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28 publications mentioning ath-MIR168b

Open access articles that are associated with the species Arabidopsis thaliana and mention the gene name MIR168b. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 34
mir162 which target DCL1 mRNA and mir168 which target AGO1 mRNA reduced their expression in the roots of plants subjected to water deficit. [score:7]
Expression of miR162 and miR168 in various organs and seedling phase of M. truncatula plants. [score:3]
In M. truncatula miR162 and miR168 are expressed in different plant organs (Additional file 9). [score:3]
Figure 5miR162 and miR168 expression in shoots and roots of M. truncatula in different water status. [score:3]
Click here for file Expression of miR162 and miR168 in various organs and seedling phase of M. truncatula plants. [score:3]
In M. truncatula their mRNAs are targeted by miR162 and miR168 respectively [2, 3]. [score:3]
The AGO1 homeostasis is maintained by the post-transcriptional regulation of AGO1 by miR168 and the stabilization of miR168 levels by AGO1 [65]. [score:2]
The expression of both miR168 and miR162 did not seem to change in the shoots of plants subjected to water deficit and in the recovered plants when compared with the controls (Figure 5). [score:2]
The membrane was first hybridized with miR162 probe and then striped and rehybridized with miR168 probe. [score:1]
For miR168, again two bands are visible, one of 21-nt that corresponds to the miRNA [2, 3] and a faint band of 24-nt. [score:1]
However we could not correlate this with the variation of miR168 accumulation (Figure 5). [score:1]
Likewise, AGO1 mRNA contains a complementary sequence for miR168 which leads to AGO1 -mediated cleavage of AGO1 mRNA [28]. [score:1]
The Locked Nucleic Acid (LNA) -modified oligonucleotides (Exiqon, Vedbaek, Denmark) complementary to miR168 and miR162 and the molecular weight probes were labeled with γP [32]-ATP (PerkinElmer, Waltham, Massachusetts, USA) according to Trindade et al [4]. [score:1]
The membrane was first hybridized with miR168 probe and then striped and rehybridized with miR162 probe. [score:1]
The accumulation of miR162 and miR168 (numbers indicated under each lane) was quantified according to U6 small nuclear RNA loading control and normalized to control conditions. [score:1]
miR162 and miR168 northern blot analysis. [score:1]
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2
[+] score: 34
Since over -expression of endogenous AtAGO1, elicited by the mutation of its miR168 target site, leads to a miRNA-pathway loss-of-function effect (Vaucheret et al., 2004, 2006), the excessive expression of HsAGO2, unregulated by any homeostatic mechanism, might perturb miRNA activity in a similar fashion. [score:9]
For the 35S:4mAGO1 construct, four silent mutations introducing mismatches to the miR168 target site in AtAGO1, as per (Vaucheret et al., 2004), were generated via a site directed mutagenesis strategy based on that of (Liu and Naismith, 2008). [score:5]
When this circuit is disturbed via the mutation of AtAGO1's miR168 target site, the over-accumulation of AtAGO1 protein leads—somewhat counter intuitively—to a general perturbation of miRNA -mediated regulation in the plant, manifesting in pleiotropic defects that resemble those apparent in a range of miRNA-pathway loss-of-function alleles (Morel et al., 2002; Vaucheret et al., 2004; Vazquez et al., 2004). [score:5]
To generate these, a cDNA for AtAGO1 was placed under a double 35S promoter (35S:AtAGO1) and, in a separate construct (35S: 4mAGO1), four silent mutations identical to those described by Vaucheret et al. (2004) were created, introducing four mismatches to AtAGO1' s miR168 target site, which should render it resistant to miR168 -mediated regulation. [score:5]
Emphasizing the importance of its role, AtAGO1 homeostasis must be delicately maintained by a feedback mechanism in which miR168 directs the AtAGO1 -dependent silencing of AtAGO1 expression. [score:4]
The five miRNA targets examined, AtAGO1 (miR168), DCL1 (miR162), PHABULOSA (PHB) (miR165/166), MYB33 (miR159), and CUP-SHAPED COTYLEDONS 2 (CUC2) (miR164), all showed elevated un-cleaved mRNA abundances in ago1–27 plants relative to wild type (Figure 2B), consistent with what has previously been reported (Morel et al., 2002). [score:3]
AGO1 homeostasis entails coexpression of MIR168 and AGO1 and preferential stabilization of miR168 by AGO1. [score:3]
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3
[+] score: 26
Other miRNAs from this paper: ath-MIR159a, ath-MIR168a, ath-MIR159b, ath-MIR159c
Mutations that interfere with the activity of miR168 (for example, sqn, ago1and hen1) disrupt this feedback mechanism, thereby making AGO1 more susceptible to other factors that regulate its expression. [score:5]
We suspect this is because the miR168 -dependent feedback mechanism that regulates AGO1 expression [13] partially corrects for slight increases in the level of this protein in fbw2 mutants. [score:4]
AGO1 is a target of miR168 and negatively regulates its own activity by promoting the activity and stability of miR168 [9, 13] and by promoting the activity of siRNAs derived from the AGO1 transcript [14]. [score:4]
Although we were initially surprised by this result, we recognized that the phenotypes of these double mutants are remarkably similar to the phenotype of plants transformed with a miR168-resistant version of AGO1, which results in the over -expression of AGO1 (Figure 5K) [9, 13]. [score:3]
miR168 represses AGO1 in an AGO1 -dependent fashion: a decrease in the activity of AGO1 leads to a decrease in miR168 activity and a subsequent increase in AGO1 expression, whereas an increase in AGO1 activity has the opposite effect. [score:3]
In wild-type plants, the expression of AGO1 is maintained at a constant level by a negative feedback loop involving miR168. [score:3]
An important component of this homeostatic mechanism is the negative regulation of AGO1 by miR168 [9, 13]. [score:2]
Many hst-3 pAGO1::FLAG-AGO1 and se-1 pAGO1::FLAG-AGO1 primary transformants had phenotypes that were almost identical to hst-3 fbw2 and se-1 fbw2, and strongly resembled plants containing miR168-resistant AGO1 mRNA constructs (Figure 5H - J). [score:1]
hyl1-3 fbw2-1 and se-1 fbw2-1 double mutants have phenotypes similar to hyl1-3 and se-1 plants transformed with ARGONAUTE1::FLAG-AGO1 (I and J) or WT plants containing the miR168 insensitive 2m-AGO1 construct (K). [score:1]
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4
[+] score: 24
Among these targets, it can be mentioned members of the SOD family (targets of miR398), ARF8 an auxin response factor (target of miR167), DCL1 (target of miR162), AGO1 (target of miR168) and AGO2 (target of miR403). [score:13]
Moreover, miR168 and its target AGO1 are both up-regulated after several virus infection even though the final outcome is less AGO1 protein by means of translational repression [58, 59]. [score:8]
AGO1 homeostasis entails coexpression of MIR168 and AGO1 and preferential stabilization of miR168 by AGO1. [score:3]
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5
[+] score: 15
miR168 has also been shown to be up-regulated by heat in Brassica rapa (Bilichak et al., 2015) and celery (Li et al., 2014a), but down-regulated in Chinese white poplar (Chen et al., 2012) and rice (Sailaja et al., 2014). [score:7]
A differential expression of bra-miR168 following heat shock in the parental tissues was observed to be negatively correlated with transcript levels of its putative target braAGO1 in the corresponding tissues, suggesting the important role of bra-miR168 in heat responses (Bilichak et al., 2015). [score:5]
Bra-miR168 and its target braAGO1 are also suggested to be putative messengers that mediate meiotic epigenetic inheritance in Brassica rapa (Bilichak et al., 2015). [score:3]
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6
[+] score: 12
However, contrary to the reported independence of miR168 accumulation in the absence of DRB activity in whole plants, these analyses also revealed that in the specific tissue analyzed by sRNA sequencing, the SAM region, DRB1 is required for wild-type miR168 accumulation and target gene expression regulation (Figure 2B). [score:6]
Furthermore, and as illustrated for miR168, miR162 accumulation remained at wild-type levels in the absence of DRB2, DRB3 and DRB5 expression (Figure S3). [score:3]
Compared to wild-type plants, no change in the levels of miR168, the precursor transcript PRI-MIR168A or the target gene AGO1 were observed in drb2 plants (Figure 4B). [score:2]
MiR168 was selected as the unchanged miRNA class representative, as we have previously demonstrated that the accumulation of this miRNA is largely unaffected by the loss of activity of any of the five members of the Arabidopsis DRB protein family in whole plant samples [25], [35]. [score:1]
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7
[+] score: 10
While the expressions of 14 families (miR156/miR157, miR158, miR160, miR162, miR165/miR166, miR168, miR169, miR171, miR390, miR393, miR394, miR396, miR398, and miR399) were dramatically reduced, 3 families (miR159, miR167, and miR172) were up-regulated in CsCl -treated seedlings. [score:6]
As shown in the radial chart in Fig 4C, expression of the miR157, miR160, miR165, miR168, miR171, miR319, and miR403 families was decreased by around 80% to 140% in CsCl -treated seedlings. [score:3]
miR167, miR168, miR172, miR396, and miR398) were notably increased (Fig 3B, S2 Fig). [score:1]
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8
[+] score: 7
In addition, the sequencing results also revealed that various other stress-regulated miRNAs were expressed in response to LPS which include: miR161, miR165, miR166, miR167, miR168 miR401, miR403, miR405 and miR5635. [score:4]
Experimental studies in Arabidopsis and other plants have shown that abiotic and biotic stresses induce differential expression of a set of miRNAs such as: miR156, miR159, miR165, miR167, miR168, miR169, miR319, miR393, miR395, miR396, miR398, miR399, and miR402 [7, 18- 23]. [score:3]
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9
[+] score: 7
Among them are miR162 and miR168 (targeting DCL1 and AGO1 transcripts, respectively), this observation pointing to a pathogen-regulation of the miRNA machinery itself (Baldrich et al. 2015). [score:4]
Two miRNAs, miR162 and miR168, are involved in the regulation of the miRNA pathway itself by guiding cleavage of DCL1 and AGO1 mRNAs, respectively. [score:2]
The antiviral function of AGO18 depends on its activity to sequester miR168 to alleviate repression of rice AGO1 which is essential for antiviral RNA interference AGO1 (Wu et al. 2015). [score:1]
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10
[+] score: 7
miR168 (19 reads) and miR403 (7 reads) were detected, targeting ARGONAUTE1 and ARGONAUTE2 respectively. [score:3]
Our results confirm that the homeostatic feedback loop that regulates steady-state levels of AGO1 and DCL1 proteins is likely to operate, as both miR168 and miR162 were found. [score:2]
However, it has been proposed [17] that microRNA function is necessary for pollen development given the presence and functioning of miR168 which operates to modulate ARGONAUTE1 transcript levels in a mechanism maintaining AGO1 protein levels by a feedback loop [2, 26]. [score:2]
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11
[+] score: 4
For example, miR159, miR165, miR166, and miR168 are usually incorporated into AGO1 -based RISCs, but associate with other AGOs in AGO1 -deficient Arabidopsis mutants, where this redirection is supposed to be mediated by stabilizing proteins (Vaucheret, 2009; Zhu et al., 2011). [score:2]
Rather, as reported in (Vaucheret, 2009) for 21-nt and 22-nt isoforms of miR168 miRNA, changes in miRNA length were shown to influence the downstream efficiency of the RISC. [score:1]
AGO1 homeostasis involves differential production of 21-nt and 22-nt miR168 species by MIR168a and MIR168b. [score:1]
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12
[+] score: 3
Other miRNAs from this paper: ath-MIR168a, sly-MIR168a, sly-MIR168b
For instance, p19 of Cymbidium ring spot virus represses the AGO1-directed antiviral response by specific induction of miR168, which in turn, negatively regulates AGO1 mRNA levels [26]. [score:3]
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13
[+] score: 3
Examples include ath-MIR168b which was observed 86,634 times in the four different AGO association datasets, whereas the ath-MIR168b+1 sequence was observed only 34 times. [score:1]
A recent report described and confirmed the occurrence of a long (22 nt) form of ath-miR168 [12]. [score:1]
The biological significance of sequence length heterogeneity has been recently identified for a mature miRNA in Arabidopsis thaliana, in which ath-MIR168 is processed as miRNAs of 21 and 22 nucleotides in length from its two genomic loci. [score:1]
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14
[+] score: 3
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR159a, ath-MIR160a, ath-MIR160b, ath-MIR160c, ath-MIR162a, ath-MIR162b, ath-MIR164a, ath-MIR164b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR168a, ath-MIR169a, ath-MIR172a, ath-MIR172b, ath-MIR159b, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR162a, osa-MIR164a, osa-MIR164b, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR169a, ath-MIR167d, ath-MIR169b, ath-MIR169c, ath-MIR169d, ath-MIR169e, ath-MIR169f, ath-MIR169g, ath-MIR169h, ath-MIR169i, ath-MIR169j, ath-MIR169k, ath-MIR169l, ath-MIR169m, ath-MIR169n, ath-MIR172c, ath-MIR172d, ath-MIR395a, ath-MIR395b, ath-MIR395c, ath-MIR395d, ath-MIR395e, ath-MIR395f, ath-MIR396a, ath-MIR396b, ath-MIR399a, ath-MIR399b, ath-MIR399c, ath-MIR399d, ath-MIR399e, ath-MIR399f, osa-MIR395b, osa-MIR395d, osa-MIR395e, osa-MIR395g, osa-MIR395h, osa-MIR395i, osa-MIR395j, osa-MIR395k, osa-MIR395l, osa-MIR395s, osa-MIR395t, osa-MIR395c, osa-MIR395a, osa-MIR395f, osa-MIR395u, osa-MIR396a, osa-MIR396b, osa-MIR396c, osa-MIR399a, osa-MIR399b, osa-MIR399c, osa-MIR399d, osa-MIR399e, osa-MIR399f, osa-MIR399g, osa-MIR399h, osa-MIR399i, osa-MIR399j, osa-MIR399k, ath-MIR408, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR164c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR160e, osa-MIR160f, osa-MIR162b, osa-MIR164c, osa-MIR164d, osa-MIR164e, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR168a, osa-MIR168b, osa-MIR169b, osa-MIR169c, osa-MIR169d, osa-MIR169e, osa-MIR169f, osa-MIR169g, osa-MIR169h, osa-MIR169i, osa-MIR169j, osa-MIR169k, osa-MIR169l, osa-MIR169m, osa-MIR169n, osa-MIR169o, osa-MIR169p, osa-MIR169q, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR171h, osa-MIR408, osa-MIR172d, osa-MIR167j, osa-MIR166m, osa-MIR166j, osa-MIR164f, zma-MIR156d, zma-MIR156f, zma-MIR156g, zma-MIR156b, zma-MIR156c, zma-MIR156e, zma-MIR156a, zma-MIR156h, zma-MIR156i, zma-MIR160a, zma-MIR160c, zma-MIR160d, zma-MIR160b, zma-MIR164a, zma-MIR164d, zma-MIR164b, zma-MIR164c, zma-MIR169a, zma-MIR169b, zma-MIR167a, zma-MIR167b, zma-MIR167d, zma-MIR167c, zma-MIR160e, zma-MIR166a, zma-MIR162, zma-MIR166h, zma-MIR166e, zma-MIR166i, zma-MIR166f, zma-MIR166g, zma-MIR166b, zma-MIR166c, zma-MIR166d, zma-MIR172a, zma-MIR172d, zma-MIR172b, zma-MIR172c, osa-MIR396e, zma-MIR395b, zma-MIR395c, zma-MIR395a, zma-MIR396b, zma-MIR396a, zma-MIR399a, zma-MIR399c, zma-MIR399b, zma-MIR399d, zma-MIR399e, zma-MIR399f, zma-MIR156j, zma-MIR159a, zma-MIR159b, zma-MIR159c, zma-MIR159d, zma-MIR166k, zma-MIR166j, zma-MIR167e, zma-MIR167f, zma-MIR167g, zma-MIR167h, zma-MIR167i, zma-MIR168a, zma-MIR168b, zma-MIR169c, zma-MIR169f, zma-MIR169g, zma-MIR169h, zma-MIR169i, zma-MIR169k, zma-MIR169j, zma-MIR169d, zma-MIR169e, zma-MIR172e, zma-MIR166l, zma-MIR166m, zma-MIR171h, zma-MIR408a, zma-MIR156k, zma-MIR160f, osa-MIR529a, osa-MIR395m, osa-MIR395n, osa-MIR395o, osa-MIR395p, osa-MIR395q, osa-MIR395v, osa-MIR395w, osa-MIR395r, osa-MIR529b, osa-MIR169r, osa-MIR396f, zma-MIR396c, zma-MIR396d, osa-MIR2118a, osa-MIR2118b, osa-MIR2118c, osa-MIR2118d, osa-MIR2118e, osa-MIR2118f, osa-MIR2118g, osa-MIR2118h, osa-MIR2118i, osa-MIR2118j, osa-MIR2118k, osa-MIR2118l, osa-MIR2118m, osa-MIR2118n, osa-MIR2118o, osa-MIR2118p, osa-MIR2118q, osa-MIR2118r, osa-MIR2275a, osa-MIR2275b, zma-MIR2118a, zma-MIR2118b, zma-MIR2118c, zma-MIR2118d, zma-MIR2118e, zma-MIR2118f, zma-MIR2118g, zma-MIR2275a, zma-MIR2275b, zma-MIR2275c, zma-MIR2275d, osa-MIR396g, osa-MIR396h, osa-MIR396d, zma-MIR156l, zma-MIR159e, zma-MIR159f, zma-MIR159g, zma-MIR159h, zma-MIR159i, zma-MIR159j, zma-MIR159k, zma-MIR160g, zma-MIR164e, zma-MIR164f, zma-MIR164g, zma-MIR164h, zma-MIR166n, zma-MIR167j, zma-MIR169l, zma-MIR169m, zma-MIR169n, zma-MIR169o, zma-MIR169p, zma-MIR169q, zma-MIR169r, zma-MIR395d, zma-MIR395e, zma-MIR395f, zma-MIR395g, zma-MIR395h, zma-MIR395i, zma-MIR395j, zma-MIR395k, zma-MIR395l, zma-MIR395m, zma-MIR395n, zma-MIR395o, zma-MIR395p, zma-MIR396e, zma-MIR396f, zma-MIR396g, zma-MIR396h, zma-MIR399g, zma-MIR399h, zma-MIR399i, zma-MIR399j, zma-MIR408b, zma-MIR529, osa-MIR395x, osa-MIR395y, osa-MIR2275c, osa-MIR2275d, ath-MIR156i, ath-MIR156j
The six most abundantly expressed miRNA families were miR166, miR168, miR167, miR156, miR159, and miRs6. [score:3]
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15
[+] score: 3
Nonetheless, much is known about how AGO1 is regulated by the miR168 -mediated feedback loop during ABA responses. [score:2]
MicroRNAs are also key candidates for dynamic and long-distance abiotic stress signaling and ABA induce the accumulation of a number of small RNAs, including miR168. [score:1]
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16
[+] score: 2
miR171, miR398, miR168, and miR167 oscillate diurnally but are not under clock-control (Sire et al., 2009). [score:1]
Diurnal oscillation in the accumulation of Arabidopsis, miR167 miR168 miR171 and miR398. [score:1]
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17
[+] score: 1
Among these miRNAs, the miR168 and miR166 families had the most reads in the control and treatment groups, respectively. [score:1]
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18
[+] score: 1
Accumulations of miR159, miR168 and miR165 are insensitive to decreased DCL1 activity unlike other miRNAs such as miR173, indicative of alternative dicer activity for the processing of these miRNAs [51]. [score:1]
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19
[+] score: 1
Diurnal oscillation in the accumulation of Arabidopsis microRNAs, miR167, miR168, miR171 and miR398. [score:1]
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20
[+] score: 1
Among these families, the miR156 family had the most family members (13), with 93.7% of all conserved miRNA reads, followed by miR166 (6), miR168 (5), miR167 (4), miR397 (4), miR390 (3), and miR399 (3) etc. [score:1]
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21
[+] score: 1
The abundance of miR160, miR168, miR170, miR393, miR395, miR408 and miR850 were specifically increased. [score:1]
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22
[+] score: 1
miR390 and miR168 were used as IP controls, and U6 as loading control. [score:1]
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23
[+] score: 1
The newly identified genes were members of diverse gene families such as major facilitator super family (MFS) transporters [AT1G08900, AT1G30560, AT1G33440, AT1G72140, AT1G80530, AT2G26690, AT2G34355, AT3G20460, AT3G45680, AT3G47960, AT4G19450, STP8 (AT5G26250), AT5G27350, and AT5G62680], MATE efflux transporters (AT1G71140, AT5G17700, AT5G19700, and AT5G38030), microRNA genes [MIR156b (AT4G30972), MIR161 (AT1G48267), MIR162b (AT5G23065), MIR164 (AT5G01747), MIR167c (AT3G04765), MIR168b (AT5G45307), MIR396a (AT2G10606), MIR402 (AT1G77235), MIR777a (AT1G70645), and MIR848a (AT5G13887)], various transcription factors (MYB, NAC domain, WRKY, etc. [score:1]
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24
[+] score: 1
In Arabidopsis, miR156, miR158, miR159, miR165, miR167, miR168, miR169, miR171, miR319, miR393, miR394 and miR396 are drought-responsive. [score:1]
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25
[+] score: 1
04)>GCU(76.99), 5:G(219813.04)>CUA(325.81), 7:U(158835.06)>GCA(466.4), 8:G(158835.06)>CUA(326.2), 9:C(158835.06)>GUA(602.6), 10:C(158835.06)>GU(431.4), 11:A(219826.94)>GCU(3686.02), 12:G(219826.94)>CU(1018.4), 13:C(158835.06)>GU(336.74) MIR168a, MIR168b 9:U(15948.36)>CA(36.6), 10:G(15933.26)>A(24.3), 12:A(15933.26)>GC(22.2), 13:G(15933.26)>UA(43.1), 14:G(15933.26)>UA(41.3) MIR169a, MIR169b, MIR169c, MIR169d, MIR169e, MIR169h, MIR169i, MIR169j, MIR169k, MIR169l, MIR169m, MIR169n 6:C(24472.01)>GU(40.32), 8:A(87930.11)>GCU(77.31), 9:G(58812. [score:1]
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26
[+] score: 1
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR159a, ath-MIR160a, ath-MIR160b, ath-MIR160c, ath-MIR164a, ath-MIR164b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR168a, ath-MIR171a, ath-MIR172a, ath-MIR172b, ath-MIR159b, ath-MIR319a, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR164a, osa-MIR164b, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR171a, ath-MIR167d, ath-MIR172c, ath-MIR172d, ath-MIR393a, ath-MIR393b, ath-MIR396a, ath-MIR396b, ath-MIR398a, osa-MIR393a, osa-MIR396a, osa-MIR396b, osa-MIR396c, osa-MIR398a, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR164c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR319a, osa-MIR160e, osa-MIR160f, osa-MIR164c, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR168a, osa-MIR168b, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR393b, osa-MIR172d, osa-MIR167j, osa-MIR166m, osa-MIR166j, osa-MIR437, osa-MIR396e, osa-MIR444a, osa-MIR528, osa-MIR531a, osa-MIR1425, osa-MIR444b, osa-MIR444c, osa-MIR444d, osa-MIR444e, osa-MIR444f, osa-MIR531b, osa-MIR1862a, osa-MIR1862b, osa-MIR1862c, osa-MIR1873, osa-MIR1862d, osa-MIR1862e, osa-MIR396f, osa-MIR396g, osa-MIR396h, osa-MIR396d, osa-MIR1862f, osa-MIR1862g, ath-MIR5021, osa-MIR5072, osa-MIR5077, ath-MIR156i, ath-MIR156j, osa-MIR531c
Till now, many plants have been explored e. g. in rice, miR168 was found to bound human/mouse low-density lipoprotein receptor adapter protein 1 (LDLRAP1) mRNA and reduce its protein level in liver, and consequently decrease LDL removal from mouse plasma [43]. [score:1]
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This was observed in a previous study on tandem duplicated paralogs of miR395 in rice and miR168 in Brassicaceae [34], [35]. [score:1]
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Liu et al [7] identified 14 miRNAs induced by high-salinity, drought and low temperature in Arabidopsis thaliana on a microarray -based analysis, among which miR168, miR171 and miR396 responded to all three stresses. [score:1]
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