sort by

11 publications mentioning mmu-mir-541

Open access articles that are associated with the species Mus musculus and mention the gene name mir-541. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 181
Other miRNAs from this paper: mmu-mir-26a-1, mmu-mir-96, mmu-mir-26a-2
Suppression of miR-541 was correlated with upregulation of p15, MDM2 and phospho-MDM2, but downregulation of phospho-p53 in the testicular tissue irrespective of prior exposure of the mice to MC-LR (Figure 5C). [score:9]
We found that the mimic increased the expression of miR-541, whereas the inhibitor suppressed the expression of miR-541. [score:9]
Upregulating miR-541 suppressed the expression of p15, MDM2 (murine double minute2) and phospho-MDM2, while it increased the level of phospho-p53 (Figure 2C). [score:8]
Upregulating the expression of miR-541 suppressed the levels of p15, MDM2, and phospho-MDM2 while it increased the level of phospho-p53 (Figure 5C). [score:8]
On the contrary, suppressing the expression of miR-541 could substantially attenuate MC-LR -induced cell apoptosis through increasing the expression of p15. [score:7]
In the current work, we confirmed that miR-541 can directly target the 3′-UTR of p15, a cyclin -dependent kinase (CDK) inhibitor, which is proposed to be involved in cell cycle regulation [28]. [score:7]
Furthermore, forced expression of miR-541 promoted MC-LR -induced cell apoptosis, whereas inhibiting miR-541 suppressed apoptosis (Figure 2D,E). [score:7]
To examine the regulatory effects of miR-541 and p15 on cellular functions, we transfected GC-1 cells with miR-541 -inhibitor, miR-541 -mimic, and their negative controls followed by exposure to 500 nM MC-LR for 24 h. miR-541 was decreased in cells transfected with miR-541 -inhibitor and highly increased in cells transfected with miR-541 -mimic (Figure 2A). [score:6]
The expression of miR-541 was significantly upregulated after exposure to MC-LR at these concentrations (Figure 1B). [score:6]
In our study, we found that miR-541 could induce cell apoptosis through diminishing the expression of p15, which would in turn downregulate both total MDM2 and phospho-MDM2, while increase phospho-p53. [score:6]
Interestingly, miR-541 -inhibitor was potent in antagonizing MC-LR -mediated upregulation of miR-541 (Figure 5A). [score:6]
We constructed lentiviral vectors that contain miR-541 -mimic or miR-541 -inhibitor, or their respective negative controls (inhibitor-NC and mimic-NC) separately (Genechem, Shanghai, China). [score:5]
The expression of p15 mRNA remained unchanged following transfection with either miR-541 -mimic or the inhibitor (Figure 2B). [score:5]
Among these regulated miRNAs, miR-541 stood out as the most significantly upregulated. [score:5]
Figure S1: Toxic effects of MC-LR on GC-1 cells; Figure S2: Intracellular MC-LR detected in GC-1 cells exposed to MC-LR for 24 h; Figure S3: Efferent duct injection; Figure S4: GV306 plasmid vector map; Table S1: Animals and treatment; Table S2: The sequence design of miR-541 -inhibitor, inhibitor negtive control, miR-541 -mimic and mimic negative control; Table S3: Oligonucleotide Sequences used in this study; Table S4: The wild type and mutant type of p15 3′UTR sequences containing the miR-541 binding site. [score:5]
We found that the MC-LR -induced toxic effects may be mediated by overexpression of miR-541, which promotes cell apoptosis by targeting p15 and a group of downstream apoptosis -associated proteins. [score:5]
Moreover, testicular structural damage caused by MC-LR was relieved by suppressing intracellular expression of miR-541. [score:5]
Next, we tried to predict the potential targets of miR-541 from TargetScan Human V 6.2, miRanda, and miRBase. [score:5]
MC-LR Regulates the Expression of miR-541 and p15 in Vivo. [score:4]
The Expression of p15 Is Regulated by miR-541 in GC-1 Cells. [score:4]
In this study, we confirmed that miR-541 was upregulated after treatment of the cells with MC-LR. [score:4]
However, mRNA expression of p15 was not modulated by the regulation of miR-541, which was consistent with our in vitro data (Figure 5B). [score:4]
Our results suggest that miR-541 may regulate the expression of p15 in a post-transcriptional manner. [score:4]
miR-541 was significantly upregulated when higher concentrations of MC-LR (15 μg/kg and 30 μg/kg) were delivered (Figure 4A). [score:4]
p15 stood out as one of the target mRNAs of miR-541. [score:3]
Exactly the opposite trends were observed if intracellular miR-541 was suppressed. [score:3]
Inhibition of miR-541 Protects GC-1 Cells from MC-LR-Induced Cell Death in Vivo. [score:3]
Inhibition of miR-541 Protects GC-1 Cells from MC-LR-Induced Cell Death in Vitro. [score:3]
Furthermore, inhibiting miR-541 effectively restored testicular structures, mature sperm counts, and testicular cell viability following exposure to MC-LR (Figure 5E,F). [score:3]
293T cells were seeded into 24-well plates, co -transfected with 50 nM miR-541 or a scrambled mimic and 600 ng of a dual luciferase vector expressing the wild-type or mutant 3′-UTR p15 sequences. [score:3]
Constructs containing miR-541 -mimic, miR-541 -inhibitor, and their negative controls were delivered through the efferent ducts. [score:3]
Interfering with the intracellular expression of miR-541 may protect germ cells from the cytotoxic effects of MC-LR. [score:3]
The expression of miR-541 and p15 was determined in testicular tissue from either control mice or mice treated with MC-LR. [score:3]
Luciferase activity of the system containing wild-type, but not mutated, p15 3′-UTR was substantially decreased after co-transfection with miR-541 mimic (Figure 1E), which supported the prediction that p15 was the target gene of miR-541. [score:3]
In our study, we demonstrated that miR-541 could mediate post-transcriptional regulation of p15 in testes. [score:2]
In conclusion, we revealed the roles of miR-541 in apoptotic regulation and its potential contribution to MC-LR induced toxicity in spermatogonial cells, which may provide a mechanistic explanation for reproductive malfunction including declines in sperm quality. [score:2]
p15 3′-UTR and p15 3′-UTR with mutations in the predicted miR-541 binding site were cloned separately into the luciferase reporter system (GV306 vector). [score:2]
We further investigated the effect of manipulated miR-541 expression on the protein levels of p15, MDM2, phospho-MDM2, p53 and phospho-p53 either with or without exposure to MC-LR by Western blot. [score:1]
miR-541 was significantly increased in GC-1 cells after exposure to 500 nM MC-LR. [score:1]
The 3′-UTR of p15 has a miR-541 binding sequence. [score:1]
The specific function of miR-541 and p15 in germline aroused our interest. [score:1]
The GV306 vectors were then co -transfected with either miR-541 mimic or its negative control. [score:1]
In summary, the current study has revealed an important link among MC-LR, miR-541, and p15 in the male reproductive system using mouse mo dels. [score:1]
The binding site for miR-541 in the 3′-UTR of p15 was highly conserved (Figure 1C,D). [score:1]
[1 to 20 of 44 sentences]
2
[+] score: 40
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-16-1, hsa-mir-17, hsa-mir-18a, hsa-mir-19a, hsa-mir-19b-1, hsa-mir-19b-2, hsa-mir-21, hsa-mir-23a, hsa-mir-30a, hsa-mir-98, hsa-mir-16-2, mmu-let-7g, mmu-let-7i, mmu-mir-15b, mmu-mir-30a, mmu-mir-30b, mmu-mir-101a, mmu-mir-125a, mmu-mir-125b-2, mmu-mir-9-2, mmu-mir-132, mmu-mir-133a-1, mmu-mir-135a-1, mmu-mir-150, mmu-mir-155, mmu-mir-204, mmu-mir-205, hsa-mir-30c-2, hsa-mir-30d, mmu-mir-30e, hsa-mir-34a, hsa-mir-204, hsa-mir-205, hsa-mir-217, mmu-mir-34c, mmu-mir-34b, mmu-let-7d, hsa-let-7g, hsa-let-7i, hsa-mir-15b, hsa-mir-30b, hsa-mir-125b-1, hsa-mir-132, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-135a-1, hsa-mir-135a-2, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-125a, hsa-mir-125b-2, hsa-mir-150, mmu-mir-19b-2, mmu-mir-30c-1, mmu-mir-30c-2, mmu-mir-30d, mmu-let-7a-1, mmu-let-7a-2, mmu-let-7b, mmu-let-7c-1, mmu-let-7c-2, mmu-let-7e, mmu-let-7f-1, mmu-let-7f-2, mmu-mir-15a, mmu-mir-16-1, mmu-mir-16-2, mmu-mir-18a, mmu-mir-21a, mmu-mir-23a, mmu-mir-34a, mmu-mir-98, mmu-mir-322, mmu-mir-338, hsa-mir-155, mmu-mir-17, mmu-mir-19a, mmu-mir-135a-2, mmu-mir-19b-1, mmu-mir-9-1, mmu-mir-9-3, mmu-mir-125b-1, mmu-mir-217, hsa-mir-30c-1, hsa-mir-34b, hsa-mir-34c, hsa-mir-30e, hsa-mir-338, mmu-mir-133a-2, mmu-mir-133b, hsa-mir-133b, hsa-mir-18b, hsa-mir-503, mmu-mir-503, mmu-mir-744, mmu-mir-18b, hsa-mir-541, hsa-mir-744, mmu-mir-133c, mmu-mir-21b, mmu-let-7j, mmu-mir-21c, mmu-mir-30f, mmu-let-7k, mmu-mir-9b-2, mmu-mir-9b-1, mmu-mir-9b-3
However, a direct target site of miR-541 on OPN/SPP1 has not been identified yet, indicating an unknown indirect mechanism. [score:5]
In fact, knockdown of miR-541 upregulated OPN/SPP1 and mineralization. [score:5]
In attempt to clarify a function of miR-541 and miR-155 during osteogenesis of hMSC, we transiently transfected these cells with antagonists/anti-miR targeting these two types of miRNAs. [score:3]
Target of miR-30 family, miR-34 family, let-7 family, miR-15/16 family (including miR-322/424), miR-21 family, miR-541/654 was predicted and selected using cut off score −0.2. [score:3]
Human miR-541 and miR-155 function and expression pattern in hMSC/hMBSC osteoblastic differentiation. [score:3]
0058796.g010 Figure 10(A) Expression pattern of miR-541 and miR-155 with (osteo) or w/o (control) osteo-induction. [score:3]
Expression and function of miR-541 and miR-155 during hBMSC/MSC osteogenesis. [score:3]
Taken together, these data indicate that miR-541 is a negative regulator of osteoblast differentiation of hMSC. [score:2]
Despite this apparently low efficiency, a significant increase by 1.8-fold in the osteoblastic marker OPN/SPP1 mRNA level was observed by miR-541 knock down (Fig 10C). [score:2]
There were no significant changes in ALP staining of upon miR-155 or miR-541 knock down. [score:2]
Besides, mouse miR-541 was strongly induced in 4 hours of initial induction, and then reduced in the later stage (Fig 3B and Fig 4), while in hMSC, miR-541 was gradually reduced in long culture, suggesting some role for miR-541 in osteogenesis. [score:1]
We focused on the miR-30 family and miR-541 in this study, while still further analyzing roles of OstemiR in MSC differentiation. [score:1]
miR-541 and miR-155 were induced in 4 hours after the osteo-stimulation to KUSA-A1, but not on the day 14 (Fig 3B, Fig 4). [score:1]
Together with these results and data interpretations, we propose the tuning mo del of canonical and novel osteogenic factors by the OstemiRs including miR-30 family and miR-541. [score:1]
The efficiency of anti-miR-155 or anti-miR-541 knockdown was of approximately 20–40% compared with the control siRNA transfections (Fig 10B). [score:1]
Tuning mo del of canonical and novel osteogenic factors by miRNA-30 family and miR-541 during MSC osteogenesis. [score:1]
Human OPN/SPP1 in hBMSC/MSC is attenuated by miR-541. [score:1]
ALP mRNA level in anti-miR-541 transfectants was higher than that in the control. [score:1]
In accordance, calcium deposition on the anti-miR541 -treated cells was more rich than those of controls in a result of alizarin red S staining on day 7, comparing center regions in each wells (Fig 10D). [score:1]
[1 to 20 of 19 sentences]
3
[+] score: 6
We have shown that in tumor samples compared to normal samples, the majority of miRNAs (miR-216, miR-217, miR-100, miR-345, miR-141, miR-483-3p, miR-26b, miR-150, Let-7b, Let-195 and miR-96) were downregulated, and few were upregulated (miR-146b, miR-205, miR-31, miR-192, miR-194 21, miR-379, miR-431, miR-541, and miR-199b). [score:6]
[1 to 20 of 1 sentences]
4
[+] score: 5
The array uncovered the induction of 117 miRNAs with the signal intensity ≥500 (the fluorescence amount of each miRNA probe is measured by a photo multiplier tube or charge-coupled device and signal scaled across the range of detection for the platform) in GA muscle (Table 1, Fig. 1A and 1B), including the highly downregulated miRNAs (≥1.5-fold) miR-194-5p, miR-101b-3p, miR-148a-3p, miR-199b-5p, miR-335-5p, miR-127-3p, miR-379-5p, miR-541-5p, miR-382-5p, miR-329-3p, miR-299-5p and miR-434-3p, and the highly up-regulated miRNAs (≥1.5 fold), miR-146b-5p and miR-146a-5p (Fig. 1C). [score:5]
[1 to 20 of 1 sentences]
5
[+] score: 4
On that basis, we identified miRNAs (e. g. miR-409-5p, miR-433, miR-541, miR-742) that, in the adult retina, are clearly detected in the vitreal part of the Inner Nuclear Layer (INL) and are likely to be expressed in amacrine cells (blue arrows in the third column of Figure 4A; Database). [score:3]
For miR-409-5p, miR-433, miR-541 and miR-742 (top bracket; panel A) a weak staining is detectable in the inner neuroblastic layer (INBL; blue arrowheads) at P0. [score:1]
[1 to 20 of 2 sentences]
6
[+] score: 3
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-16-1, hsa-mir-17, hsa-mir-20a, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-26a-1, hsa-mir-26b, hsa-mir-29a, hsa-mir-30a, hsa-mir-93, hsa-mir-101-1, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-107, hsa-mir-16-2, mmu-let-7g, mmu-let-7i, mmu-mir-15b, mmu-mir-23b, mmu-mir-29b-1, mmu-mir-30a, mmu-mir-30b, mmu-mir-101a, mmu-mir-124-3, mmu-mir-125a, mmu-mir-130a, mmu-mir-9-2, mmu-mir-135a-1, mmu-mir-136, mmu-mir-138-2, mmu-mir-140, mmu-mir-144, mmu-mir-145a, mmu-mir-146a, mmu-mir-149, mmu-mir-152, mmu-mir-10b, mmu-mir-181a-2, mmu-mir-182, mmu-mir-183, mmu-mir-185, mmu-mir-24-1, mmu-mir-191, mmu-mir-193a, mmu-mir-195a, mmu-mir-200b, mmu-mir-204, hsa-mir-30c-2, hsa-mir-30d, mmu-mir-30e, hsa-mir-7-1, hsa-mir-7-2, hsa-mir-7-3, hsa-mir-10a, hsa-mir-10b, hsa-mir-34a, hsa-mir-181a-2, hsa-mir-181b-1, hsa-mir-181c, hsa-mir-182, hsa-mir-183, hsa-mir-204, hsa-mir-181a-1, hsa-mir-221, hsa-mir-222, hsa-mir-200b, mmu-mir-301a, mmu-mir-34c, mmu-mir-34b, mmu-let-7d, mmu-mir-130b, hsa-let-7g, hsa-let-7i, hsa-mir-15b, hsa-mir-23b, hsa-mir-30b, hsa-mir-124-1, hsa-mir-124-2, hsa-mir-124-3, hsa-mir-130a, hsa-mir-135a-1, hsa-mir-135a-2, hsa-mir-138-2, hsa-mir-140, hsa-mir-144, hsa-mir-145, hsa-mir-152, hsa-mir-191, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-125a, hsa-mir-136, hsa-mir-138-1, hsa-mir-146a, hsa-mir-149, hsa-mir-185, hsa-mir-193a, hsa-mir-195, hsa-mir-320a, mmu-mir-30c-1, mmu-mir-30c-2, mmu-mir-30d, mmu-let-7a-1, mmu-let-7a-2, mmu-let-7b, mmu-let-7c-1, mmu-let-7c-2, mmu-let-7e, mmu-let-7f-1, mmu-let-7f-2, mmu-mir-15a, mmu-mir-16-1, mmu-mir-16-2, mmu-mir-20a, mmu-mir-23a, mmu-mir-24-2, mmu-mir-26a-1, mmu-mir-26b, mmu-mir-29a, mmu-mir-29c, mmu-mir-93, mmu-mir-34a, mmu-mir-330, mmu-mir-339, mmu-mir-340, mmu-mir-135b, mmu-mir-101b, hsa-mir-200c, hsa-mir-181b-2, mmu-mir-107, mmu-mir-10a, mmu-mir-17, mmu-mir-200c, mmu-mir-181a-1, mmu-mir-320, mmu-mir-26a-2, mmu-mir-221, mmu-mir-222, mmu-mir-29b-2, mmu-mir-135a-2, mmu-mir-124-1, mmu-mir-124-2, mmu-mir-9-1, mmu-mir-9-3, mmu-mir-138-1, mmu-mir-181b-1, mmu-mir-181c, mmu-mir-7a-1, mmu-mir-7a-2, mmu-mir-7b, hsa-mir-29c, hsa-mir-30c-1, hsa-mir-101-2, hsa-mir-34b, hsa-mir-34c, hsa-mir-301a, hsa-mir-130b, hsa-mir-30e, hsa-mir-26a-2, hsa-mir-361, mmu-mir-361, hsa-mir-376a-1, mmu-mir-376a, hsa-mir-340, hsa-mir-330, hsa-mir-135b, hsa-mir-339, hsa-mir-335, mmu-mir-335, mmu-mir-181b-2, mmu-mir-376b, mmu-mir-434, mmu-mir-467a-1, hsa-mir-376b, hsa-mir-485, hsa-mir-146b, hsa-mir-193b, hsa-mir-181d, mmu-mir-485, hsa-mir-376a-2, hsa-mir-320b-1, hsa-mir-320c-1, hsa-mir-320b-2, mmu-mir-301b, mmu-mir-674, mmu-mir-146b, mmu-mir-467b, mmu-mir-669c, mmu-mir-708, mmu-mir-676, mmu-mir-181d, mmu-mir-193b, mmu-mir-467c, mmu-mir-467d, hsa-mir-541, hsa-mir-708, hsa-mir-301b, mmu-mir-467e, mmu-mir-467f, mmu-mir-467g, mmu-mir-467h, hsa-mir-320d-1, hsa-mir-320c-2, hsa-mir-320d-2, mmu-mir-467a-2, mmu-mir-467a-3, mmu-mir-467a-4, mmu-mir-467a-5, mmu-mir-467a-6, mmu-mir-467a-7, mmu-mir-467a-8, mmu-mir-467a-9, mmu-mir-467a-10, hsa-mir-320e, hsa-mir-676, mmu-mir-101c, mmu-mir-195b, mmu-mir-145b, mmu-let-7j, mmu-mir-130c, mmu-mir-30f, mmu-let-7k, mmu-mir-9b-2, mmu-mir-124b, mmu-mir-9b-1, mmu-mir-9b-3
Other differentially expressed miRNAs specific to the mouse mo del were also validated by qPCR, including miR-195 (member of the miR-15 family) and miR-541 family members. [score:3]
[1 to 20 of 1 sentences]
7
[+] score: 3
Other miRNAs from this paper: mmu-mir-34c, mmu-mir-34b, mmu-mir-34a
Lu et al. [30] showed that inhibition of growth and metastasis of non-small-cell lung cancer cell by miR-541-3p could be reversed by TGIF2. [score:3]
[1 to 20 of 1 sentences]
8
[+] score: 3
The panel of the 776 miRNAs screened in this study included other miRNAs whose genes are also located in this region, such as miR-300 and miR-541, but their expression was not modulated with passage of VERO cells. [score:3]
[1 to 20 of 1 sentences]
9
[+] score: 1
Effects of miR-541 on neurite outgrowth during neuronal differentiation. [score:1]
[1 to 20 of 1 sentences]
10
[+] score: 1
86 miR-873-as 1,016 e-miR-743b-5p 23.75 miR-449c-as 515e-miR-715 || 69.35 miR-541-as 439 e-miR-881* 56.21 miR-148b-as 336 e-miR-370 97.41 miR-546-as 333 e-miR-3067 100 miR-3074-as 262 e-miR-448-5p 100miR-451-as ‡ 286 e-miR-669o-5p 99.35 † Novel miR* that are processed within the expected window of the mature strand are labelled “generic”. [score:1]
[1 to 20 of 1 sentences]
11
[+] score: 1
Other miRNAs from this paper: mmu-mir-1a-1, mmu-mir-127, mmu-mir-134, mmu-mir-136, mmu-mir-154, mmu-mir-181a-2, mmu-mir-143, mmu-mir-196a-1, mmu-mir-196a-2, mmu-mir-21a, rno-mir-329, mmu-mir-329, mmu-mir-1a-2, mmu-mir-181a-1, mmu-mir-181b-1, mmu-mir-181c, mmu-mir-375, mmu-mir-379, mmu-mir-181b-2, rno-mir-21, rno-mir-127, rno-mir-134, rno-mir-136, rno-mir-143, rno-mir-154, rno-mir-181c, rno-mir-181a-2, rno-mir-181b-1, rno-mir-181b-2, rno-mir-196a, rno-mir-181a-1, mmu-mir-196b, rno-mir-196b-1, mmu-mir-412, mmu-mir-370, oar-mir-431, oar-mir-127, oar-mir-432, oar-mir-136, mmu-mir-431, mmu-mir-433, rno-mir-431, rno-mir-433, ssc-mir-181b-2, ssc-mir-181c, ssc-mir-136, ssc-mir-196a-2, ssc-mir-21, rno-mir-370, rno-mir-412, rno-mir-1, mmu-mir-485, rno-mir-541, rno-mir-493, rno-mir-379, rno-mir-485, mmu-mir-668, bta-mir-21, bta-mir-181a-2, bta-mir-127, bta-mir-181b-2, bta-mir-181c, mmu-mir-181d, mmu-mir-493, rno-mir-181d, rno-mir-196c, rno-mir-375, mmu-mir-1b, bta-mir-1-2, bta-mir-1-1, bta-mir-134, bta-mir-136, bta-mir-143, bta-mir-154a, bta-mir-181d, bta-mir-196a-2, bta-mir-196a-1, bta-mir-196b, bta-mir-329a, bta-mir-329b, bta-mir-370, bta-mir-375, bta-mir-379, bta-mir-412, bta-mir-431, bta-mir-432, bta-mir-433, bta-mir-485, bta-mir-493, bta-mir-541, bta-mir-181a-1, bta-mir-181b-1, ssc-mir-1, ssc-mir-181a-1, mmu-mir-432, rno-mir-668, ssc-mir-143, ssc-mir-181a-2, ssc-mir-181b-1, ssc-mir-181d, ssc-mir-196b-1, ssc-mir-127, ssc-mir-432, oar-mir-21, oar-mir-181a-1, oar-mir-493, oar-mir-433, oar-mir-370, oar-mir-379, oar-mir-329b, oar-mir-329a, oar-mir-134, oar-mir-668, oar-mir-485, oar-mir-154a, oar-mir-154b, oar-mir-541, oar-mir-412, mmu-mir-21b, mmu-mir-21c, ssc-mir-196a-1, ssc-mir-196b-2, ssc-mir-370, ssc-mir-493, bta-mir-154c, bta-mir-154b, oar-mir-143, oar-mir-181a-2, chi-mir-1, chi-mir-127, chi-mir-134, chi-mir-136, chi-mir-143, chi-mir-154a, chi-mir-154b, chi-mir-181b, chi-mir-181c, chi-mir-181d, chi-mir-196a, chi-mir-196b, chi-mir-21, chi-mir-329a, chi-mir-329b, chi-mir-379, chi-mir-412, chi-mir-432, chi-mir-433, chi-mir-485, chi-mir-493, rno-mir-196b-2, bta-mir-668, ssc-mir-375
Other families that had a high abundance of reads were miR-134, miR-136, miR-154, miR-370, miR-412, miR-431, miR-432, miR-433, miR-485, miR-493, miR-541; a total of 11 miRNA families. [score:1]
[1 to 20 of 1 sentences]