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4 publications mentioning ocu-mir-34a

Open access articles that are associated with the species Oryctolagus cuniculus and mention the gene name mir-34a. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

[+] score: 17
For example, miR-146b and miR-34a were up-regulated in the liver tissues of patients with non-alcoholic steatohepatitis [11], while in the CCl [4] induced liver fibrosis, miR-199a-5p and miR-199a-3p were positively and significantly correlated to the progression of liver fibrosis [12]. [score:4]
To identify miRNAs that reflected the schistosome infections and PZQ chemotherapy, six miRNA candidates (miR-146b, miR-122, miR-223, miR-199a-5p, miR-199a-3p, miR-34a) were selected for analysis in serum that were commonly deregulated in human liver diseases. [score:4]
Expression levels of serum miR-223 (B), miR-122 (C), miR-34a (D), miR-199a-5p (E) miR-199a-3p (F), and miR-146b (G) were detected in the three groups of mice. [score:3]
The expression levels of miR-34a, miR-223, miR-122, miR-146b, miR-199a-5p, miR-199a-3p were determined using the SYBR Green Master Mix kit (TaKaRa, Dalian, China). [score:3]
In mouse hosts, quantitative PCR result revealed that circulating miR-223, miR-122 and miR-34a were significantly elevated after infection (FigureĀ  1B-D). [score:1]
We analyzed the serum levels of six selected candidate miRNA molecules (miR-146b, miR-122, miR-223, miR-199a-5p, miR-199a-3p, miR-34a) from mice, rabbits, buffalos and humans infected with Schistosoma japonicum using qPCR. [score:1]
To test this hypothesis, we selected six candidate serum miRNAs for analysis (miR-146b, miR-122, miR-223, miR-199a-5p, miR-199a-3p, miR-34a) in the murine mo del of human schistosomiasis and then performed validation in other host species including rabbits, buffalos and human patients infected with S. japonicum. [score:1]
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[+] score: 16
In this study, we found that, in addition to lowered levels of miR-122, expression of miR-223, miR-34a, and miR-127 was also down-regulated. [score:6]
miR-223 is noteworthy because it is frequently suppressed in human HCC [52], and miR-34a, miR-127 and miR-200b are down-regulated in the rat liver during experimental hepatocarcinogenesis [36]. [score:6]
Tryndyak V. P. Ross S. A. Beland F. A. Pogribny I. P. Down-regulation of the microRNAs miR-34a, miR-127, and miR-200b in rat liver during hepatocarcinogenesis induced by a methyl -deficient diet Mol. [score:4]
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[+] score: 4
We reported the involvement of several miRNAs including miR-34a [9, 10] and miR-221 [11] in JNK-regulated chondrogenic differentiation. [score:2]
Our laboratory also showed that JNK signaling is involved in the differentiation of chondroprogenitors in chicks through regulation of miR-34a [9, 10] and miR-221 levels [11]. [score:2]
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[+] score: 4
[34] Recent reports also showed that several miRNAs (miR-34a) and transcription factors (Ī²-catenin and slug) regulate CD171 expression in endometrial adenocarcinomas. [score:4]
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