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9 publications mentioning rno-mir-542-3

Open access articles that are associated with the species Rattus norvegicus and mention the gene name mir-542-3. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

[+] score: 45
miR-542-3p suppresses osteoblast cell proliferation and differentiation, targets BMP-7 signaling and inhibits bone formation. [score:7]
miR-542-3p inhibits colorectal cancer cell proliferation, migration and invasion by targeting OTUB1. [score:5]
Since miR-21-3p is related to myocardial hypertrophy and miR-542-3p is regulated to p53 expression and more importantly, both of them were regulated by YQFM, we hypothesized that YQFM might have the effects against cardiac hypertrophy and apoptosis. [score:5]
MicroRNA-542-3p suppresses tumor cell invasion via targeting akt pathway in human astrocytoma. [score:4]
Based on these findings, we presumed that YQFM could alleviate hypertrophy and apoptosis in heart tissue during CHF in order to maintain heart function by regulating the miRNAs expression such as miR-21-3p and miR-542-3p. [score:4]
Down-regulation of mir-542-3p promotes neointimal formation in the aging rat. [score:4]
miR-542-3p could modulate expression level of p53 (Wang et al., 2014), cell apoptosis and was connected with cancer (Wang et al., 2016c), ischemic stroke (He et al., 2016), liver failure (Ding et al., 2015), neointimal formation (Qian et al., 2015), diabetic cardiomyopathy and osteogenesis (Chavali et al., 2014; Kureel et al., 2014). [score:3]
Figure 5MiR-21-3p and miR-542-3p expression for validation of miRNA chip (n = 5). [score:3]
MiR-542 has been reported to inhibit the cancer cells from proliferation, invasion and migration (Cai et al., 2015; Yuan et al., 2017). [score:2]
p53 is positively regulated by miR-542-3p. [score:2]
The close relationship between these miRNAs especially for miR-21-3p and miR-542 and myocardial function drew our attention. [score:1]
MiR-21-3p and miR-542-3 were further verified by RT-PCR. [score:1]
However, the p-values of miR-21-3p and miR-542-3p between YQFM treated and MI group are 0.11 and 0.40, respectively, which is not statistical significant. [score:1]
Among them, miR-21-3p, miR216-5p, and miR542-3p are related to myocardial hypertrophy, cancer and apoptosis, respectively. [score:1]
MiR-21-3p and miR-542-3p were two important altered miRNAs found in YQFM treated group according to miRNA profiling study. [score:1]
Finally, as Table 1 showed, seven differentially expressed miRNAs were picked out complying with fold change (fc)≥4 (or ≤1/4) (both Sham group and YQFM group compared to MI group) and Reverse Rate (RR) (YQFM group compared to MI group) between 1 and 2. Finally, miR-21-3p, miR216-5p, miR219a-2-3p, miR381-3p, miR466c-5p, miR542-3p, and miR-702-5p were considered as the differentially reversed miRNAs regulated by YQFM. [score:1]
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[+] score: 14
In addition, Yoon et al. (2010) suggested that survivin is a direct target of miR-542-3p and growth inhibition by miR-542-3p may have a potential utility as an anti-cancer therapy. [score:6]
To the best of our knowledge, reports of miR-542 and miR-1949 expressions in health or damaged cardiac tissue were not yet described. [score:3]
Induction of growth arrest by miR-542-3p that targets survivin. [score:3]
Ablation showed diminished expressions of mir-542-5p and mir-1949 compared to control and occlusion. [score:2]
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[+] score: 10
Specifically, upregulation of miR-466b (1.99-fold change, p = 0.017), miR-542-5p (3.09-fold change, p = 0.020), miR-568 (2.14-fold change, p = 0.020), miR-3580-3p (1.99-fold change, p = 0.017), miR-3581 (1.79-fold change, p = 0.019), and miR-3597-5p (1.9-fold change, p = 0.037), and downregulation of miR-345-5p (−1.56-fold change, p = 0.006) and miR-743b (−2.74-fold change, p = 0.031) were detected for aGvHD samples compared to controls (Figures 5A,B). [score:6]
The upregulation of miR-542-5p or miR-466b was not confirmed in the validation samples (Figure 5C and data not shown). [score:4]
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[+] score: 10
Similarly, we found that AA treatment increased expression of miR-450a and miR-542, and that AA induced a DNA damage response indicated by up-regulation of miR-34a. [score:6]
A recent study demonstrates that AA treatment altered a number of miRNAs in proximal tubular epithelial cells such as up-regulation of miR-192, miR-194, miR-450a, and miR-542. [score:4]
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[+] score: 9
587E-02rno-miR-3801.8745395down1.176E-02rno-miR-1901.8834999down1.528E-02rno-miR-542-3p1.8835889down7.955E-03rno-miR-331.9224428down3.279E-03rno-miR-1441.9507241down4.969E-02rno-miR-542-5p2.2845843down5.943E-03rno-miR-5433.0247948down1.067E-03 rno-miR-296* 3.0831256 down 2.85E-06 Notes: The Agilent rat miRNA microarray identified 24 significantly deregulated miRNAs (fold change >1.5, expression either upregulated or down-regulated; * p <0.05) in the hippocampus of the TLE rat when compared with the control (n = 6 for each group). [score:9]
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[+] score: 5
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-16-1, hsa-mir-17, hsa-mir-21, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-25, hsa-mir-26a-1, hsa-mir-26b, hsa-mir-30a, hsa-mir-31, hsa-mir-96, hsa-mir-99a, hsa-mir-16-2, hsa-mir-30c-2, hsa-mir-30d, hsa-mir-182, hsa-mir-183, hsa-mir-211, hsa-mir-217, hsa-mir-218-1, hsa-mir-218-2, hsa-mir-221, hsa-mir-222, hsa-let-7g, hsa-let-7i, hsa-mir-15b, hsa-mir-23b, hsa-mir-30b, hsa-mir-125b-1, hsa-mir-132, hsa-mir-143, hsa-mir-145, hsa-mir-191, hsa-mir-125a, hsa-mir-125b-2, hsa-mir-126, hsa-mir-184, hsa-mir-190a, hsa-mir-195, rno-mir-322-1, rno-let-7d, rno-mir-335, rno-mir-342, rno-mir-135b, hsa-mir-30c-1, hsa-mir-299, hsa-mir-30e, hsa-mir-26a-2, hsa-mir-379, hsa-mir-382, hsa-mir-342, hsa-mir-135b, hsa-mir-335, rno-let-7a-1, rno-let-7a-2, rno-let-7b, rno-let-7c-1, rno-let-7c-2, rno-let-7e, rno-let-7f-1, rno-let-7f-2, rno-let-7i, rno-mir-15b, rno-mir-16, rno-mir-17-1, rno-mir-21, rno-mir-23a, rno-mir-23b, rno-mir-24-1, rno-mir-24-2, rno-mir-25, rno-mir-26a, rno-mir-26b, rno-mir-30c-1, rno-mir-30e, rno-mir-30b, rno-mir-30d, rno-mir-30a, rno-mir-30c-2, rno-mir-31a, rno-mir-96, rno-mir-99a, rno-mir-125a, rno-mir-125b-1, rno-mir-125b-2, rno-mir-126a, rno-mir-132, rno-mir-143, rno-mir-145, rno-mir-183, rno-mir-184, rno-mir-190a-1, rno-mir-191a, rno-mir-195, rno-mir-211, rno-mir-217, rno-mir-218a-2, rno-mir-218a-1, rno-mir-221, rno-mir-222, rno-mir-299a, hsa-mir-384, hsa-mir-20b, hsa-mir-409, hsa-mir-412, hsa-mir-489, hsa-mir-494, rno-mir-489, rno-mir-412, rno-mir-543, rno-mir-542-1, rno-mir-379, rno-mir-494, rno-mir-382, rno-mir-409a, rno-mir-20b, hsa-mir-542, hsa-mir-770, hsa-mir-190b, hsa-mir-543, rno-mir-466c, rno-mir-17-2, rno-mir-182, rno-mir-190b, rno-mir-384, rno-mir-673, rno-mir-674, rno-mir-770, rno-mir-31b, rno-mir-191b, rno-mir-299b, rno-mir-218b, rno-mir-126b, rno-mir-409b, rno-let-7g, rno-mir-190a-2, rno-mir-322-2, rno-mir-542-2
Differential expression of miR-23a, miR-23b,miR-542–3p, miR-211, and miR-17–5p in granulosa/cumulus cells from women undergoing assisted reproduction suggests aberrant miRNA expression may be an underlying etiology in female infertility [16, 17]. [score:5]
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[+] score: 3
Other miRNAs from this paper: mmu-mir-30a, mmu-mir-101a, mmu-mir-125a, mmu-mir-125b-2, mmu-mir-132, mmu-mir-134, mmu-mir-135a-1, mmu-mir-138-2, mmu-mir-142a, mmu-mir-150, mmu-mir-154, mmu-mir-182, mmu-mir-183, mmu-mir-24-1, mmu-mir-194-1, mmu-mir-200b, mmu-mir-122, mmu-mir-296, mmu-mir-21a, mmu-mir-27a, mmu-mir-92a-2, mmu-mir-96, rno-mir-322-1, mmu-mir-322, rno-mir-330, mmu-mir-330, rno-mir-339, mmu-mir-339, rno-mir-342, mmu-mir-342, rno-mir-135b, mmu-mir-135b, mmu-mir-19a, mmu-mir-100, mmu-mir-139, mmu-mir-212, mmu-mir-181a-1, mmu-mir-214, mmu-mir-224, mmu-mir-135a-2, mmu-mir-92a-1, mmu-mir-138-1, mmu-mir-181b-1, mmu-mir-125b-1, mmu-mir-194-2, mmu-mir-377, mmu-mir-383, mmu-mir-181b-2, rno-mir-19a, rno-mir-21, rno-mir-24-1, rno-mir-27a, rno-mir-30a, rno-mir-92a-1, rno-mir-92a-2, rno-mir-96, rno-mir-100, rno-mir-101a, rno-mir-122, rno-mir-125a, rno-mir-125b-1, rno-mir-125b-2, rno-mir-132, rno-mir-134, rno-mir-135a, rno-mir-138-2, rno-mir-138-1, rno-mir-139, rno-mir-142, rno-mir-150, rno-mir-154, rno-mir-181b-1, rno-mir-181b-2, rno-mir-183, rno-mir-194-1, rno-mir-194-2, rno-mir-200b, rno-mir-212, rno-mir-181a-1, rno-mir-214, rno-mir-296, mmu-mir-376b, mmu-mir-370, mmu-mir-433, rno-mir-433, mmu-mir-466a, rno-mir-383, rno-mir-224, mmu-mir-483, rno-mir-483, rno-mir-370, rno-mir-377, mmu-mir-542, rno-mir-542-1, mmu-mir-494, mmu-mir-20b, mmu-mir-503, rno-mir-494, rno-mir-376b, rno-mir-20b, rno-mir-503-1, mmu-mir-1224, mmu-mir-551b, mmu-mir-672, mmu-mir-455, mmu-mir-490, mmu-mir-466b-1, mmu-mir-466b-2, mmu-mir-466b-3, mmu-mir-466c-1, mmu-mir-466e, mmu-mir-466f-1, mmu-mir-466f-2, mmu-mir-466f-3, mmu-mir-466g, mmu-mir-466h, mmu-mir-504, mmu-mir-466d, mmu-mir-872, mmu-mir-877, rno-mir-466b-1, rno-mir-466b-2, rno-mir-466c, rno-mir-872, rno-mir-877, rno-mir-182, rno-mir-455, rno-mir-672, mmu-mir-466l, mmu-mir-466i, mmu-mir-466f-4, mmu-mir-466k, mmu-mir-466j, rno-mir-551b, rno-mir-490, rno-mir-1224, rno-mir-504, mmu-mir-466m, mmu-mir-466o, mmu-mir-466c-2, mmu-mir-466b-4, mmu-mir-466b-5, mmu-mir-466b-6, mmu-mir-466b-7, mmu-mir-466p, mmu-mir-466n, mmu-mir-466b-8, rno-mir-466d, mmu-mir-466q, mmu-mir-21b, mmu-mir-21c, mmu-mir-142b, mmu-mir-466c-3, rno-mir-322-2, rno-mir-503-2, rno-mir-466b-3, rno-mir-466b-4, rno-mir-542-2
ABCG1 may be a target gene of miR-542. [score:3]
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[+] score: 2
Lulla et al. identified that mir-135b, mir-150, mir-542-5p, and mir-652 were differentially expressed in osteosarcoma compared with normal osteoblasts [15]. [score:2]
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[+] score: 2
9 -45.6 mmu-miR-27b -1.8 -71.4 -462.7 mmu-miR-214* -2.6 -5.0 -43.5 mmu-let-7c-1* -73.2 -204.4 -334.1 mmu-miR-34c -9.4 -26.1 -42.7 mmu-miR-542–3p -5.9 -195.6 -319.8 mmu-miR-706 -9.3 -5.0 -38.7 mmu-miR-487b -2.0 -161.5 -263.9 mmu-miR-467b* -10.1 -2.2 -33.6 rno-miR-17–3p -1.6 -152.0 -248.5 mmu-miR-323–3p -3.7 -23.3 -29.8 mmu-miR-10b -2. 4 -136.6 -223.3 mmu-miR-202–3p -6.5 -5.9 -21.4 mmu-miR-29b -3.0 -135.1 -220.9 mmu-miR-339–5p -1.6 -9.6 -19.6 mmu-miR-297a* -2.4 -128.4 -209.8 mmu-miR-181c -2.0 -10.5 -14.6 mmu-miR-692 -41.5 -115.8 -189.2 mmu-miR-203 -4.6 -6.4 -13.8 mmu-miR-208 -40.6 -113.5 -185.5 mmu-miR-467a* -2.6 -3.9 -11.4 mmu-miR-467c -38.9 -108.6 -177. [score:1]
8 -55.3 -90.4 mmu-miR-34c* -3.7 -1.7 -6.5 mmu-miR-709 -2.9 -2.4 -4.3 mmu-miR-326 -3.9 -2.9 -5.6 rno-miR-664 -1.8 -2.2 -3.6 mmu-miR-350 -1.4 -110.5 -5.1 mmu-let-7e 1.4 -7.6 -5.1 mmu-miR-542–5p -1.9 -5.2 -5.1 rno-miR-20b-5p -3.0 -4.5 -5.0 mmu-miR-374 -1.7 -3.7 -4.4 To understand the cardiovascular benefit of resveratrol in ischaemia/repurfusion, we included longevinex, a commercial formulation of resveratrol by gavage to rat. [score:1]
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