![]() |
miRBase |
![]() |
![]() |
![]() 28 publications mentioning ssc-mir-29b-2Open access articles that are associated with the species Sus scrofa and mention the gene name mir-29b-2. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary. |
|
1 |
![]()
Other miRNAs from this paper: ssc-mir-29b-1, ssc-mir-7-2, ssc-mir-18a, ssc-mir-21, ssc-mir-29c, ssc-mir-15a, ssc-mir-221, ssc-mir-29a, ssc-mir-193a, ssc-mir-92b, ssc-mir-34c-1, ssc-mir-339-1, ssc-mir-451, ssc-mir-7-1, ssc-mir-34c-2, ssc-mir-339-2, ssc-mir-671, ssc-mir-7134
This concurs with the expression of the apoptosis-related genes, all down-regulated at day 3 compared to their initial up-regulation on day 1. The same pattern holds true for the expression IFIH1 and TLR3 and the miRNAs that target them (ssc-miR-29b, ssc-miR-18a, and hsa-miR-590-3p).
[score:12]
Common to all five miRNAs found to correlate negatively with apoptosis-related gene expression (ssc-miR-15a, ssc-miR-18a, ssc-miR-21, ssc-miR-29b, and hsa-miR-590-3p) is that they were significantly up-regulated on day 3 after challenge, and not regulated at other time points.
[score:7]
Finally, we also found ssc-miR-29b to be modestly up-regulated on day 1 and 3 after challenge, suggesting that ssc-miR-29b mediated up-regulation of IL28B via COX2 and IRF7 may also contribute to the antiviral response in IAV infection in our pig mo del.
[score:7]
They furthermore demonstrated that hsa-miR-29 (especially hsa-miR-29b-3p) indirectly mediate IFNL1 up-regulation by controlling DNA methyltransferase (DNMT3A and DNMT3B) activity in human pulmonary epithelial cells (A549) and a subsequent IRF3/IRF7 dependent transcription of IFNL1 via COX2 (PTGS2), a potent inflammatory protein in IAV infection [69].
[score:5]
This negative correlation between miRNA expression and their target transcripts thus suggest the specific involvement of ssc-miR-15a, ssc-miR-18a, ssc-miR-21, ssc-miR-29b, and hsa-miR-590-3p in the modulation of important signaling cascades of apoptosis and viral recognition.
[score:5]
In addition to the abovementioned possible involvement of ssc-miR-29b in the IFN-λ3 response, our results likewise suggested miRNA -mediated modulation of other branches of the innate response to IAV infection in the pig.
[score:1]
[1 to 20 of 6 sentences]
|
2 |
![]()
Other miRNAs from this paper: hsa-let-7c, hsa-mir-29a, hsa-mir-31, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-192, hsa-mir-148a, hsa-mir-210, hsa-let-7g, hsa-mir-30b, hsa-mir-122, hsa-mir-150, hsa-mir-155, hsa-mir-29c, hsa-mir-361, hsa-mir-362, hsa-mir-342, hsa-mir-323a, ssc-mir-122, ssc-mir-148a, ssc-mir-29b-1, ssc-mir-323, ssc-let-7c, ssc-mir-29c, hsa-mir-491, hsa-mir-146b, hsa-mir-654, hsa-mir-769, ssc-mir-30b, ssc-mir-210, ssc-mir-146b, ssc-let-7g, ssc-mir-29a, ssc-mir-192, ssc-mir-361, ssc-mir-342, ssc-mir-769, hsa-mir-323b, ssc-mir-155, ssc-mir-362, ssc-mir-491, ssc-mir-31, ssc-mir-150-1, ssc-mir-150-2
In our study, CASP3 was significantly upregulated by qRT-PCR analysis (Figure 6) and targeted by the downregulated miRNAs: miR-342-3p, miR-29b, miR-29c, miR-29a, let-7g and miR-30b, which can be expected to develop miRNA -based therapeutics for influenza disease.
[score:11]
Therefore, the downregulated miR-29 may regulate the T helper 1 (Th1) cell differentiation to secrete more IFN-gamma and mediate elimination of intracellular pathogens, but dysregulated T cell responses may also contribute to pathologic inflammation.
[score:6]
MiR-29a and miR-29b were reported to be downregulated in lung tissues from mice infected with reconstructed 1918 or a nonlethal seasonal influenza virus, Tx/91 [17].
[score:4]
Both miR-29a and miR-29b could repress IFN-gamma production by direct targeting of both T-box transcription factor T-bet and Eomesodermin (Eomes), two transcription factors known to induce IFN-gamma production [63].
[score:4]
The expression of hsa-miR-150, hsa-miR-31, hsa-miR-155, hsa-miR-29a, hsa-miR-29b, hsa-miR-342-5p, and hsa-miR-146b-5p were present in lower abundance, whereas hsa-miR-148a and hsa-miR-886-3p were present in higher abundance in PBMCs from critically ill patients infected with H1N1 influenza virus than that from healthy controls.
[score:3]
validation of differentially expressed miRNAs and ROC analysisThe microarray data were validated by performing, qRT-PCR for nine miRNAs, including hsa-miR-146b-5p, hsa-miR-148a, hsa-miR-150, hsa-miR-31, hsa-miR-155, hsa-miR-29a, hsa-miR-29b, hsa-miR-342-5p, and hsa-miR-886-3p.
[score:3]
The microarray data were validated by performing, qRT-PCR for nine miRNAs, including hsa-miR-146b-5p, hsa-miR-148a, hsa-miR-150, hsa-miR-31, hsa-miR-155, hsa-miR-29a, hsa-miR-29b, hsa-miR-342-5p, and hsa-miR-886-3p.
[score:1]
[1 to 20 of 7 sentences]
|
3 |
![]()
Other miRNAs from this paper: ssc-mir-184, ssc-mir-29b-1, ssc-mir-7-2, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-21, ssc-mir-29c, ssc-mir-1, ssc-mir-215, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-29a, ssc-mir-30e, ssc-mir-27b, ssc-mir-192, ssc-mir-339-1, ssc-mir-708, ssc-mir-194b, ssc-let-7a-2, ssc-let-7d, ssc-let-7f-2, ssc-mir-7-1, ssc-mir-194a, ssc-mir-339-2, ssc-mir-370
The down-regulated miR-29b and miR-29a are both or each connected to up-regulated SMARCE1, HBO1, NKX6-1, HOXA10, HBP1, OTUD4, that could be further considered as potent targets during infection.
[score:8]
Five miRNAs were nodal and were either up-regulated (miR-194 and miR-let7b) or down-regulated (miR-27B, miR29-a and miR29-b1).
[score:7]
In particular, human EIF2C1 mRNA is a predicted target of two human miRNAs (miR-let7b and miR29b) and EIF2C4 mRNA is a predicted target of human miR-let7b [29].
[score:5]
EIF2C1 and EIF2C4 were respectively connected to miR29b and miR-27b, both down-regulated.
[score:4]
[1 to 20 of 4 sentences]
|
4 |
![]()
Other miRNAs from this paper: ssc-mir-122, ssc-mir-125b-2, ssc-mir-181b-2, ssc-mir-20a, ssc-mir-23a, ssc-mir-26a, ssc-mir-29b-1, ssc-mir-181c, ssc-mir-214, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-103-1, ssc-mir-107, ssc-mir-21, ssc-mir-29c, ssc-mir-30c-2, bta-mir-26a-2, bta-mir-29a, bta-let-7f-2, bta-mir-103-1, bta-mir-20a, bta-mir-21, bta-mir-26b, bta-mir-30d, bta-mir-499, bta-mir-99a, bta-mir-125b-1, bta-mir-126, bta-mir-181a-2, bta-mir-199a-1, bta-mir-30b, bta-mir-107, bta-mir-10a, bta-mir-127, bta-mir-142, bta-mir-181b-2, bta-mir-30e, bta-mir-92a-2, bta-let-7d, bta-mir-132, bta-mir-138-2, bta-mir-17, bta-mir-181c, bta-mir-192, bta-mir-199b, bta-mir-200a, bta-mir-200c, bta-mir-214, bta-mir-23a, bta-mir-29b-2, bta-mir-29c, bta-mir-455, bta-let-7g, bta-mir-10b, bta-mir-30a, bta-mir-200b, bta-let-7a-1, bta-let-7f-1, bta-mir-122, bta-mir-30c, bta-let-7i, bta-mir-25, bta-let-7a-2, bta-let-7a-3, bta-let-7b, bta-let-7c, bta-let-7e, bta-mir-103-2, bta-mir-125b-2, bta-mir-99b, ssc-mir-99b, ssc-mir-17, ssc-mir-30b, ssc-mir-199b, bta-mir-1-2, bta-mir-1-1, bta-mir-129-1, bta-mir-129-2, bta-mir-133a-2, bta-mir-133a-1, bta-mir-133b, bta-mir-135b, bta-mir-138-1, bta-mir-143, bta-mir-144, bta-mir-146b, bta-mir-146a, bta-mir-181d, bta-mir-190a, bta-mir-199a-2, bta-mir-202, bta-mir-206, bta-mir-211, bta-mir-212, bta-mir-223, bta-mir-26a-1, bta-mir-29d, bta-mir-30f, bta-mir-338, bta-mir-33a, bta-mir-33b, bta-mir-375, bta-mir-429, bta-mir-451, bta-mir-92a-1, bta-mir-92b, bta-mir-29e, bta-mir-29b-1, bta-mir-181a-1, bta-mir-181b-1, ssc-mir-133a-1, ssc-mir-1, ssc-mir-146b, ssc-mir-181a-1, ssc-mir-30a, bta-mir-199c, ssc-mir-206, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-133b, ssc-mir-29a, ssc-mir-30d, ssc-mir-30e, ssc-mir-199a-2, ssc-mir-499, ssc-mir-143, ssc-mir-10a, ssc-mir-10b, ssc-mir-103-2, ssc-mir-181a-2, ssc-mir-181b-1, ssc-mir-181d, ssc-mir-99a, ssc-mir-92a-2, ssc-mir-92a-1, ssc-mir-92b, ssc-mir-192, ssc-mir-142, ssc-mir-127, ssc-mir-202, ssc-mir-129a, ssc-mir-455, ssc-mir-125b-1, ssc-mir-338, ssc-mir-133a-2, ssc-mir-146a, bta-mir-26c, ssc-mir-30c-1, ssc-mir-126, ssc-mir-199a-1, ssc-mir-451, ssc-let-7a-2, ssc-mir-129b, ssc-mir-429, ssc-let-7d, ssc-let-7f-2, ssc-mir-132, ssc-mir-138, ssc-mir-144, ssc-mir-190a, ssc-mir-212, bta-mir-133c, ssc-mir-26b, ssc-mir-200b, ssc-mir-223, ssc-mir-375, ssc-mir-33b
In humans, the high expression of miR-129 in ovaries is associated with the control of cell growth and differentiation in the final process of ovary maturation through downregulation of its target mRNAs, whereas miR-29 expression levels, which progressively increase throughout oogenesis, may also be important (Sirotkin et al., 2009).
[score:10]
microRNA-29, a key regulator of collagen expression in systemic sclerosis.
[score:4]
By contrast, the miR-29 and miR-129 families showed significantly increased expression in ovaries compared to testes (Xiao et al., 2014).
[score:2]
Biological functions of miR-29b contribute to positive regulation of osteoblast differentiation.
[score:2]
[1 to 20 of 4 sentences]
|
5 |
![]()
Other miRNAs from this paper: ssc-mir-122, ssc-mir-145, ssc-mir-29b-1, ssc-let-7c, ssc-mir-196a-2, ssc-mir-29c, ssc-let-7a-1, ssc-let-7e, ssc-mir-99a, ssc-mir-148b, ssc-mir-92b, ssc-mir-208b, ssc-mir-374a, ssc-mir-339-1, ssc-mir-362, ssc-mir-4332, ssc-mir-451, ssc-let-7a-2, ssc-mir-182, ssc-mir-196a-1, ssc-mir-339-2
CAV2 expression was upregulated in DSP-TP; this gene was the target of miR-29b and miR-122, which hadapproximately seven- and fivefold lower expression in the DSP-TP than in the LL-YY, respectively.
[score:10]
The expressions of five genes (IRS1, CARNS1, MYOZ2, ANKRD2, and PLIN2) and four miRNAs (miR-4332, miR-451, miR-196a, and miR-29b) were significantly different between the DSP and the YY (S5 and S6 Figs).
[score:3]
Some DE miRNAs, such as miR-29b, miR-122, miR-145-5p, let-7c, miR-4332, miR-182, miR-92b-3p, miR-29c,let-7a, and let-7e, and some DEGs such as CAV2, MYOZ2, FRZB, FASN, SCD, ESR1, and ADORA1, may be factors in the regulation of muscle growth and lipid deposition.
[score:2]
Therefore, we determined that the genes CAV2, MYOZ2, and FRZB and the miRNAs miR-29b, miR-122, miR-145-5p, and miR-let-7c are the key candidates for regulating muscle growth.
[score:2]
Based on the fold change, the top five were miR-4332 (FC = 0.024), miR-29b (FC = 0.140), miR-92b-3p (FC = 0.183), miR-362 (FC = 5.406), and miR-122 (FC = 0.193).
[score:1]
[1 to 20 of 5 sentences]
|
6 |
![]()
Other miRNAs from this paper: hsa-mir-17, hsa-mir-18a, hsa-mir-19b-2, hsa-mir-22, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-29a, hsa-mir-92a-2, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-106a, hsa-mir-7-1, hsa-mir-7-2, hsa-mir-7-3, hsa-mir-216a, hsa-mir-217, hsa-mir-221, hsa-mir-222, hsa-let-7i, hsa-mir-27b, hsa-mir-135a-1, hsa-mir-135a-2, hsa-mir-142, hsa-mir-191, hsa-mir-29c, hsa-mir-135b, hsa-mir-18b, ssc-mir-106a, ssc-mir-216-1, ssc-mir-217-1, ssc-mir-24-1, ssc-mir-29b-1, ssc-mir-7-2, ssc-let-7i, ssc-mir-103-1, ssc-mir-18a, ssc-mir-29c, hsa-mir-455, hsa-mir-216b, hsa-mir-103b-1, hsa-mir-103b-2, ssc-mir-17, ssc-mir-221, ssc-mir-29a, ssc-mir-191, ssc-mir-103-2, ssc-mir-27b, ssc-mir-24-2, ssc-mir-92a-2, ssc-mir-142, ssc-mir-222, ssc-mir-19b-2, ssc-mir-455, ssc-mir-22, ssc-mir-18b, ssc-mir-7-1, ssc-mir-216-2, ssc-mir-217-2
In addition, the miR-29 family is associated with the up-regulation of the tumor suppressor p53, which is central to many cellular stress responses and for inducing apoptosis [31].
[score:6]
The second group, e. g. ; the miR-29 family (containing miR-29a –b –c) and miR-22, miR-24, miR-27b and miR-142-5p showed increasing expression with age progression, with a particularly high expression in the adult tissues.
[score:5]
Therefore, it is plausible that miRNAs prominently expressed at this time (for instance miR-24 and the miR-29 family members) might regulate this process.
[score:4]
The miR-29 family (miR-29a, miR-29b and miR-29c) has previously been shown to be effective biomarkers of radiation -induced brain responses [29].
[score:1]
[1 to 20 of 4 sentences]
|
7 |
![]()
Other miRNAs from this paper: ssc-mir-148a, ssc-mir-29b-1, ssc-mir-214, ssc-mir-124a-2, ssc-mir-186, ssc-mir-29c, ssc-mir-124a-1, ssc-mir-133a-1, ssc-mir-1, ssc-mir-206, ssc-mir-133b, ssc-mir-29a, ssc-mir-499, ssc-mir-345-1, ssc-mir-148b, ssc-mir-208b, ssc-mir-424, ssc-mir-133a-2, ssc-mir-487b, ssc-mir-451, ssc-mir-345-2
There were 17 DEMs, represented by miR-124 and miR-214, dominating the miRNA-regulated gene expression in the early metaphase of embryonic stage and 10 DEMs, miR-29 for instance, were highly expressed at the postnatal stage.
[score:6]
Maurer B. Stanczyk J. Jungel A. Akhmetshina A. Trenkmann M. Brock M. Kowal-Bielecka O. Gay R. E. Michel B. A. Distler J. H. MicroRNA-29, a key regulator of collagen expression in systemic sclerosis Arthritis Rheumatol.
[score:3]
Our results were consistent with previous studies, which demonstrated miR-29 targeting to the collagen family members such as COL4A1, COL1A2 and COL1A1 at ADU time points, miR-148 to the MITF and EIF4BP2 genes at early embryonic stage, and miR-487 to the IRS1 gene at the middle stage [23, 24].
[score:3]
Three category patterns from the DEMs were clustered based on the expression percentages including early-middle embryonic stage from miR-124 to miR-424, late embryonic stage from miR-345 to miR-451 and adult stage from miR-29b to miR-133a, respectively.
[score:3]
[1 to 20 of 4 sentences]
|
8 |
![]()
Other miRNAs from this paper: ssc-mir-106a, ssc-mir-15b, ssc-mir-24-1, ssc-mir-29b-1, ssc-mir-27a, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-18a, ssc-mir-29c, ssc-mir-15a, ssc-mir-16-2, ssc-mir-16-1, ssc-mir-34a, ssc-mir-133a-1, ssc-mir-1, ssc-mir-206, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-133b, ssc-mir-29a, ssc-mir-423, ssc-mir-151, ssc-mir-27b, ssc-mir-24-2, ssc-mir-34c-1, ssc-mir-424, ssc-mir-432, ssc-mir-338, ssc-mir-376a, ssc-mir-133a-2, ssc-mir-381, ssc-mir-376c, ssc-mir-376b, ssc-mir-126, ssc-let-7a-2, ssc-mir-18b, ssc-let-7d, ssc-let-7f-2, ssc-mir-34c-2
Secondly, targets for a sub-set of down-regulated miR (miR-15, miR-16, miR-27, miR-29, miR-34 and miR-106), of which 44 targets were identified based on our previous criteria were predicted (see Additional file 4).
[score:8]
These miR have been implicated in multiple cellular processes including cell growth (miR-24), apoptosis (miR-16, miR-24 and miR-29), and cell cycle regulation in normal (miR-16) and cancerous cells (miR-24, miR-27, miR-29 and miR-34) [9, 40- 46].
[score:2]
[1 to 20 of 2 sentences]
|
9 |
![]()
Other miRNAs from this paper: ssc-mir-106a, ssc-mir-125b-2, ssc-mir-23a, ssc-mir-26a, ssc-let-7i, ssc-mir-21, ssc-mir-9-1, ssc-mir-9-2, ssc-mir-99b, ssc-mir-17, ssc-mir-133a-1, ssc-mir-30a, ssc-mir-206, ssc-mir-378-1, ssc-mir-133b, ssc-mir-30d, ssc-mir-10a, ssc-mir-99a, ssc-mir-345-1, ssc-mir-92a-2, ssc-mir-92a-1, ssc-mir-92b, ssc-mir-100, ssc-mir-450b, ssc-mir-1306, ssc-mir-339-1, ssc-mir-19b-2, ssc-mir-19b-1, ssc-mir-129a, ssc-mir-505, ssc-mir-125b-1, ssc-mir-450c, ssc-mir-133a-2, ssc-mir-378-2, ssc-mir-218b, ssc-let-7d, ssc-mir-9-3, ssc-mir-137, ssc-mir-339-2, ssc-mir-345-2, ssc-mir-1249-1, ssc-mir-1249-2, ssc-mir-378b, ssc-mir-26b
Only 27 miRNAs followed the same expression profile in both approaches, and 11 of them where DE in both groups: 4 miRNAs (miR-26b-5p, miR-29b-2-5p, miR-450b-5p and miR-450c-5p) were only expressed in infected groups and the remaining 7 miRNAs (let-7b-3p, miR-193b, miR-345-5p, miR-1306-5p, miR-2779, miR-2898 and miR-4286) were more expressed in mock-infected or healthy group, although all of them were expressed at low levels (CN<100) in both profiles.
[score:9]
[1 to 20 of 1 sentences]
|
10 |
![]()
Other miRNAs from this paper: ssc-mir-122, ssc-mir-148a, ssc-mir-29b-1, ssc-mir-32, ssc-mir-21, ssc-mir-29c, ssc-mir-15a, ssc-mir-29a, ssc-mir-191, ssc-mir-143, ssc-mir-152, ssc-mir-664, ssc-mir-192, ssc-mir-142, ssc-mir-155, ssc-mir-146a, ssc-mir-126, ssc-mir-451, ssc-mir-2320, ssc-mir-144, ssc-mir-223
MicroRNAs like miR-29a and miR-32 have been found to repress the expression of viral mRNAs by possible recognition and targeting of viral nucleic acids with miR-29 specifically targeting HIV-1 3’UTR region [11], [12].
[score:7]
[1 to 20 of 1 sentences]
|
11 |
![]()
Other miRNAs from this paper: hsa-mir-29b-1, hsa-mir-29b-2, mmu-mir-29b-1, mmu-mir-29b-2, ssc-mir-29b-1
BAG3 also downregulates the microRNA-29b which leads to upregulation of the anti-apoptosis protein Mcl-l leading to resistance to anticancer drugs [110].
[score:7]
[1 to 20 of 1 sentences]
|
12 |
![]()
Other miRNAs from this paper: ssc-mir-125b-2, ssc-mir-145, ssc-mir-181b-2, ssc-mir-24-1, ssc-mir-29b-1, ssc-mir-181c, ssc-mir-124a-2, ssc-mir-136, ssc-mir-21, ssc-mir-9-2, ssc-mir-124a-1, ssc-mir-181a-1, ssc-mir-30a, ssc-mir-29a, ssc-mir-30e, ssc-mir-181a-2, ssc-mir-181b-1, ssc-mir-181d, ssc-mir-27b, ssc-mir-24-2, ssc-mir-125a, ssc-mir-98, ssc-mir-500, ssc-mir-125b-1, ssc-mir-146a, ssc-mir-132
Of the differentially expressed miRNAs six (miR-30a-3p, miR-132, miR-27b*, miR-29b, miR-146a and miR-9-2) were altered at more than one time point in PRRSV-infected macrophages (Figure 1).
[score:3]
A total of forty cellular miRNAs were differentially expressed in PRRSV infected macrophages, six of which (miR-30a-3p, miR-132, miR-27b*, miR-29b, miR-146a and miR-9-2) were altered at more than one time point (Figure 1).
[score:3]
Among these six miRNA, miR-30a-3p, miR-132, miR-27b*, miR-29b, miR-146a and miR-9-2, were altered at more than one time point.
[score:1]
[1 to 20 of 3 sentences]
|
13 |
![]()
Other miRNAs from this paper: ssc-mir-23a, ssc-mir-28, ssc-mir-29b-1, ssc-mir-186, ssc-mir-21, ssc-mir-29c, ssc-mir-30c-2, ssc-mir-15a, ssc-mir-16-2, ssc-mir-16-1, ssc-mir-29a, ssc-mir-423, ssc-mir-23b, ssc-mir-92b, ssc-mir-22, ssc-mir-146a, ssc-mir-30c-1, ssc-mir-31, ssc-mir-150-1, ssc-mir-150-2, ssc-mir-182, ssc-mir-223
Only four miRNAs (hsa-miR-223-5p, ssc-miR-31, ssc-miR-29a, and ssc-miR-182) were differentially expressed at 24 h pi, whereas 10 miRNAs were differentially expressed at 72 h pi (ssc-miR-29b, hsa-miR-203a-3p, hsa-miR-449a, ssc-miR-21, ssc-miR-29a, hsa-miR-23a-3p, ssc-miR-23b, ssc-miR-30c-5p, ssc-miR-423-5p, and hsa-miR-150-5p).
[score:5]
The miR-29 family is well characterised and commonly described to be involved in cancers as tumor suppressors 49.
[score:1]
However, the highest number of significantly regulated miRNAs compared to before challenge was found at 14d pi, at which time point the infection had completely cleared (ssc-miR-15a, ssc-miR-29b, ssc-miR-29a, hsa-miR-449a, ssc-miR-186, ssc-miR-22-5p, ssc-miR-28-5p, hsa-miR-203a-3p, ssc-miR-146a-5p, hsa-miR-150-5p, ssc-miR-23b, hsa-miR-223-3p, hsa-miR-23a-3p, hsa-miR-16-5p).
[score:1]
[1 to 20 of 3 sentences]
|
14 |
![]()
Other miRNAs from this paper: mmu-mir-1a-1, mmu-mir-29b-1, mmu-mir-181a-2, mmu-mir-206, mmu-mir-148a, mmu-mir-29a, mmu-mir-29c, mmu-mir-322, mmu-mir-1a-2, mmu-mir-181a-1, mmu-mir-214, mmu-mir-29b-2, mmu-mir-181b-1, mmu-mir-181c, mmu-mir-181b-2, ssc-mir-148a, ssc-mir-181b-2, ssc-mir-29b-1, ssc-mir-95, ssc-mir-181c, ssc-mir-214, ssc-mir-29c, mmu-mir-486a, mmu-mir-503, bta-mir-29a, bta-mir-148a, bta-mir-181a-2, bta-mir-181b-2, bta-mir-181c, bta-mir-214, bta-mir-29b-2, bta-mir-29c, mmu-mir-181d, mmu-mir-1b, bta-mir-1-2, bta-mir-1-1, bta-mir-181d, bta-mir-206, bta-mir-29d, bta-mir-486, bta-mir-95, bta-mir-29e, bta-mir-29b-1, bta-mir-181a-1, bta-mir-181b-1, ssc-mir-1, ssc-mir-503, ssc-mir-181a-1, ssc-mir-206, ssc-mir-29a, ssc-mir-486-1, ssc-mir-181a-2, ssc-mir-181b-1, ssc-mir-181d, mmu-mir-486b, ssc-mir-486-2, bta-mir-503
For example, miR-29, miR-181 and miR-148a can promote myoblast differentiation by inhibiting the expression of downstream target genes Akt3, Hox-A1 and ROCK1 at protein levels [10– 12].
[score:7]
[1 to 20 of 1 sentences]
|
15 |
![]()
Other miRNAs from this paper: hsa-mir-16-1, hsa-mir-29a, hsa-mir-93, hsa-mir-101-1, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-16-2, mmu-mir-29b-1, mmu-mir-101a, mmu-mir-125a, mmu-mir-125b-2, mmu-mir-144, mmu-mir-155, mmu-mir-143, hsa-mir-34a, hsa-mir-214, hsa-mir-217, hsa-mir-221, mmu-mir-34c, mmu-mir-34b, hsa-mir-125b-1, hsa-mir-143, hsa-mir-144, hsa-mir-125a, hsa-mir-125b-2, mmu-mir-16-1, mmu-mir-16-2, mmu-mir-29a, mmu-mir-29c, mmu-mir-93, mmu-mir-34a, mmu-mir-101b, hsa-mir-155, mmu-mir-214, mmu-mir-221, mmu-mir-29b-2, mmu-mir-125b-1, mmu-mir-217, hsa-mir-29c, hsa-mir-101-2, hsa-mir-34b, hsa-mir-34c, ssc-mir-125b-2, ssc-mir-217-1, ssc-mir-29b-1, ssc-mir-214, ssc-mir-29c, bta-mir-29a, bta-mir-101-2, bta-mir-16b, bta-mir-221, bta-mir-125a, bta-mir-125b-1, bta-mir-34b, bta-mir-214, bta-mir-29b-2, bta-mir-29c, bta-mir-93, bta-mir-34c, bta-mir-125b-2, bta-mir-34a, ssc-mir-16-2, ssc-mir-16-1, ssc-mir-34a, ssc-mir-221, bta-mir-101-1, bta-mir-143, bta-mir-144, bta-mir-155, bta-mir-16a, bta-mir-217, bta-mir-29d, bta-mir-29e, bta-mir-29b-1, ssc-mir-101-1, ssc-mir-101-2, ssc-mir-29a, ssc-mir-143, ssc-mir-125a, ssc-mir-34c-1, ssc-mir-125b-1, ssc-mir-155, mmu-mir-101c, ssc-mir-34c-2, ssc-mir-144, ssc-mir-217-2
In addition to tissue-specific ageing, it is increasingly evident that many miRNA regulate gene expressions in well-known ageing pathways, most notably in the p53 tumor suppressor pathway (miR-34, miR-29 and miR-217, etc. )
[score:6]
[1 to 20 of 1 sentences]
|
16 |
![]()
Other miRNAs from this paper: mmu-mir-15b, mmu-mir-23b, mmu-mir-27b, mmu-mir-29b-1, mmu-mir-101a, mmu-mir-138-2, mmu-mir-181a-2, mmu-mir-148a, mmu-mir-192, mmu-mir-15a, mmu-mir-18a, mmu-mir-20a, mmu-mir-23a, mmu-mir-29a, mmu-mir-29c, mmu-mir-27a, mmu-mir-92a-2, rno-mir-20a, rno-mir-101b, mmu-mir-101b, mmu-mir-17, mmu-mir-19a, mmu-mir-181a-1, mmu-mir-29b-2, mmu-mir-19b-1, mmu-mir-92a-1, mmu-mir-138-1, mmu-mir-181b-1, mmu-mir-181c, mmu-mir-181b-2, rno-mir-15b, rno-mir-17-1, rno-mir-18a, rno-mir-19b-1, rno-mir-19a, rno-mir-23a, rno-mir-23b, rno-mir-27b, rno-mir-27a, rno-mir-29b-2, rno-mir-29a, rno-mir-29b-1, rno-mir-29c-1, rno-mir-92a-1, rno-mir-92a-2, rno-mir-101a, rno-mir-138-2, rno-mir-138-1, rno-mir-181c, rno-mir-181a-2, rno-mir-181b-1, rno-mir-181b-2, rno-mir-192, rno-mir-181a-1, ssc-mir-148a, ssc-mir-15b, ssc-mir-181b-2, ssc-mir-19a, ssc-mir-20a, ssc-mir-23a, ssc-mir-29b-1, ssc-mir-181c, ssc-mir-27a, ssc-mir-18a, ssc-mir-29c, mmu-mir-181d, rno-mir-17-2, rno-mir-181d, ssc-mir-15a, ssc-mir-17, ssc-mir-101-1, ssc-mir-101-2, ssc-mir-181a-1, ssc-mir-29a, ssc-mir-181a-2, ssc-mir-181b-1, ssc-mir-181d, ssc-mir-27b, ssc-mir-23b, ssc-mir-92a-2, ssc-mir-92a-1, ssc-mir-192, ssc-mir-19b-1, mmu-mir-101c, ssc-mir-138, ssc-mir-194a, rno-mir-148a, rno-mir-15a, rno-mir-29c-2, rno-mir-29b-3
Another gene family that we evidenced prevalently expressed in the WBCs is mir-29 (mir-29a, mir-29b, mir-29c; p = 2.19 E-4).
[score:3]
We found that miRNAs with higher expression in WBCs includes different miRNA families: mir-15, mir-17, mir-181, mir-23, mir-27 and mir-29 families.
[score:3]
[1 to 20 of 2 sentences]
|
17 |
![]()
Other miRNAs from this paper: ssc-mir-145, ssc-mir-29b-1, ssc-mir-29c, ssc-mir-99b, ssc-mir-30b, ssc-mir-130a, ssc-mir-199b, ssc-mir-1, ssc-mir-29a, ssc-mir-30e, ssc-mir-499, ssc-mir-10b, ssc-mir-208b, ssc-mir-335, ssc-mir-338, ssc-mir-144, ssc-mir-141
Also, overexpression of miR-29 promotes myogenic differentiation in C2C12 cells, due to the reduction in TGF-beta, which inhibits differentiation 42.
[score:5]
[1 to 20 of 1 sentences]
|
18 |
![]()
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-17, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-26a-1, hsa-mir-27a, hsa-mir-29a, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-199a-1, hsa-mir-208a, hsa-mir-148a, hsa-mir-10a, hsa-mir-181a-2, hsa-mir-181c, hsa-mir-199a-2, hsa-mir-181a-1, hsa-mir-214, hsa-mir-221, hsa-let-7g, hsa-let-7i, hsa-mir-1-2, hsa-mir-23b, hsa-mir-27b, hsa-mir-125b-1, hsa-mir-128-1, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-143, hsa-mir-125b-2, hsa-mir-126, hsa-mir-127, hsa-mir-206, hsa-mir-1-1, hsa-mir-128-2, hsa-mir-29c, hsa-mir-26a-2, hsa-mir-378a, hsa-mir-148b, hsa-mir-133b, hsa-mir-424, ssc-mir-125b-2, ssc-mir-148a, ssc-mir-23a, ssc-mir-24-1, ssc-mir-26a, ssc-mir-29b-1, ssc-mir-181c, ssc-mir-214, ssc-mir-27a, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-103-1, ssc-mir-128-1, ssc-mir-29c, hsa-mir-486-1, hsa-mir-499a, hsa-mir-503, hsa-mir-411, hsa-mir-378d-2, hsa-mir-208b, hsa-mir-103b-1, hsa-mir-103b-2, ssc-mir-17, ssc-mir-221, ssc-mir-133a-1, ssc-mir-1, ssc-mir-503, ssc-mir-181a-1, ssc-mir-206, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-378-1, ssc-mir-133b, ssc-mir-29a, ssc-mir-199a-2, ssc-mir-128-2, ssc-mir-499, ssc-mir-143, ssc-mir-10a, ssc-mir-486-1, ssc-mir-103-2, ssc-mir-181a-2, ssc-mir-27b, ssc-mir-24-2, ssc-mir-23b, ssc-mir-148b, ssc-mir-208b, ssc-mir-424, ssc-mir-127, ssc-mir-125b-1, hsa-mir-378b, hsa-mir-378c, ssc-mir-411, ssc-mir-133a-2, ssc-mir-126, ssc-mir-199a-1, ssc-mir-378-2, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-378f, hsa-mir-378g, hsa-mir-378h, hsa-mir-378i, hsa-mir-499b, ssc-let-7a-2, ssc-mir-486-2, hsa-mir-378j, ssc-let-7d, ssc-let-7f-2, hsa-mir-486-2, ssc-mir-378b
Exceptions were some previously reported muscle-related miRNAs such as miR-181c/d-5p, miR-29b/c, miR-221/222 and miR-208, all of which expressed at a very low level (average RPM <100).
[score:3]
PLOS ONE 7. 122 van Rooij E, Sutherland LB, Thatcher JE, DiMaio JM, Naseem RH et al. (2008) Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis.
[score:2]
[1 to 20 of 2 sentences]
|
19 |
![]()
Other miRNAs from this paper: ssc-mir-122, ssc-mir-125b-2, ssc-mir-135-1, ssc-mir-135-2, ssc-mir-145, ssc-mir-15b, ssc-mir-20a, ssc-mir-24-1, ssc-mir-26a, ssc-mir-29b-1, ssc-mir-7-2, ssc-mir-183, ssc-mir-27a, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-103-1, ssc-mir-124a-2, ssc-mir-139, ssc-mir-204, ssc-mir-21, ssc-mir-29c, ssc-mir-30c-2, ssc-mir-99b, ssc-mir-15a, ssc-mir-16-2, ssc-mir-16-1, ssc-mir-30b, ssc-mir-210, ssc-mir-124a-1, ssc-mir-1, ssc-mir-30a, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-29a, ssc-mir-30d, ssc-mir-30e, ssc-mir-191, ssc-mir-152, ssc-mir-103-2, ssc-mir-24-2, ssc-mir-222, ssc-mir-125b-1, ssc-mir-362, ssc-mir-1296, ssc-mir-30c-1, ssc-let-7a-2, ssc-let-7d, ssc-let-7f-2, ssc-mir-7-1, ssc-mir-137, ssc-mir-212, ssc-mir-3613, ssc-mir-2366-1, ssc-mir-2366-2, ssc-mir-26b, ssc-mir-141, ssc-mir-375
The up-regulation of miR-141, miR-200a, miR-200c, miR-26a, and miR-29 we detected were also accordant either with mice or with humans.
[score:4]
Bak et al. reported 8 miRNAs (mir-7, mir-7b, mir-375, mir-141, mir-200a, mir-200c, mir-25and mir-152) with more than 3-fold enrichment in the pituitary of adult mice in a profile of the miRNAs in the central nervous system [36]; and Farh et al. and Landgraf et al. reported 6 (mir-7, mir-212, mir-26a, mir-191, mir-375 and mir-29) when comparing the miRNAs in normal and tumor pituitary cells [37], [38].
[score:1]
[1 to 20 of 2 sentences]
|
20 |
![]()
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-16-1, hsa-mir-21, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-26a-1, hsa-mir-29a, hsa-mir-30a, hsa-mir-31, hsa-mir-99a, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-16-2, hsa-mir-192, hsa-mir-148a, hsa-mir-10b, hsa-mir-181a-2, hsa-mir-181a-1, hsa-mir-215, hsa-mir-223, hsa-mir-224, hsa-mir-200b, hsa-mir-15b, hsa-mir-27b, hsa-mir-125b-1, hsa-mir-141, hsa-mir-143, hsa-mir-152, hsa-mir-125b-2, hsa-mir-126, hsa-mir-146a, hsa-mir-184, hsa-mir-200c, hsa-mir-155, hsa-mir-29c, hsa-mir-200a, hsa-mir-99b, hsa-mir-296, hsa-mir-30e, hsa-mir-26a-2, hsa-mir-378a, hsa-mir-342, hsa-mir-148b, hsa-mir-451a, ssc-mir-125b-2, ssc-mir-148a, ssc-mir-15b, ssc-mir-184, ssc-mir-224, ssc-mir-23a, ssc-mir-24-1, ssc-mir-26a, ssc-mir-29b-1, ssc-let-7f-1, ssc-mir-103-1, ssc-mir-21, ssc-mir-29c, hsa-mir-486-1, hsa-mir-499a, hsa-mir-671, hsa-mir-378d-2, bta-mir-26a-2, bta-mir-29a, bta-let-7f-2, bta-mir-103-1, bta-mir-148a, bta-mir-16b, bta-mir-21, bta-mir-499, bta-mir-99a, bta-mir-125b-1, bta-mir-126, bta-mir-181a-2, bta-mir-27b, bta-mir-31, bta-mir-15b, bta-mir-215, bta-mir-30e, bta-mir-148b, bta-mir-192, bta-mir-200a, bta-mir-200c, bta-mir-23a, bta-mir-29b-2, bta-mir-29c, bta-mir-10b, bta-mir-24-2, bta-mir-30a, bta-mir-200b, bta-let-7a-1, bta-mir-342, bta-let-7f-1, bta-let-7a-2, bta-let-7a-3, bta-mir-103-2, bta-mir-125b-2, bta-mir-15a, bta-mir-99b, hsa-mir-664a, ssc-mir-99b, hsa-mir-103b-1, hsa-mir-103b-2, ssc-mir-15a, ssc-mir-16-2, ssc-mir-16-1, bta-mir-141, bta-mir-143, bta-mir-146a, bta-mir-152, bta-mir-155, bta-mir-16a, bta-mir-184, bta-mir-24-1, bta-mir-223, bta-mir-224, bta-mir-26a-1, bta-mir-296, bta-mir-29d, bta-mir-378-1, bta-mir-451, bta-mir-486, bta-mir-671, bta-mir-29e, bta-mir-29b-1, bta-mir-181a-1, ssc-mir-181a-1, ssc-mir-215, ssc-mir-30a, bta-mir-2318, bta-mir-2339, bta-mir-2430, bta-mir-664a, bta-mir-378-2, ssc-let-7a-1, ssc-mir-378-1, ssc-mir-29a, ssc-mir-30e, ssc-mir-499, ssc-mir-143, ssc-mir-10b, ssc-mir-486-1, ssc-mir-152, ssc-mir-103-2, ssc-mir-181a-2, ssc-mir-27b, ssc-mir-24-2, ssc-mir-99a, ssc-mir-148b, ssc-mir-664, ssc-mir-192, ssc-mir-342, ssc-mir-125b-1, oar-mir-21, oar-mir-29a, oar-mir-125b, oar-mir-181a-1, hsa-mir-378b, hsa-mir-378c, ssc-mir-296, ssc-mir-155, ssc-mir-146a, bta-mir-148c, ssc-mir-126, ssc-mir-378-2, ssc-mir-451, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-378f, hsa-mir-378g, hsa-mir-378h, hsa-mir-378i, hsa-mir-451b, hsa-mir-499b, ssc-let-7a-2, ssc-mir-486-2, hsa-mir-664b, hsa-mir-378j, ssc-let-7f-2, ssc-mir-31, ssc-mir-671, bta-mir-378b, bta-mir-378c, hsa-mir-486-2, oar-let-7a, oar-let-7f, oar-mir-103, oar-mir-10b, oar-mir-143, oar-mir-148a, oar-mir-152, oar-mir-16b, oar-mir-181a-2, oar-mir-200a, oar-mir-200b, oar-mir-200c, oar-mir-23a, oar-mir-26a, oar-mir-29b-1, oar-mir-30a, oar-mir-99a, bta-mir-664b, chi-let-7a, chi-let-7f, chi-mir-103, chi-mir-10b, chi-mir-125b, chi-mir-126, chi-mir-141, chi-mir-143, chi-mir-146a, chi-mir-148a, chi-mir-148b, chi-mir-155, chi-mir-15a, chi-mir-15b, chi-mir-16a, chi-mir-16b, chi-mir-184, chi-mir-192, chi-mir-200a, chi-mir-200b, chi-mir-200c, chi-mir-215, chi-mir-21, chi-mir-223, chi-mir-224, chi-mir-2318, chi-mir-23a, chi-mir-24, chi-mir-26a, chi-mir-27b, chi-mir-296, chi-mir-29a, chi-mir-29b, chi-mir-29c, chi-mir-30a, chi-mir-30e, chi-mir-342, chi-mir-378, chi-mir-451, chi-mir-499, chi-mir-671, chi-mir-99a, chi-mir-99b, bta-mir-378d, ssc-mir-378b, oar-mir-29b-2, ssc-mir-141, ssc-mir-200b, ssc-mir-223, bta-mir-148d
Members of the miRNA-29 family have been shown to revert aberrant methylation in lung cancer by targeting DNA methyltransferases 3A and 3B (Fabbri et al., 2007).
[score:3]
MicroRNA-29 family reverts aberrant methylation in lung cancer by targeting DNA methyltransferases 3A and 3B.
[score:2]
[1 to 20 of 2 sentences]
|
21 |
![]()
Other miRNAs from this paper: hsa-mir-29a, hsa-mir-29b-1, hsa-mir-29b-2, mmu-mir-1a-1, mmu-mir-29b-1, mmu-mir-133a-1, mmu-mir-206, hsa-mir-34a, hsa-mir-214, mmu-mir-34c, mmu-mir-34b, hsa-mir-1-2, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-206, mmu-mir-29a, mmu-mir-29c, mmu-mir-34a, hsa-mir-1-1, mmu-mir-1a-2, mmu-mir-214, mmu-mir-29b-2, hsa-mir-29c, hsa-mir-34b, hsa-mir-34c, mmu-mir-133a-2, mmu-mir-133b, hsa-mir-133b, ssc-mir-29b-1, ssc-mir-214, ssc-mir-29c, hsa-mir-486-1, hsa-mir-499a, mmu-mir-486a, mmu-mir-499, mmu-mir-208b, hsa-mir-208b, mmu-mir-1b, ssc-mir-34a, ssc-mir-133a-1, ssc-mir-1, ssc-mir-206, ssc-mir-133b, ssc-mir-29a, ssc-mir-499, ssc-mir-486-1, ssc-mir-208b, ssc-mir-34c-1, mmu-mir-486b, ssc-mir-133a-2, hsa-mir-499b, ssc-mir-486-2, mmu-mir-133c, ssc-mir-34c-2, hsa-mir-486-2
Many miRNAs, such as miR-1, miR-133, miR-29, miR-214, miR-206, miR-486, miR-208b, and miR-499 were involved in the regulation of skeletal myogenesis by binding to its target genes 36, 37.
[score:4]
[1 to 20 of 1 sentences]
|
22 |
![]()
Other miRNAs from this paper: ssc-mir-148a, ssc-mir-181b-2, ssc-mir-23a, ssc-mir-26a, ssc-mir-29b-1, ssc-mir-181c, ssc-mir-214, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-128-1, ssc-mir-21, ssc-mir-29c, ssc-mir-99b, ssc-mir-199b, ssc-mir-133a-1, ssc-mir-1, ssc-mir-181a-1, ssc-mir-30a, ssc-mir-206, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-378-1, ssc-mir-133b, ssc-mir-29a, ssc-mir-30d, ssc-mir-199a-2, ssc-mir-128-2, ssc-mir-320, ssc-mir-143, ssc-mir-10b, ssc-mir-181a-2, ssc-mir-181b-1, ssc-mir-181d, ssc-mir-100, ssc-mir-196b-1, ssc-mir-542, ssc-mir-127, ssc-mir-369, ssc-mir-155, ssc-mir-133a-2, ssc-mir-126, ssc-mir-199a-1, ssc-mir-378-2, ssc-let-7a-2, ssc-let-7d, ssc-let-7f-2, ssc-mir-196b-2, ssc-mir-378b, ssc-mir-141
miR-29 negatively regulates skeletal myogenesis by targeting Ring1 and YY1 -binding protein (Rybp) [43].
[score:4]
[1 to 20 of 1 sentences]
|
23 |
![]()
Other miRNAs from this paper: mmu-mir-29b-1, mmu-mir-29a, mmu-mir-29c, mmu-mir-29b-2, mmu-mir-365-1, mmu-mir-365-2, ssc-mir-29b-1, ssc-mir-29c, ssc-mir-29a, ssc-mir-365-2, ssc-mir-365-1
Wei W. He H. B. Zhang W. Y. Zhang H. X. Bai J. B. Liu H. Z. Cao J. H. Chang K. C. Li X. Y. Zhao S. H. miR-29 targets Akt3 to reduce proliferation and facilitate differentiation of myoblasts in skeletal muscle development Cell Death Dis.
[score:4]
[1 to 20 of 1 sentences]
|
24 |
![]()
Other miRNAs from this paper: hsa-mir-20a, hsa-mir-29a, hsa-mir-30a, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-148a, hsa-mir-1-2, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-135a-1, hsa-mir-135a-2, hsa-mir-137, hsa-mir-127, hsa-mir-186, hsa-mir-206, hsa-mir-1-1, hsa-mir-29c, hsa-mir-378a, hsa-mir-135b, hsa-mir-335, hsa-mir-133b, hsa-mir-20b, hsa-mir-451a, ssc-mir-135-1, ssc-mir-135-2, ssc-mir-148a, ssc-mir-20a, ssc-mir-29b-1, ssc-mir-186, ssc-mir-29c, hsa-mir-499a, hsa-mir-378d-2, ssc-mir-133a-1, ssc-mir-1, ssc-mir-30a, ssc-mir-206, ssc-mir-378-1, ssc-mir-133b, ssc-mir-29a, ssc-mir-499, ssc-mir-127, ssc-mir-335, hsa-mir-378b, hsa-mir-378c, ssc-mir-133a-2, ssc-mir-378-2, ssc-mir-451, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-378f, hsa-mir-378g, hsa-mir-378h, hsa-mir-378i, hsa-mir-451b, hsa-mir-499b, hsa-mir-378j, ssc-mir-20b-1, ssc-mir-20b-2, ssc-mir-137, ssc-mir-378b
A lecture showed that the inhibition of miR-29 by TGF-beta-Smad3 signaling through dual mechanisms promotes the trans-differentiation of mouse myoblasts into myofibroblasts [23].
[score:3]
In G2 (down), the most significant differences between breeds were observed at 2 dpn, when miR-29 and miR-499 showed drastic changes.
[score:1]
[1 to 20 of 2 sentences]
|
25 |
![]()
Other miRNAs from this paper: ssc-mir-145, ssc-mir-29b-1, ssc-mir-124a-2, ssc-mir-30c-2, ssc-mir-16-2, ssc-mir-16-1, ssc-mir-124a-1, ssc-mir-133a-1, ssc-mir-1, ssc-let-7a-1, ssc-mir-155, ssc-mir-133a-2, ssc-mir-30c-1, ssc-let-7a-2
2009.02.038) miR-29b-3p Potential regulator of TGFB1 29 5.48E-07 −5.260 Lu et al. 2016 (10.1096/fj.
[score:2]
[1 to 20 of 1 sentences]
|
26 |
![]()
Other miRNAs from this paper: ssc-mir-106a, ssc-mir-122, ssc-mir-145, ssc-mir-20a, ssc-mir-26a, ssc-mir-28, ssc-mir-29b-1, ssc-mir-140, ssc-let-7c, ssc-let-7f-1, ssc-let-7i, ssc-mir-103-1, ssc-mir-196a-2, ssc-mir-16-2, ssc-mir-16-1, ssc-mir-1, ssc-let-7a-1, ssc-let-7e, ssc-let-7g, ssc-mir-133b, ssc-mir-191, ssc-mir-320, ssc-mir-143, ssc-mir-423, ssc-mir-103-2, ssc-mir-98, ssc-mir-192, ssc-mir-196b-1, ssc-mir-676-1, ssc-mir-708, ssc-mir-505, ssc-let-7a-2, ssc-let-7d, ssc-let-7f-2, ssc-mir-20b-1, ssc-mir-20b-2, ssc-mir-196a-1, ssc-mir-196b-2, ssc-mir-676-2, ssc-mir-223, ssc-mir-375
For example, we found that the size of ssc-miR-29b in our library is 23 (read number is 88), while the size of miR-29b in miRBase is 20 (read number is 11).
[score:1]
[1 to 20 of 1 sentences]
|
27 |
![]()
miR-29a-5p has been described in many species like human, bovine, mouse, but it has not been described in pigs, where the precursor miR-29 has been included in miRBase along with the mature ssc-miR-29a-3p.
[score:1]
[1 to 20 of 1 sentences]
|
28 |
![]()
Other miRNAs from this paper: ssc-mir-122, ssc-mir-29b-1, ssc-mir-140, ssc-mir-21, ssc-mir-29c, ssc-mir-1, ssc-mir-29a, ssc-mir-328, ssc-mir-144
miR-29 was decreased in dogs with ventricular tachypacing -induced congestive heart failure (CHF) and in patients with CHF or AF [28].
[score:1]
[1 to 20 of 1 sentences]
|