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12 publications mentioning rno-mir-301b

Open access articles that are associated with the species Rattus norvegicus and mention the gene name mir-301b. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

[+] score: 15
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-26b, hsa-mir-29a, hsa-mir-30a, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-106a, mmu-let-7g, mmu-let-7i, mmu-mir-15b, mmu-mir-29b-1, mmu-mir-30a, mmu-mir-30b, mmu-mir-125a, mmu-mir-125b-2, mmu-mir-130a, mmu-mir-138-2, mmu-mir-181a-2, mmu-mir-182, hsa-mir-30c-2, hsa-mir-30d, mmu-mir-30e, hsa-mir-10a, hsa-mir-34a, hsa-mir-181a-2, hsa-mir-181b-1, hsa-mir-181c, hsa-mir-182, hsa-mir-181a-1, mmu-mir-297a-1, mmu-mir-297a-2, mmu-mir-301a, mmu-mir-34c, mmu-mir-34b, mmu-let-7d, mmu-mir-106a, mmu-mir-106b, hsa-let-7g, hsa-let-7i, hsa-mir-15b, hsa-mir-30b, hsa-mir-125b-1, hsa-mir-130a, hsa-mir-138-2, hsa-mir-125a, hsa-mir-125b-2, hsa-mir-138-1, mmu-mir-30c-1, mmu-mir-30c-2, mmu-mir-30d, mmu-let-7a-1, mmu-let-7a-2, mmu-let-7b, mmu-let-7c-1, mmu-let-7c-2, mmu-let-7e, mmu-let-7f-1, mmu-let-7f-2, mmu-mir-15a, mmu-mir-26b, mmu-mir-29a, mmu-mir-29c, mmu-mir-34a, rno-mir-301a, rno-let-7d, rno-mir-344a-1, mmu-mir-344-1, rno-mir-346, mmu-mir-346, rno-mir-352, hsa-mir-181b-2, mmu-mir-10a, mmu-mir-181a-1, mmu-mir-29b-2, mmu-mir-138-1, mmu-mir-181b-1, mmu-mir-181c, mmu-mir-125b-1, hsa-mir-106b, hsa-mir-29c, hsa-mir-30c-1, hsa-mir-34b, hsa-mir-34c, hsa-mir-301a, hsa-mir-30e, hsa-mir-362, mmu-mir-362, hsa-mir-369, hsa-mir-374a, mmu-mir-181b-2, hsa-mir-346, rno-let-7a-1, rno-let-7a-2, rno-let-7b, rno-let-7c-1, rno-let-7c-2, rno-let-7e, rno-let-7f-1, rno-let-7f-2, rno-let-7i, rno-mir-10a, rno-mir-15b, rno-mir-26b, rno-mir-29b-2, rno-mir-29a, rno-mir-29b-1, rno-mir-29c-1, rno-mir-30c-1, rno-mir-30e, rno-mir-30b, rno-mir-30d, rno-mir-30a, rno-mir-30c-2, rno-mir-34b, rno-mir-34c, rno-mir-34a, rno-mir-106b, rno-mir-125a, rno-mir-125b-1, rno-mir-125b-2, rno-mir-130a, rno-mir-138-2, rno-mir-138-1, rno-mir-181c, rno-mir-181a-2, rno-mir-181b-1, rno-mir-181b-2, rno-mir-181a-1, hsa-mir-449a, mmu-mir-449a, rno-mir-449a, mmu-mir-463, mmu-mir-466a, hsa-mir-483, hsa-mir-493, hsa-mir-181d, hsa-mir-499a, hsa-mir-504, mmu-mir-483, rno-mir-483, mmu-mir-369, rno-mir-493, rno-mir-369, rno-mir-374, hsa-mir-579, hsa-mir-582, hsa-mir-615, hsa-mir-652, hsa-mir-449b, rno-mir-499, hsa-mir-767, hsa-mir-449c, hsa-mir-762, mmu-mir-301b, mmu-mir-374b, mmu-mir-762, mmu-mir-344d-3, mmu-mir-344d-1, mmu-mir-673, mmu-mir-344d-2, mmu-mir-449c, mmu-mir-692-1, mmu-mir-692-2, mmu-mir-669b, mmu-mir-499, mmu-mir-652, mmu-mir-615, mmu-mir-804, mmu-mir-181d, mmu-mir-879, mmu-mir-297a-3, mmu-mir-297a-4, mmu-mir-344-2, mmu-mir-466b-1, mmu-mir-466b-2, mmu-mir-466b-3, mmu-mir-466c-1, mmu-mir-466e, mmu-mir-466f-1, mmu-mir-466f-2, mmu-mir-466f-3, mmu-mir-466g, mmu-mir-466h, mmu-mir-493, mmu-mir-504, mmu-mir-466d, mmu-mir-449b, hsa-mir-374b, hsa-mir-301b, rno-mir-466b-1, rno-mir-466b-2, rno-mir-466c, rno-mir-879, mmu-mir-582, rno-mir-181d, rno-mir-182, rno-mir-463, rno-mir-673, rno-mir-652, mmu-mir-466l, mmu-mir-669k, mmu-mir-466i, mmu-mir-669i, mmu-mir-669h, mmu-mir-466f-4, mmu-mir-466k, mmu-mir-466j, mmu-mir-1193, mmu-mir-767, rno-mir-362, rno-mir-504, rno-mir-582, rno-mir-615, mmu-mir-3080, mmu-mir-466m, mmu-mir-466o, mmu-mir-466c-2, mmu-mir-466b-4, mmu-mir-466b-5, mmu-mir-466b-6, mmu-mir-466b-7, mmu-mir-466p, mmu-mir-466n, mmu-mir-344e, mmu-mir-344b, mmu-mir-344c, mmu-mir-344g, mmu-mir-344f, mmu-mir-374c, mmu-mir-466b-8, hsa-mir-466, hsa-mir-1193, rno-mir-449c, rno-mir-344b-2, rno-mir-466d, rno-mir-344a-2, rno-mir-1193, rno-mir-344b-1, hsa-mir-374c, hsa-mir-499b, mmu-mir-466q, mmu-mir-344h-1, mmu-mir-344h-2, mmu-mir-344i, rno-mir-344i, rno-mir-344g, mmu-let-7j, mmu-mir-30f, mmu-let-7k, mmu-mir-692-3, rno-let-7g, rno-mir-15a, rno-mir-762, mmu-mir-466c-3, rno-mir-29c-2, rno-mir-29b-3, rno-mir-344b-3, rno-mir-466b-3, rno-mir-466b-4
Such a situation occurred for miR-26b, miR-30, and miR-374 downregulation, and for miR-34, miR-301, and miR-352 upregulation [121]. [score:7]
Of these miRNAs, 12 were upregulated (miR-34b, miR-138, miR-297a, miR-301, miR-449, miR-466, miR-493, miR-579, miR-582, miR. [score:4]
Three dysregulated miRNAs (miR-301, miR-369, and miR-669k) overlapped in the lungs of adenoma-bearing mice and of microadenoma-bearing mice. [score:2]
These miRNAs were miR-301, miR-369, and miR-669k, whose main functions are to regulate cell proliferation, autophagy, and aerobic glycolysis, which are mechanisms involved in the initial stage of the functional transformation of cells into their neoplastic counterpart. [score:2]
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[+] score: 14
Loss of function approach in in vitro mo del (H9C2 cells) using miR-301a inhibitor was used to test if the increase in miR-301 has a role in regulation of Kv4.2 gene expression, a voltage-gated K [+] channel. [score:6]
Specifically, mir-301 and mir-17-5p showed different expression levels after occlusion, while mir-301 expression decreased, we observed an increased in mir-17-5p expression compared to control. [score:6]
We observed a decreased miR-301 and miR-93 expressions in occlusion group compared to ablation. [score:2]
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[+] score: 9
Considering these findings, we speculate that controversial effects may not occur among different cell types, and we anticipate a consistent effect of miRNA-301a regulation of ASK1 expression and activation on hASCs [miR-301] transplanted into infarcted hearts. [score:4]
Figure 5C shows that transplanted hASCs [miR-301] had greater survivorship rates than transplanted hASCs at injected sites, with less cell death observed in hASCs [miR-301] injected heart tissue (Figure 5D). [score:1]
The fibrosis area was significantly reduced by hASCs [miR-301] injection in ischemic hearts (Figure 5B). [score:1]
Effect of hASC [miR-301] on Ischemic Myocardium. [score:1]
To determine whether miRNA-301a -transfected hASCs (hASCs [miR-301]) have a therapeutic effect on ischemic myocardium, cardiac functional improvements by hASCs [miR-301] were analyzed in normal and MI rat hearts after hASC [miR-301] transplantation. [score:1]
Effect of hASC [miR-301] on Ischemic MyocardiumTo determine whether miRNA-301a -transfected hASCs (hASCs [miR-301]) have a therapeutic effect on ischemic myocardium, cardiac functional improvements by hASCs [miR-301] were analyzed in normal and MI rat hearts after hASC [miR-301] transplantation. [score:1]
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[+] score: 7
Seven overexpressed miRNAs (miR-20a, miR-199a-5p, miR-199, miR-323, miR-301a, miR-301b and miR-130b) showed the most target mRNAs. [score:5]
The miRNAs including miR-301a, miR-301b and miR-130b regulated some important genes, including TGF-βR1, TGF-βR2, Smad2, Smad4 and Smad5 and, therefore, might be of great importance to the activation of the organ of Corti. [score:2]
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[+] score: 7
Other miRNAs from this paper: rno-mir-301a, rno-mir-130a, rno-mir-130b, rno-mir-146a
These data indicate that miR-301 negatively regulates the mRNA and protein expression of PTEN in cortical neurons. [score:4]
Western blot analysis further showed that 24 h treatment of miR-301 agomir decreased the protein expression of PTEN but 24 h treatment of miR-301 antagomir increased the level of PTEN proteins in cultured cortical neurons (Fig.   7b-c). [score:3]
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[+] score: 7
miR-448, let-7b, miR-540, miR-296, miR-880, miR-200a, miR-500, miR-10b, miR-336, miR-30d, miR-208, let-7e, miR-142-5p, miR-874, miR-375, miR-879, miR-501, and miR-188 were upregulated, while miR-301b, miR-134, and miR-652 were downregulated in TMH group (Table 5). [score:7]
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[+] score: 3
Therefore, we believe that our findings provide an important layer in Cfl2 regulation, which needs further in vivo validations for exploiting the therapeutic potential of miR-301/Cfl2 interactions in cardiac signal transduction and DCM. [score:2]
Transfection of miR-301/anti-miR301. [score:1]
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[+] score: 2
The other highly significant miRNAs displaying a steep decrease from younger to older animals (miR-301b, miR-130b, miR-20a, and miR-15b) are known to be involved in cancers (Attar et al. 2012; Funamizu et al. 2014; O’Donnell et al. 2005; Zhu et al. 2015), suggesting a role for these miRNAs in regulating MEC cell proliferation immediately after birth. [score:2]
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[+] score: 2
The endogenous control, miR-301b, was selected as the optimal normalisation strategy for MML data. [score:1]
All data were normalised to miR-301b. [score:1]
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[+] score: 2
Other miRNAs from this paper: hsa-mir-138-1, rno-mir-138-1, hsa-mir-760, hsa-mir-301b, rno-mir-760
Other genes relevant to gene regulation include epigenetic modulators (e. g. Hira, Ehmt2, Yeats2), miRNAs (e. g. Mir138-1, Mir760, Mir301b) and 16 zinc finger transcription factor genes (e. g. Zfp105, Zbtb8a, Zbtb8b, Zdhhc17). [score:2]
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[+] score: 2
These include precursor miRNAs mir-204 and mir-299a as well as mature miRNAs miR384-5p, miR-222-3p, and miR-301b-3p. [score:1]
Finally, there is mixed support for 7 of the miRNAs we reported (mir-129-1, miR-15b-3p, mir-204, miR-29c-3p, miR-301b-3p, miR-495, and mir-9a-2), with some studies showing changes consistent with our data, and other studies showing changes opposite those of our study (15, 31– 33, 38– 47). [score:1]
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[+] score: 2
Some of these miRNAs, i. e. rno-miR-34c, rno-miR-449a, rno-miR-301b, rno-miR-532-5p, rno-miR-219-5p, rno-miR-451, and rno-miR-152, were even 10-fold more abundant at E10 than at any other stages, providing a hint that these 7 miRNAs may play important roles in the regulation of progenitor cell proliferation. [score:2]
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