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31 publications mentioning ath-MIR393b

Open access articles that are associated with the species Arabidopsis thaliana and mention the gene name MIR393b. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 73
The down-regulation of miR393 and up-regulation of the corresponding LecRLK target gene indicates that this could be part of enhancing the perception capabilities of A. thaliana cells exposed to LPS. [score:9]
However, the more sensitive qPCR confirmed the expression profile of miR393 by revealing a slight down-regulation in the leaf tissue and a significant down-regulation of 6.6 fold in the callus tissue (Figure  5A, B). [score:9]
The sequencing analysis showed that in both callus and leaf tissues, various stress regulated-miRNAs were differentially expressed and real time PCR validated the expression profile of miR156, miR158, miR159, miR169, miR393, miR398, miR399 and miR408 along with their target genes. [score:8]
The qPCR showed that miR393 was expressed but significantly down-regulated in the treated callus tissue which contrasted the results obtained by sequencing analysis, which indicated that it was not expressed in the treated callus tissue. [score:8]
In both callus and leaf tissues, four miRNAs (miR156, miR169, miR398 and miR408) were up-regulated, two miRNAs (miR158, and miR393) were down-regulated with two other miRNAs (miR159 and miR396) only found in the callus tissue (Figure  5A, B). [score:7]
miR393 was reported to regulate auxin signaling and defense response by targeting TIR1, (part of the ubiquitin ligase complex SCFTIR1) which represses auxin signaling and enhances bacterial disease resistance [22, 24]. [score:6]
In this study, target prediction revealed that miR393 targets genes which encode concanavalin A-like lectin protein kinase family proteins. [score:5]
To validate the sequencing results with the bioinformatics -based analysis and based on their key function in gene regulation, the following mature miRNA were selected for expression profile analysis: miR156, mi158, miR159, miR169, miR393, miR396, miR398, miR399 and miR408. [score:4]
The expression data was then compared against the H-T sequencing data analysis which revealed that five (miR156, miR169, miR398, miR399 and miR408) of the nine miRNAs in callus tissue and six (miR158, miR159, miR169, miR393, miR396 and miR408) of the nine miRNAs in leaf tissue showed expression patterns that were similar to those observed with the H-T sequencing data. [score:4]
The greatest degree of down-regulation in response to LPS was shown by miR393 in the callus tissue. [score:4]
Experimental studies in Arabidopsis and other plants have shown that abiotic and biotic stresses induce differential expression of a set of miRNAs such as: miR156, miR159, miR165, miR167, miR168, miR169, miR319, miR393, miR395, miR396, miR398, miR399, and miR402 [7, 18- 23]. [score:3]
This observation was in contrast with the study of Navarro et al. [24] who reported that Arabidopsis miR393 expression was induced with a two fold increase following treatment with flg22. [score:3]
The expression profile of miR393 was repressed in the leaf tissue as shown by the H-T sequencing results (Table  2) but no single read was detected in the callus tissue. [score:3]
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2
[+] score: 51
Expression Changes of Drought Associated miRNAs Target Genes under PEG8000 Stress and NaHS Treatment in Arabidopsis We selected ARF8 (target gene of miR167); TIR1, AFB2 and AFB3 (target genes of miR393); GRF1, GRF2 and GRF3 (target genes of miR396); CSD1 and CSD2 (target genes of miR398) to determine possible transcriptional changes of drought -associated miRNA target genes. [score:15]
We selected ARF8 (target gene of miR167); TIR1, AFB2 and AFB3 (target genes of miR393); GRF1, GRF2 and GRF3 (target genes of miR396); CSD1 and CSD2 (target genes of miR398) to determine possible transcriptional changes of drought -associated miRNA target genes. [score:11]
On the other hand, H [2]S is involved in regulating the expression of drought associated miRNAs such as miR167, miR393, miR396 and miR398 and can therefore affect their target gene expressions and so to improve the tolerance of Arabidopsis to drought. [score:8]
Under drought stress, miR167, miR393 and miR396 are upregulated, miR169 is downregulated and miR398 is differentially regulated [17]. [score:8]
miR393 targets transport inhibitor response 1 (TIR1), auxin signaling F-box proteins 1, 2 and 3 (AFB1, AFB2 and AFB3) [20], which are involved in determining the length of the main root and hypocotyl and the number of lateral roots [21]. [score:5]
According to previous research, TIR1, AFB1, AFB2 and AFB3 (targets of miR393) affect the growth of the main root and hypocotyl and the number of lateral roots [21]. [score:3]
In Arabidopsis, miR156, miR158, miR159, miR165, miR167, miR168, miR169, miR171, miR319, miR393, miR394 and miR396 are drought-responsive. [score:1]
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3
[+] score: 19
In addition to the miR393 -mediated regulation of auxin receptors, the expression of several Auxin Response Factors (ARFs) is known to be regulated by different miRNAs in plants (Khraiwesh et al. 2012; Liu et al. 2014) Among them are miR160 (targeting ARF10, ARF16 and ARF17) and miR167 (targeting ARF6 and ARF8) (Rhoades et al. 2002). [score:9]
In the case of miR393, the two sRNAs of the duplex are functional during pathogen infection (miR393 targets TIR1, whereas miR393* regulates MEM12, a Golgi-localised SNARE protein that modulates exocytosis of antimicrobial PR1 proteins in Arabidopsis (Zhang et al. 2011). [score:4]
While auxin signalling pathway is regulated by miR160, miR167, miR390 and miR393, the JA biosynthetic pathway is under the control of miR319 and miR159, and miR159 regulate the ABA signalling pathway (Curaba et al. 2014). [score:3]
The involvement of miRNAs in PTI responses and pathogen resistance was first demonstrated in Arabidopsis where perception of flg22, a peptide derived from the general elicitor flagellin, causes an increase in miR393 accumulation which in turn negatively regulates transcripts for F-box auxin receptors. [score:2]
The Arabidopsis AGO1 is required for Verticillium pathogenicity (Ellendorff et al. 2009), whereas AGO2 is involved in pathogen immunity by binding miR393* in modulating the exocytosis of Pathogenesis-Related 1 (PR1) (Zhang et al. 2011). [score:1]
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4
[+] score: 17
Turner and collaborators have down-regulated auxin perception by overexpressing the micro -RNA miR393 that induces mRNA degradation of auxin receptors, both in soybean and M. truncatula [76]. [score:6]
Whereas miR393 over -expression resulted in less LR, it had no effect on nodulation in soybean [76], however, the same miR393 overexpression reduced both LR density and nodule number in M. truncatula [77]. [score:5]
This supports the hypothesis that determinate and indeterminate nodules do not have the same auxin requirement for their development (even if auxin perception is required for LRF in both species as shown by the reduced LR density observed in the miR393 overexpressing lines). [score:4]
Mao G. Turner M. Yu O. Subramanian S. miR393 and miR164 influence indeterminate but not determinate nodule development Plant Signal. [score:2]
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5
[+] score: 13
Unlike some miRNAs, such as miR160, miR167 and miR393, which directly target and regulate the expression of key components of the auxin response pathway, the miR165/166 targets themselves are not major components of hormone response pathways but they regulate the transcription of important components of hormone pathways. [score:10]
miR393 targets auxin receptor TIR1 and closely related F-box genes [46, 57]. [score:3]
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6
[+] score: 12
Other miRNAs from this paper: ath-MIR393a, osa-MIR393a, osa-MIR393b, mtr-MIR393a, mtr-MIR393b
Chen Z. H. Bao M. L. Sun Y. Z. Yang Y. J. Xu X. H. Wang J. H. Han N. Bian H. W. Zhu M. Y. Regulation of auxin response by miR393 -targeted TRANSPORT INHIBITOR RESPONSE PROTEIN 1 is involved in normal development in Arabidopsis Plant Mol. [score:7]
ABA also induces expression of the microRNA, miR393, which cleaves transcripts for the auxin receptors, TIR1 and AFB2, thus reducing auxin signaling and inhibiting LR growth (Figure 2) [114]. [score:5]
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7
[+] score: 10
It has been suggested that the AUXIN SIGNALING F-BOX 2 (AFB2) gene is post-transcriptionally negatively regulated by miR393, and a regulatory mechanism where miRNAs prevent undesired expression of genes involved in miRNA production has been proposed [73]. [score:5]
An alternative to this suggestion comes from the finding of numerous siRNAs in the proximity of the MIR393 target site for the F-boxes TIR1, AFB2, and AFB3 genes [74]. [score:3]
[74] suggested that the regulation of their transcripts occurs via siRNAs rather than MIR393. [score:2]
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8
[+] score: 10
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR159a, ath-MIR160a, ath-MIR160b, ath-MIR160c, ath-MIR162a, ath-MIR162b, ath-MIR164a, ath-MIR164b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR169a, ath-MIR171a, ath-MIR172a, ath-MIR172b, ath-MIR159b, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR162a, osa-MIR164a, osa-MIR164b, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR169a, osa-MIR171a, ath-MIR167d, ath-MIR169b, ath-MIR169c, ath-MIR169d, ath-MIR169e, ath-MIR169f, ath-MIR169g, ath-MIR169h, ath-MIR169i, ath-MIR169j, ath-MIR169k, ath-MIR169l, ath-MIR169m, ath-MIR169n, ath-MIR171b, ath-MIR171c, ath-MIR172c, ath-MIR172d, ath-MIR393a, ath-MIR394a, ath-MIR394b, ath-MIR395a, ath-MIR395b, ath-MIR395c, ath-MIR395d, ath-MIR395e, ath-MIR395f, osa-MIR393a, osa-MIR394, osa-MIR395b, osa-MIR395d, osa-MIR395e, osa-MIR395g, osa-MIR395h, osa-MIR395i, osa-MIR395j, osa-MIR395k, osa-MIR395l, osa-MIR395s, osa-MIR395t, osa-MIR395c, osa-MIR395a, osa-MIR395f, osa-MIR395u, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR164c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR160e, osa-MIR160f, osa-MIR162b, osa-MIR164c, osa-MIR164d, osa-MIR164e, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR169b, osa-MIR169c, osa-MIR169d, osa-MIR169e, osa-MIR169f, osa-MIR169g, osa-MIR169h, osa-MIR169i, osa-MIR169j, osa-MIR169k, osa-MIR169l, osa-MIR169m, osa-MIR169n, osa-MIR169o, osa-MIR169p, osa-MIR169q, osa-MIR171b, osa-MIR171c, osa-MIR171d, osa-MIR171e, osa-MIR171f, osa-MIR171g, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR171h, osa-MIR393b, osa-MIR172d, osa-MIR171i, osa-MIR167j, osa-MIR166m, osa-MIR166j, osa-MIR164f, zma-MIR156d, zma-MIR156f, zma-MIR156g, zma-MIR156b, zma-MIR156c, zma-MIR156e, zma-MIR156a, zma-MIR156h, zma-MIR156i, zma-MIR160a, zma-MIR160c, zma-MIR160d, zma-MIR160b, zma-MIR164a, zma-MIR164d, zma-MIR164b, zma-MIR164c, zma-MIR169a, zma-MIR169b, zma-MIR167a, zma-MIR167b, zma-MIR167d, zma-MIR167c, zma-MIR160e, zma-MIR166a, zma-MIR162, zma-MIR166h, zma-MIR166e, zma-MIR166i, zma-MIR166f, zma-MIR166g, zma-MIR166b, zma-MIR166c, zma-MIR166d, zma-MIR171a, zma-MIR171b, zma-MIR172a, zma-MIR172d, zma-MIR172b, zma-MIR172c, zma-MIR171d, zma-MIR171f, zma-MIR394a, zma-MIR394b, zma-MIR395b, zma-MIR395c, zma-MIR395a, zma-MIR156j, zma-MIR159a, zma-MIR159b, zma-MIR159c, zma-MIR159d, zma-MIR166k, zma-MIR166j, zma-MIR167e, zma-MIR167f, zma-MIR167g, zma-MIR167h, zma-MIR167i, zma-MIR169c, zma-MIR169f, zma-MIR169g, zma-MIR169h, zma-MIR169i, zma-MIR169k, zma-MIR169j, zma-MIR169d, zma-MIR169e, zma-MIR171c, zma-MIR171j, zma-MIR171e, zma-MIR171i, zma-MIR171g, zma-MIR172e, zma-MIR166l, zma-MIR166m, zma-MIR171k, zma-MIR171h, zma-MIR393a, zma-MIR156k, zma-MIR160f, osa-MIR528, osa-MIR529a, osa-MIR395m, osa-MIR395n, osa-MIR395o, osa-MIR395p, osa-MIR395q, osa-MIR395v, osa-MIR395w, osa-MIR395r, ath-MIR827, osa-MIR529b, osa-MIR1432, osa-MIR169r, osa-MIR827, osa-MIR2118a, osa-MIR2118b, osa-MIR2118c, osa-MIR2118d, osa-MIR2118e, osa-MIR2118f, osa-MIR2118g, osa-MIR2118h, osa-MIR2118i, osa-MIR2118j, osa-MIR2118k, osa-MIR2118l, osa-MIR2118m, osa-MIR2118n, osa-MIR2118o, osa-MIR2118p, osa-MIR2118q, osa-MIR2118r, osa-MIR2275a, osa-MIR2275b, zma-MIR2118a, zma-MIR2118b, zma-MIR2118c, zma-MIR2118d, zma-MIR2118e, zma-MIR2118f, zma-MIR2118g, zma-MIR2275a, zma-MIR2275b, zma-MIR2275c, zma-MIR2275d, zma-MIR156l, zma-MIR159e, zma-MIR159f, zma-MIR159g, zma-MIR159h, zma-MIR159i, zma-MIR159j, zma-MIR159k, zma-MIR160g, zma-MIR164e, zma-MIR164f, zma-MIR164g, zma-MIR164h, zma-MIR166n, zma-MIR167j, zma-MIR169l, zma-MIR169m, zma-MIR169n, zma-MIR169o, zma-MIR169p, zma-MIR169q, zma-MIR169r, zma-MIR171l, zma-MIR171m, zma-MIR171n, zma-MIR393b, zma-MIR393c, zma-MIR395d, zma-MIR395e, zma-MIR395f, zma-MIR395g, zma-MIR395h, zma-MIR395i, zma-MIR395j, zma-MIR395k, zma-MIR395l, zma-MIR395m, zma-MIR395n, zma-MIR395o, zma-MIR395p, zma-MIR482, zma-MIR528a, zma-MIR528b, zma-MIR529, zma-MIR827, zma-MIR1432, osa-MIR395x, osa-MIR395y, osa-MIR2275c, osa-MIR2275d, ath-MIR156i, ath-MIR156j
Other conserved miRNA targets includes F-box protein (miRNA393, miRNA394), ATP sulfurylase (miRNA395), CCHC type zinc finger protein (miRNA482), NAD(P) -binding protein (miRNA827), and Poly(ADP-ribose) polymerase (miRNA1432), all of them are known to play roles in the expression control of genes involved in regulation of metabolic processes. [score:6]
MiR393 targets auxin receptor genes, such as TIR1, AFB2 and AFB3, which lower auxin signals and inhibit the pathogen P. syringae [20]. [score:4]
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9
[+] score: 9
10 conserved miRNA families had highly increased expression (log [2]fold > 2) in response to Xam infection (Figure 2a), including miR160 and miR167 families which are both known to target auxin response factors (ARFs) [13] and miR393 and miR390 families which are also known to regulate auxin signaling [13]. [score:6]
miRNAs induction was found to be involved in regulating auxin signaling: miR160, miR167, miR390 and miR393 [11, 13]. [score:2]
It has been demonstrated that bacteria -induced miR393 mediates anti-bacterial defense of A. thaliana against Pseudomonas syringae pv. [score:1]
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10
[+] score: 9
Besides miR393, two other miRNA families, miR160 and miR167, are upregulated following PsDC3000 inoculation and target members of auxin-response factors (ARF) [14]. [score:6]
The Arabidopsis miR393 imparts basal resistance to the bacterial pathogen Pseudomonas syringae DC3000 by targeting the auxin receptors TIR1, ABF2 and ABF3 [13]. [score:3]
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11
[+] score: 9
While the expressions of 14 families (miR156/miR157, miR158, miR160, miR162, miR165/miR166, miR168, miR169, miR171, miR390, miR393, miR394, miR396, miR398, and miR399) were dramatically reduced, 3 families (miR159, miR167, and miR172) were up-regulated in CsCl -treated seedlings. [score:6]
In response to nitrogen concentration, miR167 and miR393 involve in the regulation of root development and growth [20, 21]. [score:3]
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12
[+] score: 9
miR393 targets mRNAs encoding the auxin receptor, TRANSPORT INHIBITOR RESPONSE1 (TIR1), and related proteins [18], [43]. [score:5]
As predicted [43], miR393 was strongly up-regulated (10-fold at 3 hr p. i. ) (Figure 3C). [score:4]
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13
[+] score: 8
Besides miR393, two other miRNA families, miR160 and miR167, are upregulated following Pseudomonas syringae pv. [score:4]
For example, in Arabidopsis, miR393 negatively regulates auxin signaling pathways and contributes to PTI [15]. [score:2]
Zhang X Arabidopsis Argonaute 2 regulates innate immunity via miRNA393* -mediated silencing of a Golgi-localized SNARE gene, MEMB12Mol. [score:2]
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14
[+] score: 7
Leaf epinasty is often associated with high auxin levels [53], and is consistent with an increase of auxin signaling caused by downregulation of miR393 activity. [score:4]
MiR393 targets a small group of auxin receptor genes. [score:2]
TCP2, TCP3, TCP4, TCP10 TCP24, MYB33, MYB65, MYB81, MYB97, MYB104, MYB120 1, 2, 3, 4 MIM393 miR393 Narrow leaves, curled downward. [score:1]
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15
[+] score: 6
For example, AGO2-miR393* complexes regulate the expression of MEMBRIN 12 (MEMB12), which is required for resistance to Pseudomonas syringae in A. thaliana [35]. [score:4]
Similarly, miR393* reads were enriched 125 and 60 fold in HA-AGO2 [DAD] immunoprecipitates from mock-inoculated and TuMV-infected rosette leaves, respectively. [score:1]
MiR390 and miR393* were shown previously to co-immunoprecipitate with AGO2 [35, 52]. [score:1]
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16
[+] score: 5
Si-Ammour A. Win dels D. Arn-Bouldoires E. Kutter C. Ailhas J. Meins F. Vazquez F. miR393 and secondary siRNAs regulate expression of the TIR1/AFB2 auxin receptor clade and auxin-related development of Arabidopsis leaves Plant Physiol. [score:5]
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17
[+] score: 5
Another nitrate regulatory module, consisting of miR393 and the AFB3 auxin receptor has been shown to control root system architecture in response to external and internal nitrate availability [37]. [score:2]
B. miR393 is regulated by nitrate in qRT-PCR experiments. [score:2]
Using the same RNA samples and quantitative real time PCR, we were able to corroborate induction of miR393 (Figure  2B), a miRNA previously identified as nitrate responsive [37]. [score:1]
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18
[+] score: 5
Regarding the control exerted by miRNAs on phytohormones, it has been shown in Arabidopsis that auxin metabolism is controlled by at least four conserved miRNA families (miR160, miR167, miR390, and miR393), which mainly exert control by regulating ARF proteins (i. e., ARF6, ARF8, ARF10, ARF16, and 17) (Rhoades et al., 2002; Mallory et al., 2005; Marin et al., 2010; Win dels and Vazquez, 2011; Kinoshita et al., 2012). [score:2]
Specifically, the regulation of TIR1 and potentially three ARFs by the miR393 and miR167 families resulted conserved. [score:2]
miR393: integrator of environmental cues in auxin signaling? [score:1]
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19
[+] score: 3
Other miRNAs from this paper: ath-MIR393a
Also, using a transgenic A. thaliana line with suppressed auxin signaling (miR393), we have found that auxin signaling in plants is necessary for the growth promotion effects produced by the strain PsJN [62], which is well correlated with the Affymetrix results reported here. [score:3]
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20
[+] score: 3
The involvement of miRNAs as key regulators of flowering time (miR159, miR172, miR156, and miR171), hormone signaling (miR159, miR160, miR167, miR164, and miR393), or shoot and root development (miR164), was reviewed by (Wang and Li, 2007). [score:3]
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21
[+] score: 3
One group of miRNAs (miR160, miR167, miR390, miR393) is specifically related to auxin signalling (Zhang et al., 2011), which is linked to camalexin and glucosinolate biosynthesis. [score:1]
In addition, miR393 has a role in the plant immune response as it is induced following exposure to the PAMP flg22 (Navarro et al., 2006; Li et al., 2010), and following inoculation of both virulent and avirulent strains of P. syringae pv. [score:1]
It has also been reported that miR393 has a role in resource allocation between the glucosinolate and camalexin pathways (Robert-Seilaniantz et al., 2011). [score:1]
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22
[+] score: 2
The abundance of some miRNA (miR172 and miR397) induced by cold stresses increased in the OE lines but many other miRNAs (miR166, miR393, miR396 and miR408) induced by cold were unaltered [36]. [score:1]
Similarly, miRNAs responsive to bacterial (miR160, miR167, miR393, miR396, miR398 and miR825) and viral infections (miR156 and miR164) were not altered in the OE lines [33- 35]. [score:1]
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23
[+] score: 2
For example, when Arabidopsis was subjected to nitrate treatment, miR167 and miR393 were induced to modulate root development [41], [42]. [score:2]
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24
[+] score: 2
Other miRNAs from this paper: ath-MIR393a
Nitrate-responsive miR393/AFB3 regulatory module controls root system architecture in Arabidopsis thaliana. [score:2]
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25
[+] score: 2
Arabidopsis argonaute 2 regulates innate immunity via miRNA393(*) -mediated silencing of a golgi-localized SNARE gene, MEMB12. [score:2]
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26
[+] score: 2
Other miRNAs from this paper: ath-MIR393a
Another possible scenario might involve the redirection of secondary metabolism from camalexin to glucosinolates (or the reverse) by the fungi, as was recently reported following activation of the Arabidopsis gene miR393 [69]. [score:2]
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27
[+] score: 2
Other miRNAs from this paper: ath-MIR393a
This work also investigated the microRNA pathway and showed that the expression levels of miR393*, which associated with AGO2-IP and targets a transcript related to exocytosis, was enhanced in P. syringae infection assay [14]. [score:2]
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28
[+] score: 1
The abundance of miR160, miR168, miR170, miR393, miR395, miR408 and miR850 were specifically increased. [score:1]
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29
[+] score: 1
We also predicted several new miRNAs that are likely to respond to the TMV-Cg virus, including miR165 [36, 37], miR156 [34, 38], miR418, miR160 [36, 38], and miR393 [36, 37, 39]. [score:1]
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30
[+] score: 1
Other miRNAs from this paper: ath-MIR393a, zma-MIR393a, zma-MIR393b, zma-MIR393c
According to Vidal et al. [21], the microRNA miR393, which is inducibled by nitrate, affects only LR growth. [score:1]
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31
[+] score: 1
Other miRNAs from this paper: ath-MIR156a, ath-MIR156b, ath-MIR156c, ath-MIR156d, ath-MIR156e, ath-MIR156f, ath-MIR159a, ath-MIR160a, ath-MIR160b, ath-MIR160c, ath-MIR164a, ath-MIR164b, ath-MIR166a, ath-MIR166b, ath-MIR166c, ath-MIR166d, ath-MIR166e, ath-MIR166f, ath-MIR166g, ath-MIR167a, ath-MIR167b, ath-MIR168a, ath-MIR168b, ath-MIR171a, ath-MIR172a, ath-MIR172b, ath-MIR159b, ath-MIR319a, osa-MIR156a, osa-MIR156b, osa-MIR156c, osa-MIR156d, osa-MIR156e, osa-MIR156f, osa-MIR156g, osa-MIR156h, osa-MIR156i, osa-MIR156j, osa-MIR160a, osa-MIR160b, osa-MIR160c, osa-MIR160d, osa-MIR164a, osa-MIR164b, osa-MIR166a, osa-MIR166b, osa-MIR166c, osa-MIR166d, osa-MIR166e, osa-MIR166f, osa-MIR167a, osa-MIR167b, osa-MIR167c, osa-MIR171a, ath-MIR167d, ath-MIR172c, ath-MIR172d, ath-MIR393a, ath-MIR396a, ath-MIR396b, ath-MIR398a, osa-MIR393a, osa-MIR396a, osa-MIR396b, osa-MIR396c, osa-MIR398a, ath-MIR156g, ath-MIR156h, ath-MIR159c, ath-MIR164c, ath-MIR167c, ath-MIR172e, osa-MIR156k, osa-MIR156l, osa-MIR159a, osa-MIR159b, osa-MIR159c, osa-MIR159d, osa-MIR159e, osa-MIR159f, osa-MIR319a, osa-MIR160e, osa-MIR160f, osa-MIR164c, osa-MIR166k, osa-MIR166l, osa-MIR167d, osa-MIR167e, osa-MIR167f, osa-MIR167g, osa-MIR167h, osa-MIR167i, osa-MIR168a, osa-MIR168b, osa-MIR172a, osa-MIR172b, osa-MIR172c, osa-MIR166g, osa-MIR166h, osa-MIR166i, osa-MIR393b, osa-MIR172d, osa-MIR167j, osa-MIR166m, osa-MIR166j, osa-MIR437, osa-MIR396e, osa-MIR444a, osa-MIR528, osa-MIR531a, osa-MIR1425, osa-MIR444b, osa-MIR444c, osa-MIR444d, osa-MIR444e, osa-MIR444f, osa-MIR531b, osa-MIR1862a, osa-MIR1862b, osa-MIR1862c, osa-MIR1873, osa-MIR1862d, osa-MIR1862e, osa-MIR396f, osa-MIR396g, osa-MIR396h, osa-MIR396d, osa-MIR1862f, osa-MIR1862g, ath-MIR5021, osa-MIR5072, osa-MIR5077, ath-MIR156i, ath-MIR156j, osa-MIR531c
miR166 family with 56 member followed by miR159 (52 members) family were found to have maximum number of members in the library, although 14 miRNA families such as miR393, miR444, miR473, miR531, miR1425, miR1862, miR1873, miR3623, miR3634, miR5072, miR5077, miR7486, miR9662, and miR9674 were found to have only one member. [score:1]
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