs responsive to bacterial (miR160, miR167, miR393, miR396, miR398 and miR825) and viral infections (miR156 and miR164) were not altered in the OE lines [33- 35]. [score:1]
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18
[+] score: 6
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR157a, ath-MIR157b, ath-MIR157c, ath-MIR157d, ath-MIR159a, ath-MIR160b, ath-MIR160c, ath-MIR165a, ath-MIR165b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR169a, ath-MIR172a, ath-MIR172b, ath-MIR159b, ath-MIR319a, ath-MIR319b, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR169a, ath-MIR167d, ath-MIR169b, ath-MIR169c, ath-MIR169d, ath-MIR169e, ath-MIR169f, ath-MIR169g, ath-MIR169h, ath-MIR169i, ath-MIR169j, ath-MIR169k, ath-MIR169l, ath-MIR169m, ath-MIR169n, ath-MIR172c, ath-MIR172d, ath-MIR394a, ath-MIR394b, ath-MIR396a, ath-MIR396b, osa-MIR394, osa-MIR396a, osa-MIR396b, osa-MIR396c, ath-MIR403, ath-MIR408, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR319c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR319a, osa-MIR319b, osa-MIR160e, osa-MIR160f, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR169b, osa-MIR169c, osa-MIR169d, osa-MIR169e, osa-MIR169f, osa-MIR169g, osa-MIR169h, osa-MIR169i, osa-MIR169j, osa-MIR169k, osa-MIR169l, osa-MIR169m, osa-MIR169n, osa-MIR169o, osa-MIR169p, osa-MIR169q, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR408, osa-MIR172d, osa-MIR167j, osa-MIR166m, osa-MIR166j, ath-MIR414, osa-MIR414, osa-MIR396e, ath-MIR856, ath-MIR858a, osa-MIR169r, osa-MIR396f, ath-MIR2111a, ath-MIR2111b, osa-MIR396g, osa-MIR396h, osa-MIR396d, ath-MIR858b, ath-MIR156i, ath-MIR156j
Similarly, miR160 targets auxin response factor 10 (ARF) and miR414 targets Zinc finger related protein (Additional file 2 Table S1). [score:5]
miR156, miR159, miR167, miR319, miR396 and miR172 possessed 5, 8, 10, 8, 7 and 6 members respectively whereas other miRNA families such as miR157, miR160, miR169, miR858, miR894, miR2111 etc. [score:1]
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[+] score: 6
Both miR393a and miR160 are auxin pathway regulators, and they were significantly up-regulated after TCV infection. [score:5]
The abundance of miR160, miR168, miR170, miR393, miR395, miR408 and miR850 were specifically increased. [score:1]
[1 to 20 of 2 sentences]
20
[+] score: 6
Ectopic expression of miR160 results in auxin hypersensitivity, cytokinin hyposensitivity, and inhibition of symbiotic nodule development in soybean. [score:6]
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[+] score: 6
Besides miR393, two other miRNA families, miR160 and miR167, are upregulated following PsDC3000 inoculation and target members of auxin-response factors (ARF) [14]. [score:6]
[1 to 20 of 1 sentences]
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[+] score: 6
Interestingly, miR160 is implicated in the regulation of auxin metabolism (Rhoades et al., 2002; Mallory et al., 2005); the mutants expressing a miR160-resistant form of ARF10 are hypersensitive to ABA (Liu et al., 2007a), therefore suggesting a point of cross-talk between ABA and auxin in imbibed seeds. [score:4]
Regarding the control exerted by miRNAs on phytohormones, it has been shown in Arabidopsis that auxin metabolism is controlled by at least four conserved miRNA families (miR160, miR167, miR390, and miR393), which mainly exert control by regulating ARF proteins (i. e., ARF6, ARF8, ARF10, ARF16, and 17) (Rhoades et al., 2002; Mallory et al., 2005; Marin et al., 2010; Win dels and Vazquez, 2011; Kinoshita et al., 2012). [score:2]
[1 to 20 of 2 sentences]
23
[+] score: 5
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR159a, ath-MIR160b, ath-MIR160c, ath-MIR162a, ath-MIR162b, ath-MIR164a, ath-MIR164b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR169a, ath-MIR171a, ath-MIR172a, ath-MIR172b, ath-MIR159b, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR162a, osa-MIR164a, osa-MIR164b, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR169a, osa-MIR171a, ath-MIR167d, ath-MIR169b, ath-MIR169c, ath-MIR169d, ath-MIR169e, ath-MIR169f, ath-MIR169g, ath-MIR169h, ath-MIR169i, ath-MIR169j, ath-MIR169k, ath-MIR169l, ath-MIR169m, ath-MIR169n, ath-MIR171b, ath-MIR171c, ath-MIR172c, ath-MIR172d, ath-MIR393a, ath-MIR393b, ath-MIR394a, ath-MIR394b, ath-MIR395a, ath-MIR395b, ath-MIR395c, ath-MIR395d, ath-MIR395e, ath-MIR395f, osa-MIR393a, osa-MIR394, osa-MIR395b, osa-MIR395d, osa-MIR395e, osa-MIR395g, osa-MIR395h, osa-MIR395i, osa-MIR395j, osa-MIR395k, osa-MIR395l, osa-MIR395s, osa-MIR395t, osa-MIR395c, osa-MIR395a, osa-MIR395f, osa-MIR395u, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR164c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR160e, osa-MIR160f, osa-MIR162b, osa-MIR164c, osa-MIR164d, osa-MIR164e, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR169b, osa-MIR169c, osa-MIR169d, osa-MIR169e, osa-MIR169f, osa-MIR169g, osa-MIR169h, osa-MIR169i, osa-MIR169j, osa-MIR169k, osa-MIR169l, osa-MIR169m, osa-MIR169n, osa-MIR169o, osa-MIR169p, osa-MIR169q, osa-MIR171b, osa-MIR171c, osa-MIR171d, osa-MIR171e, osa-MIR171f, osa-MIR171g, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR171h, osa-MIR393b, osa-MIR172d, osa-MIR171i, osa-MIR167j, osa-MIR166m, osa-MIR166j, osa-MIR164f, zma-MIR156d, zma-MIR156f, zma-MIR156g, zma-MIR156b, zma-MIR156c, zma-MIR156e, zma-MIR156a, zma-MIR156h, zma-MIR156i, zma-MIR160a, zma-MIR160c, zma-MIR160d, zma-MIR160b, zma-MIR164a, zma-MIR164d, zma-MIR164b, zma-MIR164c, zma-MIR169a, zma-MIR169b, zma-MIR167a, zma-MIR167b, zma-MIR167d, zma-MIR167c, zma-MIR160e, zma-MIR166a, zma-MIR162, zma-MIR166h, zma-MIR166e, zma-MIR166i, zma-MIR166f, zma-MIR166g, zma-MIR166b, zma-MIR166c, zma-MIR166d, zma-MIR171a, zma-MIR171b, zma-MIR172a, zma-MIR172d, zma-MIR172b, zma-MIR172c, zma-MIR171d, zma-MIR171f, zma-MIR394a, zma-MIR394b, zma-MIR395b, zma-MIR395c, zma-MIR395a, zma-MIR156j, zma-MIR159a, zma-MIR159b, zma-MIR159c, zma-MIR159d, zma-MIR166k, zma-MIR166j, zma-MIR167e, zma-MIR167f, zma-MIR167g, zma-MIR167h, zma-MIR167i, zma-MIR169c, zma-MIR169f, zma-MIR169g, zma-MIR169h, zma-MIR169i, zma-MIR169k, zma-MIR169j, zma-MIR169d, zma-MIR169e, zma-MIR171c, zma-MIR171j, zma-MIR171e, zma-MIR171i, zma-MIR171g, zma-MIR172e, zma-MIR166l, zma-MIR166m, zma-MIR171k, zma-MIR171h, zma-MIR393a, zma-MIR156k, zma-MIR160f, osa-MIR528, osa-MIR529a, osa-MIR395m, osa-MIR395n, osa-MIR395o, osa-MIR395p, osa-MIR395q, osa-MIR395v, osa-MIR395w, osa-MIR395r, ath-MIR827, osa-MIR529b, osa-MIR1432, osa-MIR169r, osa-MIR827, osa-MIR2118a, osa-MIR2118b, osa-MIR2118c, osa-MIR2118d, osa-MIR2118e, osa-MIR2118f, osa-MIR2118g, osa-MIR2118h, osa-MIR2118i, osa-MIR2118j, osa-MIR2118k, osa-MIR2118l, osa-MIR2118m, osa-MIR2118n, osa-MIR2118o, osa-MIR2118p, osa-MIR2118q, osa-MIR2118r, osa-MIR2275a, osa-MIR2275b, zma-MIR2118a, zma-MIR2118b, zma-MIR2118c, zma-MIR2118d, zma-MIR2118e, zma-MIR2118f, zma-MIR2118g, zma-MIR2275a, zma-MIR2275b, zma-MIR2275c, zma-MIR2275d, zma-MIR156l, zma-MIR159e, zma-MIR159f, zma-MIR159g, zma-MIR159h, zma-MIR159i, zma-MIR159j, zma-MIR159k, zma-MIR160g, zma-MIR164e, zma-MIR164f, zma-MIR164g, zma-MIR164h, zma-MIR166n, zma-MIR167j, zma-MIR169l, zma-MIR169m, zma-MIR169n, zma-MIR169o, zma-MIR169p, zma-MIR169q, zma-MIR169r, zma-MIR171l, zma-MIR171m, zma-MIR171n, zma-MIR393b, zma-MIR393c, zma-MIR395d, zma-MIR395e, zma-MIR395f, zma-MIR395g, zma-MIR395h, zma-MIR395i, zma-MIR395j, zma-MIR395k, zma-MIR395l, zma-MIR395m, zma-MIR395n, zma-MIR395o, zma-MIR395p, zma-MIR482, zma-MIR528a, zma-MIR528b, zma-MIR529, zma-MIR827, zma-MIR1432, osa-MIR395x, osa-MIR395y, osa-MIR2275c, osa-MIR2275d, ath-MIR156i, ath-MIR156j
AUXIN RESPONSIVE FACTORs (ARFs) are a class of targets of miRNA160 families. [score:3]
The sensitivity of seeds to ABA that is vital to the termination of seed maturation program, an essential change to increase the competence of seeds for germination, is regulated by conserved miRNA160. [score:2]
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[+] score: 5
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR159a, ath-MIR160b, ath-MIR160c, ath-MIR162a, ath-MIR162b, ath-MIR164a, ath-MIR164b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR168a, ath-MIR168b, ath-MIR169a, ath-MIR172a, ath-MIR172b, ath-MIR159b, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR162a, osa-MIR164a, osa-MIR164b, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR169a, ath-MIR167d, ath-MIR169b, ath-MIR169c, ath-MIR169d, ath-MIR169e, ath-MIR169f, ath-MIR169g, ath-MIR169h, ath-MIR169i, ath-MIR169j, ath-MIR169k, ath-MIR169l, ath-MIR169m, ath-MIR169n, ath-MIR172c, ath-MIR172d, ath-MIR395a, ath-MIR395b, ath-MIR395c, ath-MIR395d, ath-MIR395e, ath-MIR395f, ath-MIR396a, ath-MIR396b, ath-MIR399a, ath-MIR399b, ath-MIR399c, ath-MIR399d, ath-MIR399e, ath-MIR399f, osa-MIR395b, osa-MIR395d, osa-MIR395e, osa-MIR395g, osa-MIR395h, osa-MIR395i, osa-MIR395j, osa-MIR395k, osa-MIR395l, osa-MIR395s, osa-MIR395t, osa-MIR395c, osa-MIR395a, osa-MIR395f, osa-MIR395u, osa-MIR396a, osa-MIR396b, osa-MIR396c, osa-MIR399a, osa-MIR399b, osa-MIR399c, osa-MIR399d, osa-MIR399e, osa-MIR399f, osa-MIR399g, osa-MIR399h, osa-MIR399i, osa-MIR399j, osa-MIR399k, ath-MIR408, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR164c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR160e, osa-MIR160f, osa-MIR162b, osa-MIR164c, osa-MIR164d, osa-MIR164e, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR168a, osa-MIR168b, osa-MIR169b, osa-MIR169c, osa-MIR169d, osa-MIR169e, osa-MIR169f, osa-MIR169g, osa-MIR169h, osa-MIR169i, osa-MIR169j, osa-MIR169k, osa-MIR169l, osa-MIR169m, osa-MIR169n, osa-MIR169o, osa-MIR169p, osa-MIR169q, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR171h, osa-MIR408, osa-MIR172d, osa-MIR167j, osa-MIR166m, osa-MIR166j, osa-MIR164f, zma-MIR156d, zma-MIR156f, zma-MIR156g, zma-MIR156b, zma-MIR156c, zma-MIR156e, zma-MIR156a, zma-MIR156h, zma-MIR156i, zma-MIR160a, zma-MIR160c, zma-MIR160d, zma-MIR160b, zma-MIR164a, zma-MIR164d, zma-MIR164b, zma-MIR164c, zma-MIR169a, zma-MIR169b, zma-MIR167a, zma-MIR167b, zma-MIR167d, zma-MIR167c, zma-MIR160e, zma-MIR166a, zma-MIR162, zma-MIR166h, zma-MIR166e, zma-MIR166i, zma-MIR166f, zma-MIR166g, zma-MIR166b, zma-MIR166c, zma-MIR166d, zma-MIR172a, zma-MIR172d, zma-MIR172b, zma-MIR172c, osa-MIR396e, zma-MIR395b, zma-MIR395c, zma-MIR395a, zma-MIR396b, zma-MIR396a, zma-MIR399a, zma-MIR399c, zma-MIR399b, zma-MIR399d, zma-MIR399e, zma-MIR399f, zma-MIR156j, zma-MIR159a, zma-MIR159b, zma-MIR159c, zma-MIR159d, zma-MIR166k, zma-MIR166j, zma-MIR167e, zma-MIR167f, zma-MIR167g, zma-MIR167h, zma-MIR167i, zma-MIR168a, zma-MIR168b, zma-MIR169c, zma-MIR169f, zma-MIR169g, zma-MIR169h, zma-MIR169i, zma-MIR169k, zma-MIR169j, zma-MIR169d, zma-MIR169e, zma-MIR172e, zma-MIR166l, zma-MIR166m, zma-MIR171h, zma-MIR408a, zma-MIR156k, zma-MIR160f, osa-MIR529a, osa-MIR395m, osa-MIR395n, osa-MIR395o, osa-MIR395p, osa-MIR395q, osa-MIR395v, osa-MIR395w, osa-MIR395r, osa-MIR529b, osa-MIR169r, osa-MIR396f, zma-MIR396c, zma-MIR396d, osa-MIR2118a, osa-MIR2118b, osa-MIR2118c, osa-MIR2118d, osa-MIR2118e, osa-MIR2118f, osa-MIR2118g, osa-MIR2118h, osa-MIR2118i, osa-MIR2118j, osa-MIR2118k, osa-MIR2118l, osa-MIR2118m, osa-MIR2118n, osa-MIR2118o, osa-MIR2118p, osa-MIR2118q, osa-MIR2118r, osa-MIR2275a, osa-MIR2275b, zma-MIR2118a, zma-MIR2118b, zma-MIR2118c, zma-MIR2118d, zma-MIR2118e, zma-MIR2118f, zma-MIR2118g, zma-MIR2275a, zma-MIR2275b, zma-MIR2275c, zma-MIR2275d, osa-MIR396g, osa-MIR396h, osa-MIR396d, zma-MIR156l, zma-MIR159e, zma-MIR159f, zma-MIR159g, zma-MIR159h, zma-MIR159i, zma-MIR159j, zma-MIR159k, zma-MIR160g, zma-MIR164e, zma-MIR164f, zma-MIR164g, zma-MIR164h, zma-MIR166n, zma-MIR167j, zma-MIR169l, zma-MIR169m, zma-MIR169n, zma-MIR169o, zma-MIR169p, zma-MIR169q, zma-MIR169r, zma-MIR395d, zma-MIR395e, zma-MIR395f, zma-MIR395g, zma-MIR395h, zma-MIR395i, zma-MIR395j, zma-MIR395k, zma-MIR395l, zma-MIR395m, zma-MIR395n, zma-MIR395o, zma-MIR395p, zma-MIR396e, zma-MIR396f, zma-MIR396g, zma-MIR396h, zma-MIR399g, zma-MIR399h, zma-MIR399i, zma-MIR399j, zma-MIR408b, zma-MIR529, osa-MIR395x, osa-MIR395y, osa-MIR2275c, osa-MIR2275d, ath-MIR156i, ath-MIR156j
For example, miR167 targets four AUXIN RESPONSE FACTOR (ARF) genes, and miR160 targets six ARF genes. [score:5]
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[+] score: 5
In rice, osa-miR160, osa-miR164 and osa-miR172 are co-expressed with their targets in root, leaf, seedling, endosperm and embryo [46]. [score:5]
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[+] score: 4
Moreover, the balance between repressing (ARF17) and activating (ARF6 and ARF8) factors is post-transcriptionally regulated by miR160 and miR167 (Gutierrez et al., 2009). [score:2]
Because ARF17 is a target of miRNA160, a selection of other ARFs regulated by miRNAs were investigated and AR numbers were recorded in ARF6, 8, 10, and 16 mutants. [score:2]
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[+] score: 4
Besides miR393, two other miRNA families, miR160 and miR167, are upregulated following Pseudomonas syringae pv. [score:4]
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[+] score: 4
The role of floral organs in carpels, an Arabidopsis loss-of-function mutation in MicroRNA160a, in organogenesis and the mechanism regulating its expression. [score:4]
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[+] score: 4
miR160 and miR167 each target mRNAs encoding members of the ARF family of transcription factors [19]. [score:3]
Two additional miRNA families, miR160 and miR167, were significantly elevated at 3 hr p. i. by 5-fold and 6-fold, respectively (Figure 3C). [score:1]
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[+] score: 3
Among the 30 miRNA families detected at 2dpi, 10 (miR160, miR161, miR167, miR171, miR172, miR390, miR394, miR396, miR398 and miR408) displayed contrasting expression levels between viruses. [score:3]
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[+] score: 3
Bustos-Sanmamed P. Mao G. Deng Y. Elouet M. Khan G. A. Bazin J. Turner M. Subramanian S. Yu O. Crespi M. Overexpression of miR160 affects root growth and nitrogen-fixing nodule number in Medicago truncatula Funct. [score:3]
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[+] score: 3
The involvement of miRNAs as key regulators of flowering time (miR159, miR172, miR156, and miR171), hormone signaling (miR159, miR160, miR167, miR164, and miR393), or shoot and root development (miR164), was reviewed by (Wang and Li, 2007). [score:3]
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[+] score: 3
Based on A. thaliana annotation, miRNA target genes were found for several conserved miRNAs in hybrid yellow poplar (Table S4): ARF10 (miR160), CYP96A1 (miR162), NAC (miR164), PHB and DNA -binding factor (miR165/166), NF-YA8 (miR169), SCARECROW transcription factor family protein (miR170/171), SNZ (miR172), MYB (miR319), GRF (miR396), copper ion binding (miR408), SPL11 (miR529) etc. [score:3]
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[+] score: 3
We also predicted several new miRNAs that are likely to respond to the TMV-Cg virus, including miR165 [36, 37], miR156 [34, 38], miR418, miR160 [36, 38], and miR393 [36, 37, 39]. [score:1]
We also predicted several new miRNAs that are likely to respond to high-salt conditions, including miR418, miR166 [36, 40], miR160 [36, 38], miR841 [41], miR169 [37, 42, 43]. [score:1]
The partial results are shown in Table  5, and the complete results are available in Additional file 5. Table 5 Top 5 prediction results for miRNAs responding to high-salt conditions and TMV-Cg stress Stress miRNA Score High-salt ath-miR418 0.932 ath-miR166 0.929 ath-miR160 0.908 ath-miR841 0.892 ath-miR169 0.816 TMV-Cg ath-miR165 1.000 ath-miR156 0.939 ath-miR418 0.932 ath-miR160 0.908 ath-miR8177 0.899 To our knowledge, most of the existing methods mentioned previously have not been implemented as publicly available software packages. [score:1]
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35
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Other miRNAs from this paper: ath-MIR160b, ath-MIR160c
ARF10 is targeted by MiR160, but mechanisms are different in different tissues. [score:2]
In A. thaliana, the genes AtLBD16, AtLBD17, AtLBD18 and AtLBD29 provide the necessary link between auxin signaling and regeneration 26, while Micro160 (MiR160) and AtARF10 have been identified as important regulators of shoot regeneration from in vitro cultures 46. [score:1]
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36
[+] score: 2
In contrast to Pp23SR loci, McrBC digestion had little to no effect on the amplification of Pp21SR12 regions nor on ppt- MIR160a or PpTAS3a. [score:1]
Similarly, ppt- MIR160a and PpTAS3a also had very low levels of 5 mC regardless of the context. [score:1]
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37
[+] score: 2
ARF10 has been shown to restrict the size of the stem cell niche in the distil root causing the differentiation of root cap cells, it is regulated by both IAA and miR160 [58]. [score:2]
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38
[+] score: 1
One group of miRNAs (miR160, miR167, miR390, miR393) is specifically related to auxin signalling (Zhang et al., 2011), which is linked to camalexin and glucosinolate biosynthesis. [score:1]
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