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87 publications mentioning hsa-mir-23c

Open access articles that are associated with the species Homo sapiens and mention the gene name mir-23c. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 32
The major findings of this report are as follows: (1) a conserved set of miRNAs was identified that was consistently down-regulated in the migrating cell population relative to cells in the migration-restricted population, (2) miR-23b was significantly down regulated in migrating glioma cells in vitro and in cells from the invasive edge in patient tumor samples, (3) overexpression of miR-23b significantly inhibited glioma cell migration and invasion while inhibition of miR-23b expression significantly increased glioma cell migration, (4) predictive miRNA target analysis revealed a conserved miR-23b target site within the 3’ UTR of Pyk2, a non-receptor tyrosine kinase previously implicated in the regulation of glioma cell migration and invasion, (5) increased expression of miR-23b reduced the expression level of Pyk2 in glioma cells but did not significantly alter the expression level of the related focal adhesion kinase FAK, (6) expression of a Pyk2 transcript, devoid of the 3’UTR containing the miR23-b target sequence, in miR-23b over -expressing cells increased Pyk2 protein expression and partially rescued glioma cell migration. [score:32]
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2
[+] score: 26
Further downregulation of MALAT1 resulted in the decreased expression of ELAVL1, NLRP3, Caspase-1 and the pro-inflammatory cytokine IL-1β, and upregulated miR-23c expression and inhibited pyroptosis. [score:13]
These results suggested that upregulation of MALAT1 was promoting renal cell apoptosis and DN by targeting miR-23c and its downstream target EVAL1. [score:8]
The increased MALAT1 expression was associated with an increased pyroptosis of the renal epithelial cells and decreased expression of miR-23c. [score:5]
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3
[+] score: 22
Six out of ten miRNAs selected for validation were found significantly upregulated by anti-TNFα/DMARDs combination therapy (miR-16-5p, miR-23-3p, miR125b-5p, miR-126-3p, miRN-146a-5p, miR-223-3p). [score:4]
That data were further supported by ROC analyses, which showed that hsa-miR-23-3p and hsa-miR-223-3p levels at T1, with a cutoff value of 6.9 and 11.2 (relative expression at T1) respectively, were predictors of non-response to anti-TNFα/DMARD combination treatment (Figure  5B-C) with a sensitivity of 62.5% and 57.1%, and a specificity of 86.4% and 90.2% respectively. [score:3]
The miRNAs validated by RT-PCR in our cohort of patients (miR146a-5p, miR-16-5p, miR-23-3p, miR-125b-5p, miR223-3p; miR126-3p) have been previously reported to act as relevant regulators of immune cells development, playing crucial roles in the inflammatory response, and acting as key players in the pathogenesis of various chronic and autoimmune disorders, including RA itself [24]. [score:3]
Changed relative expression between T1 and T2 for miR-23, at a cutoff value of 0.83, demonstrated a sensitivity of 62.5% and a specificity of 77.6%. [score:3]
In our cohort, IL-6 receptors alpha and beta were found to be putative targets for three of the six validated miRNAs (hsa-miR-23-3p, hsa-miR-125b-5p, and hsa-miR223-3p). [score:3]
To improve accuracy of the analysis, we performed the combination of ROC curve analyses of miR-23, and miR-223. [score:1]
Further, we identified a specific plasma miRNA signature (miR-23 and miR-223) that may serve both as predictor and biomarker of response to anti-TNFα/DMARDs combination therapy. [score:1]
In total population, six of the ten miRNAs clearly distinguished RA serum samples after anti-TNFα/DMARDs combination therapy with high confidence level (P <0.05): (hsa-miR-125b, hsa-miR-126-3p, hsa-miR146a-5p, hsa-miR-16-5p, hsa-miR-23-3p, and hsa-miR-223-3p) all of them being increased after treatment (Figure  2A; Tables S3 and S4 in Additional file 1). [score:1]
The changes observed in three miRNAs (hsa-miR-146a-5p, hsa-miR-223-3p and hsa-miR-16-5p) significantly correlated with the changes observed in clinical parameters (that is, DAS28), and five of them at least with changes in inflammatory parameters such as CRP or ESR (hsa-miR-146a-5p, hsa-miR-223-3p,hsa-miR-16-5p, hsa- miR-126-3p and hsa-miR-23-3p) (Figure  4). [score:1]
Furthermore, ROC analyses demonstrated that two of these six miRNAs (hsa-miR-23-3p and hsa-miR-223-3p) can act in RA patients as both predictors of therapy response (indicating those patients who would not benefit from anti-TNFα/DMARDs combination therapy), and as biomarkers of response to anti-TNFα/DMARDs combination therapy (so that their levels would be indicative of treatment efficacy and also of the degree of response). [score:1]
ROC analysis showed the highest AUC for miR-23 and miR-223. [score:1]
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4
[+] score: 14
And the target gene possibly included TP53INP1 (tumor protein p53 inducible nuclear protein 1), TNFSF15 (tumor necrosis factor (ligand) superfamily, member 15) and TPD52 (tumor protein D52) for miR-203a-3p; TNFRSF11A (tumor necrosis factor receptor superfamily, member 11a, NFKB activator) for miR-6088; TUSC1 (tumor suppressor candidate 1), ZNF (zinc finger protein) and TP73 (tumor protein p73) for miR-1268b;WT1 (Wilms tumor 1), TPD52 (tumor protein D52), ST7L (suppression of tumorigenicity 7 like) and TUSC2 (tumor suppressor candidate 2) for miR-23c [18– 22]. [score:9]
As presented in Supplementary Tables 1 and 2, steviol treated cells exhibited different miRNA regulation, the most regulated as like miR-203a-3p (log2 =1.32) and miR-6088 (log2 =-2.54) in HCT-116, miR-1268b (log2 =19.85) and miR-23c (log2 =-2.05) in MKN-45. [score:3]
The steviol treated cancer cells presented remarkable miRNA regulation, such as miR-203a-3p (log2 =1.32), miR-6088 (log2 =-2.54) in HCT-116, miR-1268b (log2 =19.85), miR-23c (log2 =-2.05) in MKN-45. [score:2]
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5
[+] score: 14
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-27a, hsa-mir-23b, hsa-mir-27b
Since Ezh2 depletion upregulated TGF-β signaling thereby activating pro-EMT genes (Snail and Twist), it was intriguing to study if Ezh2 regulated the pro-EMT miR-23 cluster directly. [score:6]
To further confirm that Ezh2 regulates miR-23a and miR-27a expression in reprogramming, we monitored Ezh2 binding and enrichment of H3K27me3 marks on the miR-23 locus in hFibs at day 7 and Wt. [score:4]
Among those identified miR-23a and miR-27a that belong to miR-23 cluster were overrepresented following Ezh2 knockdown (Supplementary Fig. 5d). [score:2]
These results are consistent with our findings demonstrating that the miR-23 cluster is regulated by H3K27me3 activity of Ezh2 in human fibroblasts. [score:2]
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6
[+] score: 13
Inhibition of miR-23/27 effectively suppresses the development of retinal vasculature and is protective against laser -induced CNV in mice [53]. [score:6]
miR-23/27 directly target Sprouty2 and Sema6A, negative regulators of angiogenesis. [score:5]
The miR-23/27/24 clusters have also been implicated in regulating angiogenesis and choroidal neovascularization (CNV). [score:2]
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7
[+] score: 12
Up-regulated miRNAs (cfa-let-7, cfa-miR-200, cfa-miR-125, cfa-miR-34, cfa-miR-23, cfa-miR-146 clusters, cfa-miR-184 and cfa-miR-214) in adult canine testis treated with DMSO, RA or CYP26B1 inhibitor. [score:6]
MiRNA families such as miR-200 (cfa-miR-200a, cfa-miR-200b and cfa-miR-200c), Mirlet-7 (cfa-let-7a, cfa-let-7b, cfa-let-7c, cfa-let-7g and cfa-let-7f), miR-125 (cfa-miR-125a and cfa-miR-125b), miR-146 (cfa-miR-146a and cfa-miR-146b), miR-34 (cfa-miR-34a, cfa-miR-34b and cfa-miR-34c), miR-23 (cfa-miR-23a and cfa-miR-23b), cfa-miR-184, cfa-miR-214 and cfa-miR-141 were significantly up-regulated with testicular RA intervention via administration of CYP26B1 inhibitor and all-trans-RA (Figure 5). [score:6]
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8
[+] score: 11
We now demonstrate that the miR-23∼24∼27 cluster is up-regulated in CD8 [+]CD28 [−] T cells. [score:4]
This miRNA cluster, miR-23∼24∼27, has been shown to target molecules important for DNA repair (Lal et al., 2009; Srivastava et al., 2011). [score:3]
The second miRNA cluster, which is differentially expressed in CD8 [+]CD28 [−] T cells, is the miR-23∼24∼27 cluster. [score:3]
These miRNA clusters are miR-17∼92 and miR-23∼24∼27, the members of which are cleaved out of a polycistronic pri-miRNA-variant. [score:1]
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9
[+] score: 11
Among adult liver-enriched miRNAs, we confirmed a statistically-significant downregulation of transcripts that have predicted binding sites for the let-7/98, miR-22 and miR-23 seeds, which correspond to the miRNAs identified previously as expressed higher in the adult liver than in the embryo: let-7a, let-7b, let-7c, miR-22, and miR-23b. [score:6]
We observe that this score increases when targets of let-7/98 and miR-23 and miR-22 are considered together, suggesting that many genes are downregulated in the adult by the combined repression of several miRNA species. [score:5]
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10
[+] score: 11
Our 2011 study [46] was the first to show that miR-23 is downregulated in human AMD eyes and its putative protective effect is mediated by downregulation of the Fas ligand. [score:7]
We previously reported that miR-23 enhances RPE cell resistance to oxidative stress damage and is downregulated in macular RPE cells from AMD patients [46]. [score:4]
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11
[+] score: 11
However, other predicted miRNAs that also target the 3′UTR of il12A and il12B, including miR-590-5p, miR-340-5p, miR-23a, miR-23c and miR-494-3p, were not involved in the inhibition of IL-12 production in Omp25 -expressing THP-1 cells. [score:7]
In total, seven miRNA–mRNA target duplexes were predicted by these algorithms (Figure S3 in), including miR-590-5p, miR-21-5p, miR-340-5p, miR-23a, miR-23b, miR-23c, and miR-494-3p. [score:3]
Blocking of PD-1 signaling decreased miR-155, miR-21-5p and miR-23 levels and enhanced IL-12 production. [score:1]
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12
[+] score: 10
In the other hand, although miR-23a-5p was not predicted to target these transcripts, the 3p arm of miR-23 has been found by others, to be enriched in Tregs, and is predicted to target IL6ST 25, 33. [score:5]
More recently, the miR-23~27~24 cluster was shown to be a FOXP3 transcriptional target with roles in Treg biology; and the overexpression of the entire miR-23 cluster was shown to negatively impact the differentiation of Th1 and Th17 lineages [63]. [score:5]
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13
[+] score: 10
After determining the expression levels of these miRNAs in the same 7 pairs of NSCLC tissues and normal adjacent tissues, we observed that 8 miRNAs (miR-203, miR-30, let-7, miR-132, miR-181, miR-212, miR-101 and miR-9) were downregulated in the NSCLC tissues, while the other 5 miRNAs (miR-125, miR-98, miR-196, miR-23 and miR-499) were upregulated (Fig. S1). [score:9]
A total of 13 miRNAs, including miR-203, miR-30, let-7, miR-132, miR-181, miR-212, miR-101, miR-9, miR-125, miR-98, miR-196, miR-23 and miR-499, were identified as candidate miRNAs by all three computational algorithms (Table S2). [score:1]
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14
[+] score: 10
Moreover inconsistent expression of the miRNA members of miR-23 cluster has also been observed. [score:3]
Nevertheless, inconsistent expression of the miRNA members of miR-23 cluster has also been observed. [score:3]
The expression consistency of miRNAs in a miR-23 cluster has been shown in several studies (Table 1) but the cooperativity between them is yet to be worked out. [score:3]
miR-23 cluster paralogs. [score:1]
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15
[+] score: 9
Several studies examine the expression of miRNAs during neurodevelopment as well as in the adult CNS and reveal that certain miRNAs are found preferentially expressed in neurons, e. g., miR-124 and miR-128 [10], whereas others, e. g., miR-23, miR-26 and miR-29, seem restricted to astrocytes [11]. [score:6]
Another is miR-23, which, in agreement with the report of Smirnova et al. [11], was preferentially expressed in astrocytes. [score:3]
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16
[+] score: 9
In comparison with uninfected reads, highly expressed aae-mir-23, aae-mir-576 and aae-mir-320 were upregulated in CHIKV-infected Ae. [score:6]
Figures a) aae-miR-249 b) aae-miR-23 c) aal-miR-43b and d) aal-miR-5 show the predicted stem-loop structures, star and mature sequences of highly expressed novel microRNAs in Ae. [score:3]
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17
[+] score: 9
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-23b
In normoxia, c-Myc activation enhances the expression of some glycolytic genes such as HK2, PFK-M, ENO1 [159] and LDH-A [160], and is able to confer glutamine addiction by directly up -regulating the glutamine transporters ASCT2 and SLC7A25 and indirectly up -regulating enzyme glutaminase (GLS) through the inhibition of miR-23 [161]. [score:9]
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18
[+] score: 9
miR-31, miR-150 and miR-184 have shown to be downregulated in oxygen -induced retinopathy mice mo dels [20]; miR-23~24~27 cluster was upregulated in laser induced CNV mice mo dels [21]. [score:7]
Zhou Q. Gallagher R. Ufret-Vincenty R. Li X. Olson E. N. Wang S. Regulation of angiogenesis and choroidal neovascularization by members of microRNA-23∼27∼24 clusters Proc. [score:2]
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19
[+] score: 9
Similar inhibitory mechanisms (targeting MDV in this case) have been reported for miR-23, miR-378, and miR-505 (133). [score:5]
Conversely, increased replication of ALV-J was associated with the upregulation of miR-23 in the spleen of ALV-J-infected chickens. [score:4]
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20
[+] score: 8
Moreover, a microRNA can regulate the expression of multiple target genes; therefore, the miR-23 target genes that are relevant to osteoblast maturation and BMSC differentiation might be different. [score:8]
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21
[+] score: 8
Inhibition of miR-23 represses angiogenesis in vitro and postnatal retinal vascular development in vivo. [score:4]
Moreover, miR-23 is required for pathological angiogenesis in a laser -induced choroidal neovascularization mouse mo del by promoting angiogenic signaling through targeting Sprouty2 and Sema6A proteins [18]. [score:3]
It was reported miR-23 is enriched in endothelial cells and highly vascularized tissues. [score:1]
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22
[+] score: 7
It is however interesting to note that several miRNAs in addition to miRNA-210 i. e., miR-23, miR-24, miR-26a, miR-26b, miR-29a and miR-107 up-regulated through time course infection in our study were described as hypoxia-related [77], [78], negatively regulating HIF-1α through factor inhibiting-HIF-1α (FIH) [79] or induced by this TF [80]. [score:7]
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23
[+] score: 7
In addition, eight of the down-regulated miRNAs belong to the three paralog clusters miR-17/92, miR-106a/363 and miR-106b/25, while three of the up-regulated miRNAs are part of the miR-23/24 paralog clusters. [score:7]
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24
[+] score: 7
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-23b
Moreover, Lin ST et al. have shown that miR-23 is essential to regulate LMNB1 expression and to have a normal oligodendroglia development [85]. [score:5]
Thus, a perspective field of study is to highlight the potential disappearance of the regulation mediated by miR-23 in affected families. [score:2]
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25
[+] score: 7
A further confirmation on the role of mirRNAs in the regulation of mitochondrial biogenesis, glucose and fatty acid metabolism, derives from the study coducted by Safdar et al. [86] in mice, where the down-regulation of miR-23 is associated with a significant increase in PGC-1 αmRNA expression and protein content in quadriceps of C57Bl/6 J male animals three hours following an acute bout of endurance exercise. [score:7]
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26
[+] score: 7
ATP13A3 ATPase 13A3 N/A CDKN1A cyclin dependent kinase inhibitor 1A[33] GSK3B glycogen synthase kinase 3 beta[34] RAPH1 Ras association (RalGDS/AF-6) and pleckstrin homology domains 1 N/A SLC38A1 solute carrier family 38 member 1 N/A TET3 tet methylcytosine dioxygenase 3 N/A TP53INP1 tumor protein p53 inducible nuclear protein 1 N/A TSPAN14 tetraspanin 14 N/A Group C (miR-21, miR-23, miR-26a, miR-26b, miR-93, miR-223). [score:3]
In the control patient network, miR-23, miR-26a and miR-26b are strongly connected each through 11 edges; miR-93 and miR-155 are also strong connected to 10 other nodes through 10 edges. [score:1]
These nodes are represented by miR-26a, miR-26b, miR-23, miR-93 miR-146 and miR-155 for the control group. [score:1]
The C group is composed of six miRNAs–miR-23, miR-26a, miR-26b, miR-93, miR-223 and miR-21. [score:1]
To better illustrate our findings, we applied the principle of predator-prey like mathematical mo del to our groups of miRNAs, S = {miR-182, miR-146, miR-155, miR-16, miR-29a} and C = {miR-23, miR-26a, miR-26b, miR-93, miR-223, miR-21}. [score:1]
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27
[+] score: 7
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-16-1, hsa-mir-21, hsa-mir-23a, hsa-mir-27a, hsa-mir-29a, hsa-mir-101-1, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-16-2, hsa-mir-10a, hsa-mir-10b, hsa-mir-181a-2, hsa-mir-181b-1, hsa-mir-181c, hsa-mir-181a-1, hsa-mir-223, hsa-let-7g, hsa-let-7i, hsa-mir-15b, hsa-mir-23b, hsa-mir-27b, hsa-mir-146a, hsa-mir-150, hsa-mir-155, hsa-mir-181b-2, hsa-mir-29c, hsa-mir-101-2, hsa-mir-301a, hsa-mir-378a, hsa-mir-381, hsa-mir-340, hsa-mir-146b, hsa-mir-181d, hsa-mir-548a-1, hsa-mir-548b, hsa-mir-548a-2, hsa-mir-590, hsa-mir-548a-3, hsa-mir-548c, hsa-mir-548d-1, hsa-mir-548d-2, hsa-mir-378d-2, hsa-mir-301b, hsa-mir-548e, hsa-mir-548j, hsa-mir-548k, hsa-mir-548l, hsa-mir-548f-1, hsa-mir-548f-2, hsa-mir-548f-3, hsa-mir-548f-4, hsa-mir-548f-5, hsa-mir-548g, hsa-mir-548n, hsa-mir-548m, hsa-mir-548o, hsa-mir-548h-1, hsa-mir-548h-2, hsa-mir-548h-3, hsa-mir-548h-4, hsa-mir-548p, hsa-mir-548i-1, hsa-mir-548i-2, hsa-mir-548i-3, hsa-mir-548i-4, hsa-mir-548q, hsa-mir-548s, hsa-mir-378b, hsa-mir-548t, hsa-mir-548u, hsa-mir-548v, hsa-mir-548w, hsa-mir-548x, hsa-mir-378c, hsa-mir-548y, hsa-mir-548z, hsa-mir-548aa-1, hsa-mir-548aa-2, hsa-mir-548o-2, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-548h-5, hsa-mir-548ab, hsa-mir-378f, hsa-mir-378g, hsa-mir-548ac, hsa-mir-548ad, hsa-mir-548ae-1, hsa-mir-548ae-2, hsa-mir-548ag-1, hsa-mir-548ag-2, hsa-mir-548ah, hsa-mir-378h, hsa-mir-548ai, hsa-mir-548aj-1, hsa-mir-548aj-2, hsa-mir-548x-2, hsa-mir-548ak, hsa-mir-548al, hsa-mir-378i, hsa-mir-548am, hsa-mir-548an, hsa-mir-548ao, hsa-mir-548ap, hsa-mir-548aq, hsa-mir-548ar, hsa-mir-548as, hsa-mir-548at, hsa-mir-548au, hsa-mir-548av, hsa-mir-548aw, hsa-mir-548ax, hsa-mir-378j, hsa-mir-548ay, hsa-mir-548az, hsa-mir-548ba, hsa-mir-548bb, hsa-mir-548bc
In particular, both HHV-6A and 6B induced an early up-regulation of miR-301a and miR-548e (1 d. p. i. ), an increase of miR-101 and a decrease of miR-let-7c and miR-340 at 3 d. p. i., and a down-regulation of miR-23 at late time-points p. i. (6 d. p. i. ). [score:7]
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28
[+] score: 6
The authors showed that estradiol inhibits miR-23 -dependent downregulation of connexin 43 in a menopausal rat mo del, and provide new mechanisms of post-menopause-related arrhythmia [99]. [score:6]
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29
[+] score: 6
In the array, miR-23b, another member of the miR-23–27–24 clusters, was also significantly up- and downregulated by H [2]O [2] and curcumin, respectively. [score:4]
The miR-23–27–24 clusters enhance angiogenesis and choroidal neovascularization in mice by repressing sprouty2 and Sema6a proteins, which negatively regulate MAPK and VEGFR2 signaling in response to angiogenic factors [44]. [score:2]
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30
[+] score: 6
Interestingly, we found upregulated expression of miR-23 (∼twofold) to coincide with the reduction in TRF2 levels in replicative senescent BJ (PD88) and MRC-5 cells (PD58) (Fig. 3F and Supplemental Figure S1B). [score:6]
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31
[+] score: 6
Microarray analysis showed altered expression of some miRNAs in hepatomas such as let-7a, miR-21, miR-23, miR-130, whereas the hepato-specific miR-122a and others were found downregulated in 70% of HCCs and in HCC-derived cell lines [20], [46], [47], as reported in our data (Table 1). [score:6]
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32
[+] score: 6
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-23b, hsa-mir-155
Furthermore, other transcription suppressors that directly target the promoters of bic or a miR-23∼24∼27 cluster could also participate in the transcriptional repression of these precursor genes since the pri-miR-155 level was detected to correlate with miR-155 reduction (Fig. S1B,C). [score:6]
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33
[+] score: 6
The profiling of the adipose tissue-derived exosomes isolated from lean and obese individuals showed the differential expression of 88 miRNAs with significant upregulation of miR-23-b and miR-4429. [score:6]
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34
[+] score: 6
0114779.g003 Figure 3Three kinds of human colon cancer cell lines, HT29 (A), DLD1 (B) and HCT116 (C), were treated as described in Fig. 2. qRT-PCR was performed to validate the alteration of the expression of hsa-miR-302a-3p, hsa-miR-548ah-5p, hsa-miR-30a-5p, hsa-miR-23-3p, hsa-miR-195a-5p and hsa-let-7c-5p under 5-FU treatment (5-FU) and starvation. [score:3]
Three kinds of human colon cancer cell lines, HT29 (A), DLD1 (B) and HCT116 (C), were treated as described in Fig. 2. qRT-PCR was performed to validate the alteration of the expression of hsa-miR-302a-3p, hsa-miR-548ah-5p, hsa-miR-30a-5p, hsa-miR-23-3p, hsa-miR-195a-5p and hsa-let-7c-5p under 5-FU treatment (5-FU) and starvation. [score:3]
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35
[+] score: 5
miR-124 and miR-128 are primarily expressed in neurons, whereas miR-23, miR-26, and miR-29 are expressed in high amounts in astrocytes [37]. [score:5]
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36
[+] score: 5
In human gastric cancer, multiple miRNAs with aberrant expression have been identified, for example, miR-21, miR-22, miR-23, miR-127, miR-148, miR-202 and miR-433, which played oncogenic or suppressive role [20]– [25]. [score:5]
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37
[+] score: 5
There is only one report of successful therapeutic targeting of micro -RNAs in ocular disease, involving silencing mir-23 and mir-27 in CNV [14]. [score:5]
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38
[+] score: 5
In contrast, miR-23 inhibition in vivo did not alter liver ABCA1 expression despite the results achieved in vitro [33]. [score:5]
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39
[+] score: 5
ZNF91 belongs to a C [2]H [2] zinc finger (ZNF) gene family that is greatly expanded in the primate lineage and known to contain exceptionally abundant target sites for several miRNA families, including miR-23, miR-181 and miR-199 [28]. [score:3]
In particular, a circRNA generated from the ZNF91 locus (circRNA-ZNF91) contains 24 miR-23 sites (Figure  6E), 19 of which were 8-nucleotide sites. [score:1]
miR-23 and miR-296 seed matches are indicated. [score:1]
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40
[+] score: 5
Since miR-23 (JX994633), miR-218 (JX994383) and miR-338 (JX994406) are present in the elephant shark, these conserved miRNAs are likely to be involved in the regulation of Runx2 expression in elephant shark, possibly during chondrogenic differentiation through mechanisms similar to that in other jawed vertebrates. [score:4]
Of these, binding sites for miR-23, miR-218 and miR-338 were found conserved in the 3′UTR of elephant shark Runx2, while only that for miR-28 is present in zebrafish and fugu Runx2 (Fig. 8B). [score:1]
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41
[+] score: 5
Other miRNAs from this paper: hsa-mir-23a, mmu-mir-23b, hsa-mir-23b, mmu-mir-23a
For example, it has been previously shown that miR-23 a suppresses the translation of both MAFbx/atrogin-1 and MuRF-1 in a 3_-UTR -dependent manner (Wada et al., 2011). [score:5]
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42
[+] score: 5
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-26a-1, hsa-mir-26b, hsa-mir-23b, hsa-mir-26a-2
D shows that miR-23 overexpression increase tumor response to etoposide. [score:3]
Neither delay on tumorigenesis nor reduced end-point tumor size could be found in miR-23 -overexpressed xenograft in compared with wild-type (Figure  2B). [score:2]
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43
[+] score: 4
Moreover, two seed families, the miR-23 family (miR-23a-3p, miR-23b-3p) and miR-27 family (miR-27a-3p, miR-27b-3p), were upregulated. [score:4]
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44
[+] score: 4
As miR-23 is transcribed together with miR-24 and miR-27 in a polycistronic cluster, these results support our observation that expression of miR-27a increases in the hEC line (NCCIT) 7 days post RA treatment (Figure 4A). [score:3]
Both paralogs are transcribed within a polycystronic cluster together with miR-23 and miR-24. [score:1]
[1 to 20 of 2 sentences]
45
[+] score: 4
Among these miRNAs, the expression of the miR-23/27/24 cluster is involved in angiogenesis and endothelial apoptosis in cardiac ischemia and retinal vascular development (Bang et al., 2012). [score:4]
[1 to 20 of 1 sentences]
46
[+] score: 4
Moreover, the KSHV-encoded miRNAs that regulate relatively more human targets, such as miR-K12-10a and miR-K12-3, are also the ortholog of host hsa-miR-142-3p and hsa-miR-23 [38], respectively. [score:4]
[1 to 20 of 1 sentences]
47
[+] score: 4
The components of the 14-miRNA signature can be classified in five expression clusters: miR-16, miR-17-92 cluster and paralogs, miR-23/24/27a, miR-331 and miR-660. [score:3]
Finally, miR-27, part of the miR-23/24/27a cluster, activates the Wnt signaling pathway, affecting the differentiation of mesenchymal stem cells into osteoblasts [35]. [score:1]
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48
[+] score: 4
Deregulation of a number of miRNAs in our study have been previously reported in right and left atrial appendages of patients with rheumatic mitral valve disease (RMVD), including miR-222-3p, miR-4484 and miR-940 in left atrial appendage [41], miR-5190 and miR-23c in right atrial appendages [42] and miR-143-3p in both, in the LAA and RAA of patients with RMVD [43]. [score:4]
[1 to 20 of 1 sentences]
49
[+] score: 4
To determine the effects of miRNA-23 downregulation on NPC radioresistance, miRNA-23a mimic and mimic control (RiboBio) were transfected into the CNE2-IR cells using riboFect™ CP transfection kit (RiboBio) according to manufacturer's instructions, respectively. [score:4]
[1 to 20 of 1 sentences]
50
[+] score: 3
We identified one miRNA-gene, MIR4654, spanning copy loss at 1q23.3 and three miRNA target sites (mir-368, mir-23 and mir-1/206) spanning 3′ UTR of CNV-gene SEC22B at 1q21.1 (gain). [score:3]
[1 to 20 of 1 sentences]
51
[+] score: 3
For example, has-miR-126-3p has been shown to be associated with the coronary heart disease and atherosclerotic lesions, has-miR-221-5p has been associated with SMC de-differentiation, and has-miR-23a-3p, has-miR-23b-3p, has-miR-23c, has-miR-27a-3p, and has-miR-27b-3p have been shown to be involved with the progression of angiogenesis (Table 1). [score:3]
[1 to 20 of 1 sentences]
52
[+] score: 3
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-205, hsa-mir-23b, hsa-mir-200c
IRF1 is a tumor suppressor modulated by miR-23, promoting TGF-beta -induced EMT in lung cancer [45]. [score:3]
[1 to 20 of 1 sentences]
53
[+] score: 3
MB designed to detect miR-23 served as NC and denatured MB (Den MB) served as a positive control (* p <0.05). [score:1]
miRNA-23 has a completely irrelevant sequence to the let-7b, and thus a MB designed to detect miR-23 served as a NC. [score:1]
Additionally, the negative control group (100 pM MB designed to detect miR-23 was used instead of MB for let-7b) did not produce significant fluorescence with the presence of let-7b mimic, suggesting the MB for let-7b was indeed specific for detecting let-7b. [score:1]
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54
[+] score: 3
DNMT1 mediated the HPV-16 E6 suppression of miR-23. [score:3]
[1 to 20 of 1 sentences]
55
[+] score: 3
Interestingly, in another epithelial cell mo del it was shown that miR-19, miR-23 and miR-24 contribute to lower cell growth when inhibited [33]. [score:3]
[1 to 20 of 1 sentences]
56
[+] score: 3
Interestingly, exosomes derived from metastatic-cancers might also display high expression for anti-cancer miRNAs such as hsa-let-7, hsa-miR-23, hsa-miR-224 and hsa-miR-921 64, which is believed as an internal reformation technique of the cancer cells to retain malignant properties by sacking the promising threats outside. [score:3]
[1 to 20 of 1 sentences]
57
[+] score: 3
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-23b
Lin S. T. Fu Y. H. (2009) miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination. [score:3]
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58
[+] score: 3
Bioinformatics analysis identified GSK-3β as a target of both miR-23 miRNAs. [score:3]
[1 to 20 of 1 sentences]
59
[+] score: 3
Additionally, Few X linked miRNAs like hsa-mir-23c, hsa-miR-222-5p, hsa-miR-224–3p, hsa-miR-767-5p and hsa-miR-6089 were also observed to be differentially expressed. [score:3]
[1 to 20 of 1 sentences]
60
[+] score: 3
MicroRNA-23 restricts cardiac valve formation by inhibiting Has2 and extracellular hyaluronic acid production. [score:2]
In a zebrafish dicer mutant lacking mature miRNAs, endocardial cushion formation was excessive, prompting the observation that miRNA-23 is necessary to restrict the number of cells that differentiate into endocardial cushion cells (Lagendijk et al., 2011). [score:1]
[1 to 20 of 2 sentences]
61
[+] score: 3
Other miRNAs from this paper: hsa-mir-23a, mmu-mir-23b, hsa-mir-23b, mmu-mir-23a
LMNB1 is negatively regulated by miR-23, which is also implicated in ADLD variants and it has been proposed that an over representation of lamin B1 mRNA sequesters miR-23 leading to disturbances in myelin protein production [60,66]. [score:2]
Future studies should focus on lamin B1 interactions (e. g. with miR-23) which will form the basis of understand the pathways that do depend on these lamins. [score:1]
[1 to 20 of 2 sentences]
62
[+] score: 3
Other miRNAs from this paper: hsa-mir-16-1, hsa-mir-17, hsa-mir-20a, hsa-mir-21, hsa-mir-23a, hsa-mir-100, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-107, hsa-mir-16-2, mmu-mir-1a-1, mmu-mir-23b, mmu-mir-125b-2, mmu-mir-130a, mmu-mir-9-2, mmu-mir-145a, mmu-mir-181a-2, mmu-mir-184, mmu-mir-199a-1, hsa-mir-199a-1, mmu-mir-205, mmu-mir-206, hsa-mir-181a-2, hsa-mir-181b-1, hsa-mir-199a-2, hsa-mir-205, hsa-mir-181a-1, hsa-mir-214, hsa-mir-219a-1, hsa-mir-223, mmu-mir-302a, hsa-mir-1-2, hsa-mir-23b, hsa-mir-125b-1, hsa-mir-130a, hsa-mir-145, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-125b-2, hsa-mir-184, hsa-mir-206, mmu-mir-16-1, mmu-mir-16-2, mmu-mir-20a, mmu-mir-21a, mmu-mir-23a, mmu-mir-103-1, mmu-mir-103-2, rno-mir-338, mmu-mir-338, rno-mir-20a, hsa-mir-1-1, mmu-mir-1a-2, hsa-mir-181b-2, mmu-mir-107, mmu-mir-17, mmu-mir-100, mmu-mir-181a-1, mmu-mir-214, mmu-mir-219a-1, mmu-mir-223, mmu-mir-199a-2, mmu-mir-9-1, mmu-mir-9-3, mmu-mir-181b-1, mmu-mir-125b-1, hsa-mir-302a, hsa-mir-219a-2, mmu-mir-219a-2, hsa-mir-302b, hsa-mir-302c, hsa-mir-302d, hsa-mir-367, hsa-mir-372, hsa-mir-338, mmu-mir-181b-2, rno-mir-9a-1, rno-mir-9a-3, rno-mir-9a-2, rno-mir-16, rno-mir-17-1, rno-mir-21, rno-mir-23a, rno-mir-23b, rno-mir-100, rno-mir-103-2, rno-mir-103-1, rno-mir-107, rno-mir-125b-1, rno-mir-125b-2, rno-mir-130a, rno-mir-145, rno-mir-181a-2, rno-mir-181b-1, rno-mir-181b-2, rno-mir-184, rno-mir-199a, rno-mir-205, rno-mir-206, rno-mir-181a-1, rno-mir-214, rno-mir-219a-1, rno-mir-219a-2, rno-mir-223, hsa-mir-512-1, hsa-mir-512-2, rno-mir-1, mmu-mir-367, mmu-mir-302b, mmu-mir-302c, mmu-mir-302d, rno-mir-17-2, hsa-mir-1183, mmu-mir-1b, hsa-mir-302e, hsa-mir-302f, hsa-mir-103b-1, hsa-mir-103b-2, rno-mir-9b-3, rno-mir-9b-1, rno-mir-9b-2, rno-mir-219b, hsa-mir-219b, mmu-mir-145b, mmu-mir-21b, mmu-mir-21c, mmu-mir-219b, mmu-mir-219c, mmu-mir-9b-2, mmu-mir-9b-1, mmu-mir-9b-3
Another report demonstrates that miR-23 facilitates OL development by negatively regulating lamin B1 (LMNB1), a protein found to repress production of MBP, proteolipid protein 1 (PLP), and myelin oligodendrocyte glycoprotein (MOG) [19]. [score:3]
[1 to 20 of 1 sentences]
63
[+] score: 3
miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination. [score:3]
[1 to 20 of 1 sentences]
64
[+] score: 3
More refined cellular specificity in their expression was reported for astrocytes (miR-23, -26, -29), neurons (miR-124, -128) [127], and hippocampal neurons (let-7 family) [50]. [score:3]
[1 to 20 of 1 sentences]
65
[+] score: 2
Zhou Q Regulation of angiogenesis and choroidal neovascularization by members of microRNA-23~27~24 clustersProc. [score:2]
[1 to 20 of 1 sentences]
66
[+] score: 2
From these, only six (miR-16-5p, miR-23-3p, miR-125b-5p, miR-126-3p, miR-146a-5p, and miR-223-3p) reached significance with high confidence levels, and all of them were elevated exclusively in responders to treatment. [score:1]
Additionally, after performing ROC curve analysis, two miRNAs were identified to have the highest area under the curve (miR-23 and miR-223) and furthermore, when analyzed in combination, demonstrated a higher sensitivity and specificity to identify responders to treatment, in relation to the identification made with other miRNAs alone. [score:1]
[1 to 20 of 2 sentences]
67
[+] score: 2
In pericardial fluid, miR-126–3p, miR-23~27~24 cluster miRNAs, miR-221–3p and miR-222–3p were present in relatively high amounts, whereas miR-17~92 cluster miRNAs were detected sporadically (Table 6). [score:1]
Endothelial cell enriched miRNAs include miR-126–3p, miR-17~92 cluster (miR-17–5p/-3p, miR-18a-5p, miR-19a/b-3p, miR-20a-5p, miR-92a-3p), miR-23~27~24 clusters (miR-23a/b-3p, miR-24–3p, miR-27a/b-3p), miR-221–3p and miR-222–3p [28]. [score:1]
[1 to 20 of 2 sentences]
68
[+] score: 2
An anti-sense sequence of the selected mRNA region followed by a miR23 loop of 10 nucleotides (CTTCCTGTCA) was added at the 5′ end of the above sequences. [score:1]
The miR23 loop facilitates the transfer of the hairpin RNA out of the nucleus. [score:1]
[1 to 20 of 2 sentences]
69
[+] score: 2
Lastly, kshv-miR-K12-3 was found to share seed sequence homology with hsa-miR-23. [score:1]
Manzano M. Shamulailatpam P. Raja A. N. Gottwein E. Kaposi’s sarcoma -associated herpesvirus encodes a mimic of cellular miR-23 J. Virol. [score:1]
[1 to 20 of 2 sentences]
70
[+] score: 2
Manzano M. Shamulailatpam P. Raja A. N. Gottwein E. Kaposi’s sarcoma -associated herpesvirus encodes a mimic of cellular miR-23 J. Virol. [score:1]
Interestingly, several KSHV-miRNAs, including miR-K12-11, miR-K12-10a and miR-K12-3, can act as viral analogs of the human cellular miRNAs miR-155, miR-142-3p and miR-23, respectively. [score:1]
[1 to 20 of 2 sentences]
71
[+] score: 2
We designed and cloned into the pcPURhU6 vector the hairpin-type RNAs with si-6 sequence (pcPURhU6 si-6) with the 19-21 base pair (bp) stems and with various loops: (1) pcPURhU6 si-6 (21 bp)-miR26, (2) si-6 (19 bp) with 9-nt UUCAAGAGA loop [28], (3) si-6 (21 bp) with 9-nt UUCAAGAGA loop, (4) si-6 (21 bp) with 10-nt CUUCCUGUCA (loop from miRNA23), and (5) si-6 (21 bp) with 19-nt UAGUGAAGCCACAGAUGUA (loop from miRNA30) (see Figure 3). [score:1]
Constructs 4 and 5 with miRNA-origin loops miRNA23 and miRNA30, respectively, demonstrated moderate silencing activity, lowering BACE1 mRNA by 27% and 38%, respectively. [score:1]
[1 to 20 of 2 sentences]
72
[+] score: 2
From the top 30 miRNAs, we found six miRNAs (e. g., miR-23a, miR-27b, miR-103, miR-200a/b/c) to be related to lipid metabolism in human adipocyte cells: miR-23 enhances glutamine metabolism [38]; miR-27 decreases fat accumulation [27]; miR-103 regulates triglyceride content during cell differentiation [39]; and miR-200 affects insulin signaling [40]. [score:2]
[1 to 20 of 1 sentences]
73
[+] score: 2
From the remaining deregulated miRNAs, the most significant were hsa-miR-23/27 cluster and hsa-let-7, hsa-miR-30, and hsa-miR-376 families. [score:2]
[1 to 20 of 1 sentences]
74
[+] score: 2
MiR-23/27/24 clusters were involved in angiogenesis and endothelial apoptosis, which has potential therapeutic applications in both of vascular disorders and ischemic heart disease [19]. [score:2]
[1 to 20 of 1 sentences]
75
[+] score: 1
org/), a set of miRNAs were predicted to have potential interaction with circUBAP2, including miR-150, miR-135, miR-101, miR-181, miR-23, miR-149, miR-139, miR-491, miR-124, miR-301m miR-328, miR-122, miR-186, let-7, miR-132, miR-191, miR-425, miR-125, miR-149, miR-143, and miR-146a. [score:1]
[1 to 20 of 1 sentences]
76
[+] score: 1
miR-24 is clustered with miR-23 and miR-27 and has been associated with T2DM, heart, and β-cell function [130]. [score:1]
[1 to 20 of 1 sentences]
77
[+] score: 1
MiR-24 clusters with two other miRNAs, miR-23 and miR-27, on chromosome 9 (cluster-1: miR-23b, miR-27b and miR-24-1) and on chromosome 19 (cluster-1: miR-23a, miR-27a and miR-24-2). [score:1]
[1 to 20 of 1 sentences]
78
[+] score: 1
Effect of miR-23 on oxidant -induced injury in human retinal pigment epithelial cells. [score:1]
[1 to 20 of 1 sentences]
79
[+] score: 1
For certain miRNAs, such as miR-23c, editing could be seen in 100% of miRNA pool (Additional file 4: Table S6). [score:1]
[1 to 20 of 1 sentences]
80
[+] score: 1
The authors concluded that CDR1as which contains 71 binding sites for miR-7 is an exception and the next best miRNA sponge is circRNA ZNF91 which has 24 binding sites for miR-23 (25). [score:1]
[1 to 20 of 1 sentences]
81
[+] score: 1
To date, the miR-23, miR-125, miR-143, miR-145, miR-4454, and miR-4484 have not been described as participating in AF pathology. [score:1]
[1 to 20 of 1 sentences]
82
[+] score: 1
For example let-7e, miR-23-c and miR-602 are each found in three samples under one condition but not in any sample under any of the other conditions. [score:1]
[1 to 20 of 1 sentences]
83
[+] score: 1
The activity of the immunoprecipitated protein was verified using the miRNA-23a∼27a∼24-2 cluster transcribed in vitro from plasmid pGEM-T-easy_pri-miR-23, 27, 24-2(+) (also provided by V. N. Kim). [score:1]
[1 to 20 of 1 sentences]
84
[+] score: 1
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-16-1, hsa-mir-17, hsa-mir-19a, hsa-mir-19b-1, hsa-mir-19b-2, hsa-mir-23a, hsa-mir-26a-1, hsa-mir-26b, hsa-mir-27a, hsa-mir-29a, hsa-mir-30a, hsa-mir-31, hsa-mir-100, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-16-2, mmu-mir-23b, mmu-mir-27b, mmu-mir-29b-1, mmu-mir-30a, mmu-mir-30b, mmu-mir-127, mmu-mir-128-1, mmu-mir-132, mmu-mir-133a-1, mmu-mir-188, mmu-mir-194-1, mmu-mir-195a, mmu-mir-199a-1, hsa-mir-199a-1, mmu-mir-200b, mmu-mir-205, mmu-mir-206, hsa-mir-30c-2, hsa-mir-30d, mmu-mir-122, mmu-mir-30e, hsa-mir-199a-2, hsa-mir-199b, hsa-mir-205, hsa-mir-211, hsa-mir-212, hsa-mir-214, hsa-mir-217, hsa-mir-200b, hsa-mir-23b, hsa-mir-27b, hsa-mir-30b, hsa-mir-122, hsa-mir-128-1, hsa-mir-132, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-127, hsa-mir-138-1, hsa-mir-188, hsa-mir-194-1, hsa-mir-195, hsa-mir-206, mmu-mir-19b-2, mmu-mir-30c-1, mmu-mir-30c-2, mmu-mir-30d, mmu-mir-200a, mmu-let-7a-1, mmu-let-7a-2, mmu-let-7f-1, mmu-let-7f-2, mmu-mir-16-1, mmu-mir-16-2, mmu-mir-23a, mmu-mir-26a-1, mmu-mir-26b, mmu-mir-29a, mmu-mir-29c, mmu-mir-27a, mmu-mir-31, mmu-mir-351, hsa-mir-200c, mmu-mir-17, mmu-mir-19a, mmu-mir-100, mmu-mir-200c, mmu-mir-212, mmu-mir-214, mmu-mir-26a-2, mmu-mir-211, mmu-mir-29b-2, mmu-mir-199a-2, mmu-mir-199b, mmu-mir-19b-1, mmu-mir-138-1, mmu-mir-128-2, hsa-mir-128-2, mmu-mir-217, hsa-mir-194-2, mmu-mir-194-2, hsa-mir-29c, hsa-mir-30c-1, hsa-mir-200a, hsa-mir-30e, hsa-mir-26a-2, hsa-mir-379, mmu-mir-379, mmu-mir-133a-2, mmu-mir-133b, hsa-mir-133b, mmu-mir-412, mmu-mir-431, hsa-mir-431, hsa-mir-451a, mmu-mir-451a, mmu-mir-467a-1, hsa-mir-412, hsa-mir-485, hsa-mir-487a, hsa-mir-491, hsa-mir-503, hsa-mir-504, mmu-mir-485, hsa-mir-487b, mmu-mir-487b, mmu-mir-503, hsa-mir-556, hsa-mir-584, mmu-mir-665, mmu-mir-669a-1, mmu-mir-674, mmu-mir-690, mmu-mir-669a-2, mmu-mir-669a-3, mmu-mir-669c, mmu-mir-696, mmu-mir-491, mmu-mir-504, hsa-mir-665, mmu-mir-467e, mmu-mir-669k, mmu-mir-669f, hsa-mir-664a, mmu-mir-1896, mmu-mir-1894, mmu-mir-1943, mmu-mir-1983, mmu-mir-1839, mmu-mir-3064, mmu-mir-3072, mmu-mir-467a-2, mmu-mir-669a-4, mmu-mir-669a-5, mmu-mir-467a-3, mmu-mir-669a-6, mmu-mir-467a-4, mmu-mir-669a-7, mmu-mir-467a-5, mmu-mir-467a-6, mmu-mir-669a-8, mmu-mir-669a-9, mmu-mir-467a-7, mmu-mir-467a-8, mmu-mir-669a-10, mmu-mir-467a-9, mmu-mir-669a-11, mmu-mir-467a-10, mmu-mir-669a-12, mmu-mir-3473a, hsa-mir-4436a, hsa-mir-4454, mmu-mir-3473b, hsa-mir-4681, hsa-mir-3064, hsa-mir-4436b-1, hsa-mir-4790, hsa-mir-4804, hsa-mir-548ap, mmu-mir-3473c, mmu-mir-5110, mmu-mir-3473d, mmu-mir-5128, hsa-mir-4436b-2, mmu-mir-195b, mmu-mir-133c, mmu-mir-30f, mmu-mir-3473e, hsa-mir-6825, hsa-mir-6888, mmu-mir-6967-1, mmu-mir-3473f, mmu-mir-3473g, mmu-mir-6967-2, mmu-mir-3473h
Hcy also induces alteration of miRNAs related to tight junctions signaling such as miR-128, miR-132, miR-133, miR-195, miR-3473, miR-19, miR-200, miR-205, miR-214, miR-217, miR-23, miR-26, miR-29, miR-30, miR-31 AND miR-690. [score:1]
[1 to 20 of 1 sentences]
85
[+] score: 1
Other miRNAs from this paper: hsa-mir-23a, hsa-mir-23b
A sequence of GCTTCCTGTCAC derived from miR-23 [47] was used as a shRNA loop. [score:1]
[1 to 20 of 1 sentences]
86
[+] score: 1
We also showed that hypermethylated in cancer 1 (HIC1) and miR-23~27~24 clusters form a double -negative feedback loop in breast cancer. [score:1]
[1 to 20 of 1 sentences]
87
[+] score: 1
miR-24, miR-223, miR-23 and miR-199a show 100% conservation of seed region sequences between humans and mice [23]. [score:1]
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