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7 publications mentioning rno-mir-207

Open access articles that are associated with the species Rattus norvegicus and mention the gene name mir-207. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 28
Based on functional and pathway analysis and related literature examination, we selected miR-134-5p, miR-207, and miR-465-5p to represent the up-regulated miRNAs, and miR-30b-5p, miR-19a-3p, and miR-130a-3p to represent the down-regulated miRNAs. [score:7]
miR-207 has been previously shown to enhance radiation -induced apoptosis by directly targeting Akt3, while targeting miR-207 protected cochlea hair cells from ionizing radiation (Tan et al., 2014). [score:6]
Among these DEmiRNAs, 6 up- or down-regulated miRNAs were chosen for further analysis, including miR-134-5p, miR-207, miR-465-5p, miR-30b-5p, miR-19a-3p, and miR-130a-3p. [score:4]
In our network, Chst1 (carbohydrate sulfotransferase 1) is regulated by four miRNAs, including miR-30b-5p, miR-19a-3p, miR-130a-3p, and miR-134-5p, while Nrbf2 (nuclear receptor binding factor 2) is regulated by miR-30b-5p, miR-19a-3p, miR-130a-3p, and miR-207. [score:3]
The DEmiRNAs (e. g., miR-134-5p, miR-207, miR-465-5p, miR-30b-5p, miR-19a-3p, and miR-130a-3p) and common target genes, such as Chst1 and Nrbf2, may be strongly associated with the pulmonary inflammation induced by ZnO-NPs. [score:3]
MicroRNA-207 enhances radiation -induced apoptosis by directly targeting akt3 in cochlea hair cells. [score:3]
Additionally, in our study, Nrbf2 was regulated by miR-30b-5p, miR-19a-3p, miR-130a-3p, and miR-207. [score:2]
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2
[+] score: 8
We found that up-regulated miRNAs (miR-346, miR-135b, miR-30ab, miR-344, miR-18a, miR-99a, miR-210, miR-207, miR-18a, and miR-129) in ARDS were inversely correlated with the expression of their predicted targets such as Gabrb1, Mdh1, Eif2ak1, Fbln5, and Tspan8. [score:8]
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3
[+] score: 6
Few of the highly upregulated microRNAs were: miR-79, miR-183, miR-206, miR-207, miR-296-3p, miR-298, miR-380-5p, miR-433, miR-449b, miR-705, miR-761 (S1 Table). [score:4]
Majority of the dysregulated microRNAs e. g. miR-380, miR-207, miR-79, miR-129, miR-153, miR-183, etc. [score:2]
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4
[+] score: 5
RT-PCR validation of these 9 miRNAs confirmed the high expression of 7 miRNAs, of which 3 (miR-503, miR-330 and miR-293) were exclusively expressed in dnIKK2-Treg-EV, while the remaining 4 miRNAs (miR-297c, miR-207, miR-9, miR-484) were faintly detected in Tact-EV and Trest-EV too (Fig.   5A). [score:5]
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5
[+] score: 3
6 was illustrated in Fig.   8B, including rno-mir-207, rno-mir-152, rno-mir-133a, may repress the expression of negative modulators, for example, Fads6, Samd14 and Fbxo46, respectively. [score:3]
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6
[+] score: 2
For example rno-miR-495 and rno-miR-207 are common between the 24 h and 7d time points, but they are rank-ordered differently between the 2 time points. [score:1]
The top three rno-miRs at 24 h were rno-miR-495* (degree of 172), followed by rno-miR-214 (degree of 170) and rno-miR-207(degree of 143). [score:1]
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7
[+] score: 1
However, trimming for several miRNAs including rno-miR-1*, rno-miR-196a*, rno-miR-207, rno-miR-347, and rno-miR-742 was not detected, possibly due to the low abundance of trimmed isoforms rather than a selective protection of modifications. [score:1]
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